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Cellular Stress Responses in Radiotherapy
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2 of the 135 references listed on this page are in the Retraction Watch database as retracted (export 2026-10-28): 10.1002/mc.21858 (notice 2026-02-24), 10.1074/jbc.m800102200 (notice 2023-08-07).
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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inferredcites →https://doi.org/10.1080/02656736.2017.1308019openalex-snapshotGadolinium-doped iron oxide nanoparticles induced magnetic field hyperthermia combined with radiotherapy increases tumour response by vascular disruption and improved oxygenation
inferredcites →https://doi.org/10.1128/mcb.00166-09openalex-snapshotHypoxia-Induced Autophagy Is Mediated through Hypoxia-Inducible Factor Induction of BNIP3 and BNIP3L via Their BH3 Domains
inferredcites →https://doi.org/10.1128/mcb.23.4.1292-1303.2003openalex-snapshotGrowth Arrest and DNA Damage-Inducible Protein GADD34 Targets Protein Phosphatase 1α to the Endoplasmic Reticulum and Promotes Dephosphorylation of the α Subunit of Eukaryotic Translation Initiation Factor 2
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inferredcites →https://doi.org/10.4161/cc.10.14.16588openalex-snapshotThe relative contribution of pro-apoptotic p53-target genes in the triggering of apoptosis following DNA damage in vitro and in vivo
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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inferred← citeshttps://doi.org/10.1155/2021/6620306openalex-snapshotAntioxidative Stress: Inhibiting Reactive Oxygen Species Production as a Cause of Radioresistance and Chemoresistance
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