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Blood-Brain Barrier Dysfunction and the Pathogenesis of Alzheimer’s Disease
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1 of the 173 references listed on this page is in the Retraction Watch database as retracted (export 2026-11-04): 10.1038/ncomms3932 (notice 2024-04-03).
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Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/jamaneurol.2016.4370openalex-snapshot
Association of Perivascular Localization of Aquaporin-4 With Cognition and Alzheimer Disease in Aging Brains
inferredcites →https://doi.org/10.1002/ana.23648openalex-snapshot
The blood–brain barrier in health and disease
inferredcites →https://doi.org/10.1002/dneu.20954openalex-snapshot
Extracellular matrix and matrix receptors in blood–brain barrier formation and stroke
inferredcites →https://doi.org/10.1007/bf00193951openalex-snapshot
Immunolabelling of hippocampal microvessel glucose transporter protein is reduced in Alzheimer's disease
inferredcites →https://doi.org/10.1007/bf00693782openalex-snapshot
Blood-brain barrier in Alzheimer dementia and in non-demented elderly
inferredcites →https://doi.org/10.1007/s00401-003-0688-zopenalex-snapshot
Localization of claudin-3 in tight junctions of the blood-brain barrier is selectively lost during experimental autoimmune encephalomyelitis and human glioblastoma multiforme
inferredcites →https://doi.org/10.1007/s00401-011-0834-yopenalex-snapshot
Vascular β-amyloid and early astrocyte alterations impair cerebrovascular function and cerebral metabolism in transgenic arcAβ mice
inferredcites →https://doi.org/10.1007/s00401-016-1555-zopenalex-snapshot
Vascular basement membranes as pathways for the passage of fluid into and out of the brain
inferredcites →https://doi.org/10.1007/s00401-016-1580-yopenalex-snapshot
Impact of sex and APOE4 on cerebral amyloid angiopathy in Alzheimer’s disease
inferredcites →https://doi.org/10.1007/s004010000180openalex-snapshot
Claudin-1 and claudin-5 expression and tight junction morphology are altered in blood vessels of human glioblastoma multiforme
inferredcites →https://doi.org/10.1007/s00441-005-1101-0openalex-snapshot
Contribution of claudin-5 to barrier properties in tight junctions of epithelial cells
inferredcites →https://doi.org/10.1007/s40263-016-0361-4openalex-snapshot
Apolipoprotein E as a Therapeutic Target in Alzheimer’s Disease: A Review of Basic Research and Clinical Evidence
inferredcites →https://doi.org/10.1016/0006-3223(90)90410-4openalex-snapshot
Rapid dynamic CT scanning in primary degenerative dementia and age-matched controls
inferredcites →https://doi.org/10.1016/0006-8993(95)01250-8openalex-snapshot
Increased collagen content of cerebral microvessels in Alzheimer's disease
inferredcites →https://doi.org/10.1016/0022-510x(83)90157-0openalex-snapshot
Albumin and immunoglobulin in plasma and cerebrospinal fluid, and blood-cerebrospinal fluid barrier function in patients with dementia of alzheimer type and multi-infarct dementia
inferredcites →https://doi.org/10.1016/0022-510x(85)90189-3openalex-snapshot
CSF in Alzheimer's disease
inferredcites →https://doi.org/10.1016/0026-2862(85)90042-1openalex-snapshot
Morphometric analysis of CNS microvascular endothelium
inferredcites →https://doi.org/10.1016/j.bbadis.2015.12.023openalex-snapshot
Vascular contributions to cognitive impairment, clinical Alzheimer's disease, and dementia in older persons
inferredcites →https://doi.org/10.1016/j.braindev.2009.02.008openalex-snapshot
The expanding phenotype of GLUT1-deficiency syndrome
inferredcites →https://doi.org/10.1016/j.brainres.2007.08.063openalex-snapshot
Amyloid beta deposition is related to decreased glucose transporter-1 levels and hippocampal atrophy in brains of aged APP/PS1 mice
inferredcites →https://doi.org/10.1016/j.brainres.2008.06.124openalex-snapshot
Hippocampal RAGE immunoreactivity in early and advanced Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.brainres.2014.10.047openalex-snapshot
In vitro discrimination of the role of LRP1 at the BBB cellular level: Focus on brain capillary endothelial cells and brain pericytes
inferredcites →https://doi.org/10.1016/j.dadm.2017.01.005openalex-snapshot
Pathophysiologic relationship between Alzheimer's disease, cerebrovascular disease, and cardiovascular risk: A review and synthesis
inferredcites →https://doi.org/10.1016/j.jalz.2016.03.001openalex-snapshot
2016 Alzheimer's disease facts and figures
inferredcites →https://doi.org/10.1016/j.jns.2009.02.089openalex-snapshot
Blood brain barrier: ageing and microvascular disease-systematic review and meta-analysis
inferredcites →https://doi.org/10.1016/j.nbd.2007.12.005openalex-snapshot
LRP promotes endocytosis and degradation, but not transcytosis, of the amyloid-β peptide in a blood–brain barrier in vitro model
inferredcites →https://doi.org/10.1016/j.nbd.2016.07.007openalex-snapshot
The blood-brain barrier in Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.neulet.2006.03.047openalex-snapshot
Amyloid β-peptide1–42 alters tight junction protein distribution and expression in brain microvessel endothelial cells
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2006.05.016openalex-snapshot
Microvascular injury and blood–brain barrier leakage in Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2007.07.015openalex-snapshot
Blood–brain barrier: Ageing and microvascular disease – systematic review and meta-analysis
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2008.08.024openalex-snapshot
Is it appropriate to use albumin CSF/plasma ratio to assess blood brain barrier permeability?
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2010.05.025openalex-snapshot
LRP1 mediates bidirectional transcytosis of amyloid-β across the blood-brain barrier
inferredcites →https://doi.org/10.1016/j.neuron.2004.07.017openalex-snapshot
LRP/Amyloid β-Peptide Interaction Mediates Differential Brain Efflux of Aβ Isoforms
inferredcites →https://doi.org/10.1016/j.neuron.2008.01.003openalex-snapshot
The Blood-Brain Barrier in Health and Chronic Neurodegenerative Disorders
inferredcites →https://doi.org/10.1016/j.neuron.2010.09.043openalex-snapshot
Pericytes Control Key Neurovascular Functions and Neuronal Phenotype in the Adult Brain and during Brain Aging
inferredcites →https://doi.org/10.1016/j.neuron.2013.04.005openalex-snapshot
Innate Immunity in the CNS: Redefining the Relationship between the CNS and Its Environment
inferredcites →https://doi.org/10.1016/j.neuron.2014.05.004openalex-snapshot
The Intersection of Amyloid Beta and Tau at Synapses in Alzheimer’s Disease
inferredcites →https://doi.org/10.1016/j.neuron.2014.12.032openalex-snapshot
Blood-Brain Barrier Breakdown in the Aging Human Hippocampus
inferredcites →https://doi.org/10.1016/j.neuroscience.2009.01.023openalex-snapshot
Vascular amyloid alters astrocytic water and potassium channels in mouse models and humans with Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.pscychresns.2008.04.003openalex-snapshot
Blood–brain barrier permeability in Alzheimer's disease: a case–control MRI study
inferredcites →https://doi.org/10.1016/j.seizure.2013.07.003openalex-snapshot
GLUT1 deficiency syndrome 2013: Current state of the art
inferredcites →https://doi.org/10.1016/j.tins.2005.02.001openalex-snapshot
Neurovascular mechanisms of Alzheimer's neurodegeneration
inferredcites →https://doi.org/10.1016/j.tins.2015.08.003openalex-snapshot
The Molecular Constituents of the Blood–Brain Barrier
inferredcites →https://doi.org/10.1016/s0169-409x(98)00085-4openalex-snapshot
P-Glycoprotein, a gatekeeper in the blood–brain barrier
inferredcites →https://doi.org/10.1016/s0197-4580(88)80085-xopenalex-snapshot
Lack of evidence for dysfunction of the blood-brain barrier in Alzhiemer's disease: An immunohistochemical study
inferredcites →https://doi.org/10.1016/s0197-4580(97)00111-5openalex-snapshot
GLUT-1 Expression in the Cerebra of Patients with Alzheimer’s Disease
inferredcites →https://doi.org/10.1016/s0301-0082(00)00068-xopenalex-snapshot
Cerebral microvascular pathology in aging and Alzheimer's disease
inferredcites →https://doi.org/10.1016/s0962-8924(99)01578-0openalex-snapshot
Occludin and claudins in tight-junction strands: leading or supporting players?
inferredcites →https://doi.org/10.1016/s1474-4422(15)70016-5openalex-snapshot
Neuroinflammation in Alzheimer's disease
inferredcites →https://doi.org/10.1038/325253a0openalex-snapshot
Astrocytes induce blood–brain barrier properties in endothelial cells
inferredcites →https://doi.org/10.1038/cddis.2014.258openalex-snapshot
Aβ1–42 reduces P-glycoprotein in the blood–brain barrier through RAGE–NF-κB signaling
inferredcites →https://doi.org/10.1038/jcbfm.1987.1openalex-snapshot
Examination of Blood — Brain Barrier Permeability in Dementia of the Alzheimer Type with [ 68 Ga]EDTA and Positron Emission Tomography
inferredcites →https://doi.org/10.1038/jcbfm.2012.153openalex-snapshot
Immunologic Privilege in the Central Nervous System and the Blood–Brain Barrier
inferredcites →https://doi.org/10.1038/jcbfm.2013.135openalex-snapshot
Blood–Brain Barrier Dysfunction as a Cause and Consequence of Alzheimer's Disease
inferredcites →https://doi.org/10.1038/jcbfm.2014.102openalex-snapshot
Ablation of MMP9 Gene Ameliorates Paracellular Permeability and Fibrinogen–Amyloid Beta Complex Formation during Hyperhomocysteinemia
inferredcites →https://doi.org/10.1038/nature05193openalex-snapshot
Bidirectional control of CNS capillary diameter by pericytes
inferredcites →https://doi.org/10.1038/nature09513openalex-snapshot
Pericytes are required for blood–brain barrier integrity during embryogenesis
inferredcites →https://doi.org/10.1038/nature09522openalex-snapshot
Pericytes regulate the blood–brain barrier
inferredcites →https://doi.org/10.1038/nature09613openalex-snapshot
Glial and neuronal control of brain blood flow
inferredcites →https://doi.org/10.1038/ncomms3932openalex-snapshot
RETRACTED ARTICLE: Pericyte loss influences Alzheimer-like neurodegeneration in mice
inferredcites →https://doi.org/10.1038/nm.3913openalex-snapshot
Neutrophils promote Alzheimer's disease–like pathology and cognitive decline via LFA-1 integrin
inferredcites →https://doi.org/10.1038/nm889openalex-snapshot
SSeCKS regulates angiogenesis and tight junction formation in blood-brain barrier
inferredcites →https://doi.org/10.1038/nm890openalex-snapshot
RAGE mediates amyloid-β peptide transport across the blood-brain barrier and accumulation in brain
inferredcites →https://doi.org/10.1038/nn.2946openalex-snapshot
Central nervous system pericytes in health and disease
inferredcites →https://doi.org/10.1038/nn.3966openalex-snapshot
GLUT1 reductions exacerbate Alzheimer's disease vasculo-neuronal dysfunction and degeneration
inferredcites →https://doi.org/10.1038/nn.4288openalex-snapshot
Pericytes of the neurovascular unit: key functions and signaling pathways
inferredcites →https://doi.org/10.1038/nn.4475openalex-snapshot
The role of peripheral immune cells in the CNS in steady state and disease
inferredcites →https://doi.org/10.1038/nri2852openalex-snapshot
The impact of the extracellular matrix on inflammation
inferredcites →https://doi.org/10.1038/nri3265openalex-snapshot
The anatomical and cellular basis of immune surveillance in the central nervous system
inferredcites →https://doi.org/10.1038/nrn1824openalex-snapshot
Astrocyte–endothelial interactions at the blood–brain barrier
inferredcites →https://doi.org/10.1038/nrneurol.2012.263openalex-snapshot
Apolipoprotein E and Alzheimer disease: risk, mechanisms and therapy
inferredcites →https://doi.org/10.1038/sj.jcbfm.9600249openalex-snapshot
The Rat Blood—Brain Barrier Transcriptome
inferredcites →https://doi.org/10.1046/j.1471-4159.1998.71031151.xopenalex-snapshot
Basement Membrane Proteins Influence Brain Capillary Endothelial Barrier Function In Vitro
inferredcites →https://doi.org/10.1056/nejm199109053251006openalex-snapshot
Defective Glucose Transport across the Blood-Brain Barrier as a Cause of Persistent Hypoglycorrhachia, Seizures, and Developmental Delay
inferredcites →https://doi.org/10.1073/pnas.0805165106openalex-snapshot
Wnt/β-catenin signaling is required for CNS, but not non-CNS, angiogenesis
inferredcites →https://doi.org/10.1074/jbc.273.45.29745openalex-snapshot
The Tight Junction Protein ZO-1 Establishes a Link between the Transmembrane Protein Occludin and the Actin Cytoskeleton
inferredcites →https://doi.org/10.1074/jbc.m110.102277openalex-snapshot
Reduction of Brain β-Amyloid (Aβ) by Fluvastatin, a Hydroxymethylglutaryl-CoA Reductase Inhibitor, through Increase in Degradation of Amyloid Precursor Protein C-terminal Fragments (APP-CTFs) and Aβ Clearance
inferredcites →https://doi.org/10.1074/jbc.m608853200openalex-snapshot
Study of Claudin Function by RNA Interference
inferredcites →https://doi.org/10.1074/jbc.m801487200openalex-snapshot
The Low Density Lipoprotein Receptor-related Protein 1 Mediates Uptake of Amyloid β Peptides in an in Vitro Model of the Blood-Brain Barrier Cells
inferredcites →https://doi.org/10.1083/jcb.123.6.1777openalex-snapshot
Occludin: a novel integral membrane protein localizing at tight junctions.
inferredcites →https://doi.org/10.1083/jcb.136.2.399openalex-snapshot
A Synthetic Peptide Corresponding to the Extracellular Domain of Occludin Perturbs the Tight Junction Permeability Barrier
inferredcites →https://doi.org/10.1083/jcb.143.2.391openalex-snapshot
A Single Gene Product, Claudin-1 or -2, Reconstitutes Tight Junction Strands and Recruits Occludin in Fibroblasts
inferredcites →https://doi.org/10.1083/jcb.147.6.1351openalex-snapshot
Direct Binding of Three Tight Junction-Associated Maguks, Zo-1, Zo-2, and Zo-3, with the Cooh Termini of Claudins
inferredcites →https://doi.org/10.1083/jcb.153.2.263openalex-snapshot
Conversion of Zonulae Occludentes from Tight to Leaky Strand Type by Introducing Claudin-2 into Madin-Darby Canine Kidney I Cells
inferredcites →https://doi.org/10.1083/jcb.153.5.933openalex-snapshot
Endothelial Cell Laminin Isoforms, Laminins 8 and 10, Play Decisive Roles in T Cell Recruitment across the Blood–Brain Barrier in Experimental Autoimmune Encephalomyelitis
inferredcites →https://doi.org/10.1083/jcb.200302070openalex-snapshot
Size-selective loosening of the blood-brain barrier in claudin-5–deficient mice
inferredcites →https://doi.org/10.1083/jcb.201404140openalex-snapshot
ZO-1 controls endothelial adherens junctions, cell–cell tension, angiogenesis, and barrier formation
inferredcites →https://doi.org/10.1089/ars.2011.3895openalex-snapshot
Amyloid Beta Induces Oxidative Stress-Mediated Blood–Brain Barrier Changes in Capillary Amyloid Angiopathy
inferredcites →https://doi.org/10.1091/mbc.11.12.4131openalex-snapshot
Complex Phenotype of Mice Lacking Occludin, a Component of Tight Junction Strands
inferredcites →https://doi.org/10.1093/brain/awr298openalex-snapshot
Blood–brain barrier P-glycoprotein function in Alzheimer's disease
inferredcites →https://doi.org/10.1093/brain/aws271openalex-snapshot
Cerebrovascular atherosclerosis correlates with Alzheimer pathology in neurodegenerative dementias
inferredcites →https://doi.org/10.1097/00001756-200004070-00035openalex-snapshot
Neurons and ECM regulate occludin localization in brain endothelial cells
inferredcites →https://doi.org/10.1097/00002093-199912001-00015openalex-snapshot
Cerebrospinal Fluid Markers of Pathogenetic Processes in Vascular Dementia, with Special Reference to the Subcortical Subtype
inferredcites →https://doi.org/10.1097/00008571-200210000-00005openalex-snapshot
Deposition of Alzheimer's ??-amyloid is inversely correlated with P-glycoprotein expression in the brains of elderly non-demented humans
inferredcites →https://doi.org/10.1097/01.jnen.0000173894.09553.13openalex-snapshot
Basement Membrane Proteins in Multiple Sclerosis-Associated Inflammatory Cuffs: Potential Role in Influx and Transport of Leukocytes
inferredcites →https://doi.org/10.1097/nen.0b013e31818f9ca8openalex-snapshot
Perivascular Spaces and the Two Steps to Neuroinflammation
inferredcites →https://doi.org/10.1097/nen.0b013e3181fd252copenalex-snapshot
Aquaporin Expression in the Brains of Patients With or Without Cerebral Amyloid Angiopathy
inferredcites →https://doi.org/10.1097/nen.0b013e3182632566openalex-snapshot
Characteristics of Aquaporin Expression Surrounding Senile Plaques and Cerebral Amyloid Angiopathy in Alzheimer Disease
inferredcites →https://doi.org/10.1101/cshperspect.a006189openalex-snapshot
Neuropathological Alterations in Alzheimer Disease
inferredcites →https://doi.org/10.1101/cshperspect.a006379openalex-snapshot
Proteolytic Degradation of Amyloid -Protein
inferredcites →https://doi.org/10.1111/acel.12045openalex-snapshot
Regional differences in the morphological and functional effects of aging on cerebral basement membranes and perivascular drainage of amyloid‐β from the mouse brain
inferredcites →https://doi.org/10.1111/bpa.12004openalex-snapshot
Deficiency in Mural Vascular Cells Coincides with Blood–Brain Barrier Disruption in A lzheimer's Disease
inferredcites →https://doi.org/10.1111/cns.12503openalex-snapshot
Hyperglycemia Increases the Production of Amyloid Beta‐Peptide Leading to Decreased Endothelial Tight Junction
inferredcites →https://doi.org/10.1111/j.1365-2990.2006.00732.xopenalex-snapshot
Relationships in Alzheimer's disease between the extent of Aβ deposition in cerebral blood vessel walls, as cerebral amyloid angiopathy, and the amount of cerebrovascular smooth muscle cells and collagen
inferredcites →https://doi.org/10.1111/j.1471-4159.2010.06708.xopenalex-snapshot
Lack of brain‐to‐blood efflux transport activity of low‐density lipoprotein receptor‐related protein‐1 (LRP‐1) for amyloid‐β peptide(1–40) in mouse: involvement of an LRP‐1‐independent pathway
inferredcites →https://doi.org/10.1111/j.1582-4934.2009.00717.xopenalex-snapshot
Amyloid-β-induced occludin down-regulation and increased permeability in human brain endothelial cells is mediated by MAPK activation
inferredcites →https://doi.org/10.1111/j.1600-0404.1990.tb01563.xopenalex-snapshot
Blood-brain barrier disturbance in patients with Alzheimer's disease is related to vascular factors
inferredcites →https://doi.org/10.1111/j.1600-0404.1991.tb04675.xopenalex-snapshot
White-matter lesions on CT in Alzheimer patients: relation to clinical symptomatology and vascular factors
inferredcites →https://doi.org/10.1111/j.1600-0404.2007.00823.xopenalex-snapshot
The integrity of the blood?brain barrier in Alzheimer?s disease
inferredcites →https://doi.org/10.1111/j.1749-6632.1982.tb26848.xopenalex-snapshot
Evidence for Blood‐Brain Barrier Changes in Senile Dementia of the Alzheimer Type (SDAT)
inferredcites →https://doi.org/10.1111/j.1749-6632.1997.tb48468.xopenalex-snapshot
Amyloid Angiopathy and Blood–Brain Barrier Changes in Alzheimer's Disease a, b
inferredcites →https://doi.org/10.1111/jnc.13122openalex-snapshot
Aβ1–42oligomer‐induced leakage in anin vitroblood–brain barrier model is associated with up‐regulation of RAGE and metalloproteinases, and down‐regulation of tight junction scaffold proteins
inferredcites →https://doi.org/10.1111/nan.12295openalex-snapshot
Early changes in extracellular matrix in Alzheimer's disease
inferredcites →https://doi.org/10.1113/expphysiol.2010.053132openalex-snapshot
Bidirectional control of arteriole diameter by astrocytes
inferredcites →https://doi.org/10.1124/mol.109.061754openalex-snapshot
Restoring Blood-Brain Barrier P-Glycoprotein Reduces Brain Amyloid-β in a Mouse Model of Alzheimer’s Disease
inferredcites →https://doi.org/10.1126/science.6101511openalex-snapshot
γ-Glutamyl Transpeptidase in Isolated Brain Endothelial Cells: Induction by Glial Cells in Vitro
inferredcites →https://doi.org/10.1126/scitranslmed.3003748openalex-snapshot
A Paravascular Pathway Facilitates CSF Flow Through the Brain Parenchyma and the Clearance of Interstitial Solutes, Including Amyloid β
inferredcites →https://doi.org/10.1139/y92-252openalex-snapshot
Changes in the glucose transporter of brain capillaries
inferredcites →https://doi.org/10.1152/physrev.00001.2003openalex-snapshot
Transcytosis: Crossing Cellular Barriers
inferredcites →https://doi.org/10.1152/physrev.00019.2012openalex-snapshot
Claudins and the Modulation of Tight Junction Permeability
inferredcites →https://doi.org/10.1152/physrev.00049.2005openalex-snapshot
Astrocyte Control of Synaptic Transmission and Neurovascular Coupling
inferredcites →https://doi.org/10.1158/1078-0432.ccr-06-2854openalex-snapshot
The Blood-Brain Barrier and Cancer: Transporters, Treatment, and Trojan Horses
inferredcites →https://doi.org/10.1159/000334916openalex-snapshot
Neuroinflammation and Blood-Brain Barrier Changes in Capillary Amyloid Angiopathy
inferredcites →https://doi.org/10.1159/000444053openalex-snapshot
Dietary Advanced Glycation End Products and Their Potential Role in Cardiometabolic Disease in Children
inferredcites →https://doi.org/10.1161/01.res.0000182903.16652.d7openalex-snapshot
Endothelial/Pericyte Interactions
inferredcites →https://doi.org/10.1161/01.res.0000255690.03436.aeopenalex-snapshot
Phenotypic Heterogeneity of the Endothelium
inferredcites →https://doi.org/10.1161/01.str.0000143318.70292.47openalex-snapshot
Risk Factors for Vascular Dementia and Alzheimer Disease
inferredcites →https://doi.org/10.1161/01.str.27.11.2069openalex-snapshot
Alterations of the Blood-Brain Barrier and Glial Cells in White-Matter Lesions in Cerebrovascular and Alzheimer's Disease Patients
inferredcites →https://doi.org/10.1172/jci10498openalex-snapshot
Clearance of Alzheimer’s amyloid-β1-40 peptide from brain by LDL receptor–related protein-1 at the blood-brain barrier
inferredcites →https://doi.org/10.1172/jci12464openalex-snapshot
Regulated expression of claudin-4 decreases paracellular conductance through a selective decrease in sodium permeability
inferredcites →https://doi.org/10.1172/jci25247openalex-snapshot
P-glycoprotein deficiency at the blood-brain barrier increases amyloid- deposition in an Alzheimer disease mouse model
inferredcites →https://doi.org/10.1172/jci58642openalex-snapshot
A multimodal RAGE-specific inhibitor reduces amyloid β–mediated brain disorder in a mouse model of Alzheimer disease
inferredcites →https://doi.org/10.1172/jci81108openalex-snapshot
Endothelial LRP1 transports amyloid-β1–42 across the blood-brain barrier
inferredcites →https://doi.org/10.1176/jnp.10.1.78openalex-snapshot
Blood–Brain Barrier Integrity in Alzheimer's Disease Patients and Elderly Control Subjects
inferredcites →https://doi.org/10.1177/0192623310385254openalex-snapshot
Role of Astrocytes in Brain Function and Disease
inferredcites →https://doi.org/10.1182/blood.v91.10.3527openalex-snapshot
Endothelial cells in physiology and in the pathophysiology of vascular disorders.
inferredcites →https://doi.org/10.1186/1471-2121-11-68openalex-snapshot
Effect of cAMP derivates on assembly and maintenance of tight junctions in human umbilical vein endothelial cells
inferredcites →https://doi.org/10.1186/1742-2094-8-26openalex-snapshot
Neurovascular dysfunction, inflammation and endothelial activation: Implications for the pathogenesis of Alzheimer's disease
inferredcites →https://doi.org/10.1186/2045-8118-8-8openalex-snapshot
The CNS microvascular pericyte: pericyte-astrocyte crosstalk in the regulation of tissue survival
inferredcites →https://doi.org/10.1186/s12916-014-0130-5openalex-snapshot
Cardiovascular risk factors and future risk of Alzheimer’s disease
inferredcites →https://doi.org/10.1189/jlb.0710432openalex-snapshot
Vascular inflammation in central nervous system diseases: adhesion receptors controlling leukocyte–endothelial interactions
inferredcites →https://doi.org/10.1189/jlb.1012519openalex-snapshot
RAGE regulation and signaling in inflammation and beyond
inferredcites →https://doi.org/10.1212/wnl.0b013e31828726f5openalex-snapshot
Alzheimer disease in the United States (2010–2050) estimated using the 2010 census
inferredcites →https://doi.org/10.1212/wnl.46.6.1592openalex-snapshot
Cerebral amyloid angiopathy in the brains of patients with Alzheimer's disease
inferredcites →https://doi.org/10.1212/wnl.50.4.966openalex-snapshot
A population study on blood-brain barrier function in 85-year-olds
inferredcites →https://doi.org/10.1212/wnl.57.10.1763openalex-snapshot
The cerebrospinal fluid production rate is reduced in dementia of the Alzheimer’s type
inferredcites →https://doi.org/10.1242/jcs.017897openalex-snapshot
The role of adherens junctions and VE-cadherin in the control of vascular permeability
inferredcites →https://doi.org/10.1242/jcs.109.9.2287openalex-snapshot
Occludin is a functional component of the tight junction
inferredcites →https://doi.org/10.1371/journal.pone.0013741openalex-snapshot
The Mouse Blood-Brain Barrier Transcriptome: A New Resource for Understanding the Development and Function of Brain Endothelial Cells
inferredcites →https://doi.org/10.1371/journal.pone.0041636openalex-snapshot
Disruption of Arterial Perivascular Drainage of Amyloid-β from the Brains of Mice Expressing the Human APOE ε4 Allele
inferredcites →https://doi.org/10.1385/1-59259-419-0:193openalex-snapshot
A Review of Blood–Brain Barrier Transport Techniques
inferredcites →https://doi.org/10.1523/jneurosci.0350-15.2016openalex-snapshot
Aβ40 Reduces P-Glycoprotein at the Blood–Brain Barrier through the Ubiquitin–Proteasome Pathway
inferredcites →https://doi.org/10.1523/jneurosci.14-04-02225.1994openalex-snapshot
Increased levels of hemoglobin-derived and other peptides in Alzheimer's disease cerebellum
inferredcites →https://doi.org/10.1523/jneurosci.4707-08.2009openalex-snapshot
Uniquely Hominid Features of Adult Human Astrocytes
inferredcites →https://doi.org/10.1523/jneurosci.5103-08.2009openalex-snapshot
ABCG2 Is Upregulated in Alzheimer's Brain with Cerebral Amyloid Angiopathy and May Act as a Gatekeeper at the Blood–Brain Barrier for Aβ 1–40 Peptides
inferredcites →https://doi.org/10.1523/jneurosci.6102-11.2012openalex-snapshot
A 1-42-RAGE Interaction Disrupts Tight Junctions of the Blood-Brain Barrier Via Ca2+-Calcineurin Signaling
inferredcites →https://doi.org/10.1530/jme-11-0035openalex-snapshot
Endocrine cells and blood vessels work in tandem to generate hormone pulses
inferredcites →https://doi.org/10.2174/156720508785132334openalex-snapshot
Structural Changes in Alzheimers Disease Brain Microvessels
inferredcites →https://doi.org/10.3233/jad-2011-110725openalex-snapshot
Loss of Astrocyte Polarization in the Tg-ArcSwe Mouse Model of Alzheimer's Disease
inferredcites →https://doi.org/10.3233/jad-2012-112189openalex-snapshot
ABCG2- and ABCG4-Mediated Efflux of Amyloid-β Peptide 1-40 at the Mouse Blood-Brain Barrier
inferredcites →https://doi.org/10.3389/fnagi.2014.00093openalex-snapshot
The low-density lipoprotein receptor-related protein 1 and amyloid-β clearance in Alzheimer’s disease
inferredcites →https://doi.org/10.3389/fnagi.2014.00251openalex-snapshot
The Cerebrovascular Basement Membrane: Role in the Clearance of β-amyloid and Cerebral Amyloid Angiopathy
inferredcites →https://doi.org/10.3389/fnene.2010.00005openalex-snapshot
Pericyte-mediated regulation of capillary diameter: a component of neurovascular coupling in health and disease
inferredcites →https://doi.org/10.3389/fneng.2013.00007openalex-snapshot
The blood-brain barrier: an engineering perspective
inferredcites →https://doi.org/10.4049/jimmunol.158.4.1825openalex-snapshot
Cytokine-induced VCAM-1 and ICAM-1 expression in different organs of the mouse
inferredcites →https://doi.org/10.4161/cam.22680openalex-snapshot
Laminin isoforms in endothelial and perivascular basement membranes
inferredcites →https://doi.org/10.4199/c00029ed1v01y201103isp017openalex-snapshot
Regulation of Tissue Oxygenation
inferredcites →https://doi.org/10.55782/ane-2006-1623openalex-snapshot
The role of astrocytes in the physiology and pathology of the central nervous system
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Taking Advantages of Blood–Brain Barrier to Optimize Delivery of Drugs in Central Neurological Conditions
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Tau and apolipoprotein E modulate cerebrovascular tight junction integrity independent of cerebral amyloid angiopathy in Alzheimer's disease
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REMOVED: Nanowired delivery of cerebrolysin with neprilysin and p-Tau antibodies induces superior neuroprotection in Alzheimer's disease
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Emotional stress and cardiovascular health: Impacts on neurodegenerative disease progression
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A multifactorial lens on risk factors promoting the progression of Alzheimer’s disease
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Endothelial progenitor cells and cerebral small vessel disease in APOE4 carriers
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Crosstalk between peripheral immunity and central nervous system in Alzheimer’s disease
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Perfluorooctane sulfonate disrupts the blood brain barrier through the crosstalk between endothelial cells and astrocytes in mice
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MicroRNAs in central nervous system diseases: A prospective role in regulating blood-brain barrier integrity
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Neural stem cell therapy for neurovascular injury in Alzheimer's disease
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PART1 destabilized by NOVA2 regulates blood–brain barrier permeability in endothelial cells via STAU1-mediated mRNA degradation
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Modulating the blood-brain barrier in CNS disorders: A review of the therapeutic implications of secreted protein acidic and rich in cysteine (SPARC)
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Design of albumin-based nanotherapeutics for glioblastoma management: Biological barriers, targeting strategies, and translational perspectives
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An insight into the TAM system in Alzheimer’s disease
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Alzheimer's Aβ assembly binds sodium pump and blocks endothelial NOS activity via ROS-PKC pathway in brain vascular endothelial cells
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Near-infrared light-triggered methylene blue/berberine co-loaded polydopamine nanoparticles modified with sialic acid for Alzheimer’s disease therapy
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Dual-drug loaded nanoparticles of Epigallocatechin-3-gallate (EGCG)/Ascorbic acid enhance therapeutic efficacy of EGCG in a APPswe/PS1dE9 Alzheimer's disease mice model
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Blood brain barrier-on-a-chip to model neurological diseases
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Co-exposure of microcystin-LR and polystyrene microplastics induced testosterone synthesis inhibition by disrupting the hypothalamic-pituitary-testicular axis via PP2A/JNK and PKC signaling pathway in mice
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Fluid–structure interaction for highly complex, statistically defined, biological media: Homogenisation and a 3D multi-compartmental poroelastic model for brain biomechanics
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Materials for blood brain barrier modeling in vitro
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Time- and region-dependent blood-brain barrier impairment in a rat model of organophosphate-induced status epilepticus
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Involvement of noncoding RNA in blood-brain barrier integrity in central nervous system disease
inferred← citeshttps://doi.org/10.1016/j.neuint.2019.03.014openalex-snapshot
Can magnesium reduce central neurodegeneration in Alzheimer's disease? Basic evidences and research needs
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NMDA mediates disruption of blood-brain barrier permeability via Rho/ROCK signaling pathway
inferred← citeshttps://doi.org/10.1016/j.neuint.2026.106115openalex-snapshot
Redox-sensitive high mobility group box 1 (HMGB1) protein is a multipotent regulator in the pathogenesis of Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.neuint.2026.106258openalex-snapshot
Neuroinflammation and vascular pathology in Alzheimer's Disease: The NRF2/HO-1 axis as a modulator of microglial phenotypes
inferred← citeshttps://doi.org/10.1016/j.neulet.2021.136221openalex-snapshot
Molecular mechanisms of sex hormones in the development and progression of Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2019.09.017openalex-snapshot
White matter hyperintensities mediate the association between blood-brain barrier leakage and information processing speed
inferred← citeshttps://doi.org/10.1016/j.neuroimage.2018.09.044openalex-snapshot
Exploring Alzheimer's disease mouse brain through X-ray phase contrast tomography: From the cell to the organ
inferred← citeshttps://doi.org/10.1016/j.neuroimage.2020.116944openalex-snapshot
White matter hyperintensities are associated with subthreshold amyloid accumulation
inferred← citeshttps://doi.org/10.1016/j.neuron.2019.06.002openalex-snapshot
Cerebral Microvascular Injury: A Potentially Treatable Endophenotype of Traumatic Brain Injury-Induced Neurodegeneration
inferred← citeshttps://doi.org/10.1016/j.neuron.2020.02.034openalex-snapshot
Alzheimer’s Risk Factors Age, APOE Genotype, and Sex Drive Distinct Molecular Pathways
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2020.11.015openalex-snapshot
A Multilevel View of the Development of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1016/j.neurot.2023.10.009openalex-snapshot
Alzheimer's disease brain endothelial-like cells reveal differential drug transporter expression and modulation by potentially therapeutic focused ultrasound
inferred← citeshttps://doi.org/10.1016/j.npep.2026.102652openalex-snapshot
Perioperative orexin-A protects brain microvascular endothelial integrity in experimental sepsis-associated encephalopathy
inferred← citeshttps://doi.org/10.1016/j.nxnano.2026.100495openalex-snapshot
Nanomedicine approaches for Alzheimer’s disease: Advances in drug delivery and theranostic applications
inferred← citeshttps://doi.org/10.1016/j.patol.2024.100800openalex-snapshot
Histopathological changes of nervous tissue in women over 60 years of age with Alzheimer's disease and their relationship with menopause
inferred← citeshttps://doi.org/10.1016/j.phrs.2024.107101openalex-snapshot
The VEGFs/VEGFRs system in Alzheimer’s and Parkinson’s diseases: Pathophysiological roles and therapeutic implications
inferred← citeshttps://doi.org/10.1016/j.phymed.2026.157813openalex-snapshot
Unraveling the Anti-Alzheimer's mechanisms of optimal constituent from Arisaema heterophyllum Blume through multi-model and phytochemical analysis
inferred← citeshttps://doi.org/10.1016/j.preteyeres.2023.101217openalex-snapshot
Neurovascular dysfunction in glaucoma
inferred← citeshttps://doi.org/10.1016/j.prp.2025.156109openalex-snapshot
Unravelling the Alzheimer's pathogenesis: Molecular and cellular pathways to neurodegeneration and therapeutic targets
inferred← citeshttps://doi.org/10.1016/j.semcdb.2019.12.014openalex-snapshot
Cerebrospinal fluid and central nervous system development
inferred← citeshttps://doi.org/10.1016/j.trsl.2022.12.003openalex-snapshot
Vascular contributions to Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.yexcr.2018.05.025openalex-snapshot
Aβ1–42 oligomer induces alteration of tight junction scaffold proteins via RAGE-mediated autophagy in bEnd.3 cells
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2021.11.005openalex-snapshot
Computational model of brain endothelial cell signaling pathways predicts therapeutic targets for cerebral pathologies
inferred← citeshttps://doi.org/10.1017/9781009086974.017openalex-snapshot
Supplemental Tables and Figures
inferred← citeshttps://doi.org/10.1017/s0033291720000586openalex-snapshot
Migration and dementia: a meta-analysis of epidemiological studies in Europe
inferred← citeshttps://doi.org/10.1021/acsami.1c18465openalex-snapshot
Generation of Interconnected Neural Clusters in Multiscale Scaffolds from Human-Induced Pluripotent Stem Cells
inferred← citeshttps://doi.org/10.1021/acsami.8b03962openalex-snapshot
Establishment of a Human iPSC- and Nanofiber-Based Microphysiological Blood–Brain Barrier System
inferred← citeshttps://doi.org/10.1021/acschemneuro.4c00326openalex-snapshot
The Usnic Acid Analogue 4-FPBUA Enhances the Blood–Brain Barrier Function and Induces Autophagy in Alzheimer’s Disease Mouse Models
inferred← citeshttps://doi.org/10.1038/s41371-020-0352-2openalex-snapshot
Blood–brain barrier dysfunction: the undervalued frontier of hypertension
inferred← citeshttps://doi.org/10.1038/s41392-025-02426-1openalex-snapshot
Rapid amyloid-β clearance and cognitive recovery through multivalent modulation of blood–brain barrier transport
inferred← citeshttps://doi.org/10.1038/s41398-020-01102-yopenalex-snapshot
Correction: Correlation between cognition and plasma noradrenaline level in Alzheimer’s disease: a potential new blood marker of disease evolution
inferred← citeshttps://doi.org/10.1038/s41398-020-0841-7openalex-snapshot
Correlation between cognition and plasma noradrenaline level in Alzheimer’s disease: a potential new blood marker of disease evolution
inferred← citeshttps://doi.org/10.1038/s41419-024-06804-5openalex-snapshot
Mitophagy-related regulated cell death: molecular mechanisms and disease implications
inferred← citeshttps://doi.org/10.1038/s41467-026-71379-yopenalex-snapshot
Intraocular pressure induced blood retinal barrier compromise in mouse models and human glaucoma
inferred← citeshttps://doi.org/10.1038/s41536-023-00311-5openalex-snapshot
Nerve growth factor receptor (Ngfr) induces neurogenic plasticity by suppressing reactive astroglial Lcn2/Slc22a17 signaling in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1038/s41598-019-42439-9openalex-snapshot
Assembly of Human Stem Cell-Derived Cortical Spheroids and Vascular Spheroids to Model 3-D Brain-like Tissues
inferred← citeshttps://doi.org/10.1038/s41598-021-84730-8openalex-snapshot
Blast-induced temporal alterations in blood–brain barrier properties in a rodent model
inferred← citeshttps://doi.org/10.1038/s41598-025-95183-8openalex-snapshot
Exome sequencing of a Portuguese cohort of early-onset Alzheimer’s disease implicates the X-linked lysosomal gene GLA
inferred← citeshttps://doi.org/10.1038/s41598-025-99364-3openalex-snapshot
Optimized enrichment of murine blood–brain barrier vessels with a critical focus on network hierarchy in post-collection analysis
inferred← citeshttps://doi.org/10.1038/s42003-024-06591-xopenalex-snapshot
Exploring cross-tissue DNA methylation patterns: blood–brain CpGs as potential neurodegenerative disease biomarkers
inferred← citeshttps://doi.org/10.1038/s43587-026-01204-0openalex-snapshot
Fibronectin mediates APOE4-driven blood–brain barrier dysfunction in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1039/d1fo01755aopenalex-snapshot
Safflower leaf ameliorates cognitive impairment through moderating excessive astrocyte activation in APP/PS1 mice
inferred← citeshttps://doi.org/10.1039/d2lc00305hopenalex-snapshot
Blood–brain barrier (BBB)-on-a-chip: a promising breakthrough in brain disease research
inferred← citeshttps://doi.org/10.1042/bsr20193629openalex-snapshot
Peripheral inflammation promotes brain tau transmission via disrupting blood–brain barrier
inferred← citeshttps://doi.org/10.1073/pnas.1811172115openalex-snapshot
Blood-derived plasminogen drives brain inflammation and plaque deposition in a mouse model of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1080/10717544.2020.1762262openalex-snapshot
Brain delivery of quercetin-loaded exosomes improved cognitive function in AD mice by inhibiting phosphorylated tau-mediated neurofibrillary tangles
inferred← citeshttps://doi.org/10.1080/10717544.2022.2094501openalex-snapshot
Nanoparticles-based anti-aging treatment of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1080/14737175.2025.2500752openalex-snapshot
Uncertainties in anti-amyloid monoclonal antibody therapy for Alzheimer’s disease: the challenges ahead
inferred← citeshttps://doi.org/10.1080/14787210.2021.1925540openalex-snapshot
Possible effects of Porphyromonas gingivalis on the blood–brain barrier in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1080/17425247.2019.1583205openalex-snapshot
Blood-brain barrier receptors and transporters: an insight on their function and how to exploit them through nanotechnology
inferred← citeshttps://doi.org/10.1080/26895293.2025.2543709openalex-snapshot
Research progress in mechanisms of neurotoxicity induced by micro(nano)plastic exposure
inferred← citeshttps://doi.org/10.1089/ars.2022.0107openalex-snapshot
Advances in Antioxidant Nanomedicines for Imaging and Therapy of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1089/ten.teb.2020.0091openalex-snapshot
Engineering Brain-Specific Pericytes from Human Pluripotent Stem Cells
inferred← citeshttps://doi.org/10.1093/abbs/gmaa002openalex-snapshot
Inhibition of glycogen synthase kinase 3β improves cognitive function in aged mice by upregulating claudin presences in cerebral endothelial cells
inferred← citeshttps://doi.org/10.1093/brain/awaa325openalex-snapshot
Pathophysiological subtypes of Alzheimer’s disease based on cerebrospinal fluid proteomics
inferred← citeshttps://doi.org/10.1093/brain/awac236openalex-snapshot
Activation of Wnt/β-catenin pathway mitigates blood–brain barrier dysfunction in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1093/brain/awae156openalex-snapshot
The vascular contribution of apolipoprotein E to Alzheimer’s disease
inferred← citeshttps://doi.org/10.1093/brain/awz011openalex-snapshot
Selective loss of cortical endothelial tight junction proteins during Alzheimer’s disease progression
inferred← citeshttps://doi.org/10.1093/braincomms/fcac288openalex-snapshot
Ependymal cells and neurodegenerative disease: outcomes of compromised ependymal barrier function
inferred← citeshttps://doi.org/10.1093/jnen/nlaa019openalex-snapshot
Elevated Cerebrospinal Fluid and Plasma N-Cadherin in Alzheimer Disease
inferred← citeshttps://doi.org/10.1097/wad.0000000000000420openalex-snapshot
β-Amyloid Orchestrates Factor XII and Platelet Activation Leading to Endothelial Dysfunction and Abnormal Fibrinolysis in Alzheimer Disease
inferred← citeshttps://doi.org/10.1098/rsob.200286openalex-snapshot
Alzheimer's disease-like perturbations in HIV-mediated neuronal dysfunctions: understanding mechanisms and developing therapeutic strategies
inferred← citeshttps://doi.org/10.1101/2020.03.25.20043158openalex-snapshot
Pathophysiological subtypes of Alzheimer’s disease based on cerebrospinal fluid proteomics
inferred← citeshttps://doi.org/10.1101/2021.03.27.437282openalex-snapshot
In vivo dissection of Rhoa function in vascular development using zebrafish
inferred← citeshttps://doi.org/10.1101/2024.06.11.24308689openalex-snapshot
Aquaporin-4 as an early cerebrospinal fluid biomarker of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1101/2025.01.24.634732openalex-snapshot
APOE- ε4-induced Fibronectin at the blood-brain barrier is a conserved pathological mediator of disrupted astrocyte-endothelia interaction in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1101/2025.09.02.673886openalex-snapshot
Predicting emergent phenotypes from single cell populations using CELLECTION
inferred← citeshttps://doi.org/10.1101/768267openalex-snapshot
Claudin-12 deficiency causes nerve barrier breakdown, mechanical hypersensitivity and painfulness in polyneuropathy
inferred← citeshttps://doi.org/10.1109/iccasit55263.2022.9986646openalex-snapshot
Study on blood vessel segmentation of laser scattering microscopic image
inferred← citeshttps://doi.org/10.1111/acel.70719openalex-snapshot
Rethinking Senescence Hallmarks in the Brain: Lessons From Peripheral Tissues and Challenges in Defining Neuronal Senescence
inferred← citeshttps://doi.org/10.1111/apha.13642openalex-snapshot
Cognitive impairment in myocardial infarction and heart failure
inferred← citeshttps://doi.org/10.1111/bpa.12895openalex-snapshot
Distinct changes in all major components of the neurovascular unit across different neuropathological stages of Alzheimer's disease
inferred← citeshttps://doi.org/10.1111/bpa.13061openalex-snapshot
Pathological changes within the cerebral vasculature in Alzheimer’s disease: New perspectives
inferred← citeshttps://doi.org/10.1111/bpa.13097openalex-snapshot
Evidence of beta amyloid independent small vessel disease in familial Alzheimer's disease
inferred← citeshttps://doi.org/10.1111/bpa.13270openalex-snapshot
Decreased microvascular claudin‐5 levels in cerebral amyloid angiopathy associated with intracerebral haemorrhage
inferred← citeshttps://doi.org/10.1111/bph.16243openalex-snapshot
Adipose tissue as a therapeutic target for vascular damage in Alzheimer's disease
inferred← citeshttps://doi.org/10.1111/jnc.15819openalex-snapshot
Brain iron acquisition: An overview of homeostatic regulation and disease dysregulation
inferred← citeshttps://doi.org/10.1111/micc.70079openalex-snapshot
Beyond Viral Control: Tenofovir's Hidden Impact on Blood–Brain Barrier Endothelial Cells
inferred← citeshttps://doi.org/10.1111/neup.13021openalex-snapshot
Blood–brain barrier dysfunction in multiple system atrophy: A human postmortem study
inferred← citeshttps://doi.org/10.1111/nyas.13998openalex-snapshot
Emerging insights from the genetics of cerebral small‐vessel disease
inferred← citeshttps://doi.org/10.1117/12.2524427openalex-snapshot
Optical monitoring of the meningeal lymphatic clearance after opening of blood-brain barrier
inferred← citeshttps://doi.org/10.1124/jpet.124.002280openalex-snapshot
Molecular Mechanisms Underlying Amyloid Beta Peptide Mediated Upregulation of Vascular Cell Adhesion Molecule-1 in Alzheimer Disease
inferred← citeshttps://doi.org/10.1134/s1990750821030057openalex-snapshot
Features of the In Vitro Expression Profile of Hippocampal Neurogenic Niche Cells during Optogenetic Stimulation
inferred← citeshttps://doi.org/10.1136/bmj.n1954openalex-snapshot
Residential exposure to transportation noise in Denmark and incidence of dementia: national cohort study
inferred← citeshttps://doi.org/10.1152/physrev.00040.2024openalex-snapshot
Neuropathological links between T2DM and LOAD: systematic review and meta-analysis
inferred← citeshttps://doi.org/10.1155/2017/7905486openalex-snapshot
P‐glycoprotein (ABCB1) and Oxidative Stress: Focus on Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1155/2021/1995766openalex-snapshot
Identification of Chemical Components of Qi-Fu-Yin and Its Prototype Components and Metabolites in Rat Plasma and Cerebrospinal Fluid via UPLC-Q-TOF-MS
inferred← citeshttps://doi.org/10.1155/2021/6521146openalex-snapshot
DL0410 Alleviates Memory Impairment in D‐Galactose‐Induced Aging Rats by Suppressing Neuroinflammation via the TLR4/MyD88/NF‐κB Pathway
inferred← citeshttps://doi.org/10.1161/circresaha.122.319951openalex-snapshot
Post-Stroke Cognitive Impairment and Dementia
inferred← citeshttps://doi.org/10.1161/circulationaha.125.077356openalex-snapshot
Apolipoprotein D, a Novel Ligand for CD36, Is Essential for Blood–Brain Barrier Integrity
inferred← citeshttps://doi.org/10.1161/strokeaha.120.031611openalex-snapshot
Cognitive Impairment and Dementia After Stroke: Design and Rationale for the DISCOVERY Study
inferred← citeshttps://doi.org/10.1172/jci.insight.163822openalex-snapshot
LRP1 in vascular mural cells modulates cerebrovascular integrity and function in the presence of APOE4
inferred← citeshttps://doi.org/10.1177/0004867418796955openalex-snapshot
Leaky brain in neurological and psychiatric disorders: Drivers and consequences
inferred← citeshttps://doi.org/10.1177/0271678x231180035openalex-snapshot
Blood-brain barrier breakdown is linked to tau pathology and neuronal injury in a differential manner according to amyloid deposition
inferred← citeshttps://doi.org/10.1177/03601293261470424openalex-snapshot
Acupuncture at Jing-Well Points in Neurological Disorders: Clinical Efficacy, Mechanisms, and Future Directions
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