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C-Abl as a modulator of p53

https://doi.org/10.1016/j.bbrc.2005.03.152

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/ijc.10580openalex-snapshot

Escape of p53 protein from E6‐mediated degradation in HeLa cells after cisplatin therapy

inferredcites →https://doi.org/10.1002/ijc.11023openalex-snapshot

Refined mapping of 1q32 amplicons in malignant gliomas confirms MDM4 as the main amplification target

inferredcites →https://doi.org/10.1002/j.1460-2075.1996.tb00503.xopenalex-snapshot

The cytostatic function of c‐Abl is controlled by multiple nuclear localization signals and requires the p53 and Rb tumor suppressor gene products.

inferredcites →https://doi.org/10.1002/j.1460-2075.1996.tb00919.xopenalex-snapshot

MDMX: a novel p53‐binding protein with some functional properties of MDM2.

inferredcites →https://doi.org/10.1002/jcb.10535openalex-snapshot

Identification of a domain within MDMX‐S that is responsible for its high affinity interaction with p53 and high‐level expression in mammalian cells

inferredcites →https://doi.org/10.1002/jcb.10769openalex-snapshot

Cisplatin restores p53 function and enhances the radiosensitivity in HPV16 E6 containing SiHa cells

inferredcites →https://doi.org/10.1002/stem.5530130416openalex-snapshot

Role of p53 in leukemogenesis of chronic myeloid leukemia

inferredcites →https://doi.org/10.1006/geno.1997.4775openalex-snapshot

Isolation and Identification of the Human Homolog of a New p53-Binding Protein, Mdmx

inferredcites →https://doi.org/10.1006/scbi.2001.0387openalex-snapshot

Molecular evolution of chronic myeloid leukaemia

inferredcites →https://doi.org/10.1016/0092-8674(93)90384-3openalex-snapshot

The HPV-16 E6 and E6-AP complex functions as a ubiquitin-protein ligase in the ubiquitination of p53

inferredcites →https://doi.org/10.1016/0092-8674(93)90689-nopenalex-snapshot

BCR-ABL-induced oncogenesis is mediated by direct interaction with the SH2 domain of the GRB-2 adaptor protein

inferredcites →https://doi.org/10.1016/0092-8674(94)90240-2openalex-snapshot

The nuclear tyrosine kinase c-abl negatively regulates cell growth

inferredcites →https://doi.org/10.1016/j.cell.2004.06.016openalex-snapshot

Mdm2-Mediated NEDD8 Conjugation of p53 Inhibits Its Transcriptional Activity

inferredcites →https://doi.org/10.1016/j.dnarep.2004.03.027openalex-snapshot

The p53 response to DNA damage

inferredcites →https://doi.org/10.1016/j.dnarep.2004.03.033openalex-snapshot

The Chk2 protein kinase

inferredcites →https://doi.org/10.1016/j.jmb.2004.07.042openalex-snapshot

Regulation of DNA Binding of p53 by its C-terminal Domain

inferredcites →https://doi.org/10.1016/j.semcancer.2004.04.003openalex-snapshot

Manipulation of the tumor suppressor p53 for potentiating cancer therapy

inferredcites →https://doi.org/10.1016/j.tcb.2003.11.003openalex-snapshot

How do Abl family kinases regulate cell shape and movement?

inferredcites →https://doi.org/10.1016/s0014-5793(99)00254-9openalex-snapshot

MDM2 interacts with MDMX through their RING finger domains

inferredcites →https://doi.org/10.1016/s0070-2153(04)59007-5openalex-snapshot

Death by Abl: A Matter of Location

inferredcites →https://doi.org/10.1016/s0092-8674(00)80574-7openalex-snapshot

A Novel Ubiquitination Factor, E4, Is Involved in Multiubiquitin Chain Assembly

inferredcites →https://doi.org/10.1016/s0092-8674(02)01199-6openalex-snapshot

Crystal Structure of a UBP-Family Deubiquitinating Enzyme in Isolation and in Complex with Ubiquitin Aldehyde

inferredcites →https://doi.org/10.1016/s0092-8674(03)00193-4openalex-snapshot

Pirh2, a p53-Induced Ubiquitin-Protein Ligase, Promotes p53 Degradation

inferredcites →https://doi.org/10.1016/s0092-8674(03)00194-6openalex-snapshot

Structural Basis for the Autoinhibition of c-Abl Tyrosine Kinase

inferredcites →https://doi.org/10.1016/s0378-1119(99)00487-4openalex-snapshot

MDM2 — master regulator of the p53 tumor suppressor protein

inferredcites →https://doi.org/10.1016/s0950-3536(97)80004-2openalex-snapshot

3 Signal transduction pathways involved in BCR-ABL transformation

inferredcites →https://doi.org/10.1016/s0959-8049(03)00552-5openalex-snapshot

Frequent methylation of p16INK4A and p14ARF genes implicated in the evolution of chronic myeloid leukaemia from its chronic to accelerated phase

inferredcites →https://doi.org/10.1016/s1044-579x(02)00099-8openalex-snapshot

The p53–Mdm2 module and the ubiquitin system

inferredcites →https://doi.org/10.1016/s1097-2765(04)00157-1openalex-snapshot

A Dynamic Role of HAUSP in the p53-Mdm2 Pathway

inferredcites →https://doi.org/10.1016/s1535-6108(02)00047-8openalex-snapshot

Dissecting p53 tumor suppressor functions in vivo

inferredcites →https://doi.org/10.1016/s1535-6108(03)00020-5openalex-snapshot

BCR/ABL activates mdm2 mRNA translation via the La antigen

inferredcites →https://doi.org/10.1016/s1535-6108(03)00110-7openalex-snapshot

Chk1 and Chk2 kinases in checkpoint control and cancer

inferredcites →https://doi.org/10.1021/bi0271291openalex-snapshot

Overexpression of Mdm2 and MdmX Fusion Proteins Alters p53 Mediated Transactivation, Ubiquitination, and Degradation

inferredcites →https://doi.org/10.1038/21690openalex-snapshot

The tyrosine kinase c-Abl regulates p73 in apoptotic response to cisplatin-induced DNA damage

inferredcites →https://doi.org/10.1038/21697openalex-snapshot

Interaction of c-Abl and p73α and their collaboration to induce apoptosis

inferredcites →https://doi.org/10.1038/21704openalex-snapshot

p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage

inferredcites →https://doi.org/10.1038/35042675openalex-snapshot

Surfing the p53 network

inferredcites →https://doi.org/10.1038/35103059openalex-snapshot

Chk2 kinase — a busy messenger

inferredcites →https://doi.org/10.1038/358080a0openalex-snapshot

Amplification of a gene encoding a p53-associated protein in human sarcomas

inferredcites →https://doi.org/10.1038/376785a0openalex-snapshot

Activation of the c-Abl tyrosine kinase in the stress response to DMA-damaging agents

inferredcites →https://doi.org/10.1038/378203a0openalex-snapshot

Rescue of early embryonic lethality in mdm2-deficient mice by deletion of p53

inferredcites →https://doi.org/10.1038/378206a0openalex-snapshot

Rescue of embryonic lethality in Mdm2-deficient mice by absence of p53

inferredcites →https://doi.org/10.1038/382272a0openalex-snapshot

Role for c-Abl tyrosine kinase in growth arrest response to DNA damage

inferredcites →https://doi.org/10.1038/384273a0openalex-snapshot

Three distinct signalling responses by murine fibroblasts to genotoxic stress

inferredcites →https://doi.org/10.1038/387516a0openalex-snapshot

Ataxia telangiectasia mutant protein activates c-Abl tyrosine kinase in response to ionizing radiation

inferredcites →https://doi.org/10.1038/387520a0openalex-snapshot

Interaction between ATM protein and c-Abl in response to DNA damage

inferredcites →https://doi.org/10.1038/416560aopenalex-snapshot

p63 and p73 are required for p53-dependent apoptosis in response to DNA damage

inferredcites →https://doi.org/10.1038/nature01120openalex-snapshot

The prolyl isomerase Pin1 reveals a mechanism to control p53 functions after genotoxic insults

inferredcites →https://doi.org/10.1038/nature02514openalex-snapshot

The ubiquitin ligase COP1 is a critical negative regulator of p53

inferredcites →https://doi.org/10.1038/nature737openalex-snapshot

Deubiquitination of p53 by HAUSP is an important pathway for p53 stabilization

inferredcites →https://doi.org/10.1038/ncb0104-3openalex-snapshot

Controlling Abl: auto-inhibition and co-inhibition?

inferredcites →https://doi.org/10.1038/ng714openalex-snapshot

Rescue of embryonic lethality in Mdm4-null mice by loss of Trp53 suggests a nonoverlapping pathway with MDM2 to regulate p53

inferredcites →https://doi.org/10.1038/sj.cdd.4400626openalex-snapshot

c-Abl: activation and nuclear targets

inferredcites →https://doi.org/10.1038/sj.leu.2402309openalex-snapshot

Elevated Bcr-Abl expression levels are sufficient for a haematopoietic cell line to acquire a drug-resistant phenotype

inferredcites →https://doi.org/10.1038/sj.onc.1201376openalex-snapshot

Pro-apoptotic effect of the c-Abl tyrosine kinase in the cellular response to 1-β-D-arabinofuranosylcytosine

inferredcites →https://doi.org/10.1038/sj.onc.1202281openalex-snapshot

Comparative study of the p53-mdm2 and p53-MDMX interfaces

inferredcites →https://doi.org/10.1038/sj.onc.1203130openalex-snapshot

The cellular response to p53: the decision between life and death

inferredcites →https://doi.org/10.1038/sj.onc.1203290openalex-snapshot

Inhibition of v-Abl transformation by p53 and p19ARF

inferredcites →https://doi.org/10.1038/sj.onc.1203878openalex-snapshot

Regulation of cell death by the Abl tyrosine kinase

inferredcites →https://doi.org/10.1038/sj.onc.1204454openalex-snapshot

A sequence element of p53 that determines its susceptibility to viral oncoprotein-targeted degradation

inferredcites →https://doi.org/10.1038/sj.onc.1205137openalex-snapshot

MDMX stability is regulated by p53-induced caspase cleavage in NIH3T3 mouse fibroblasts

inferredcites →https://doi.org/10.1038/sj.onc.1205239openalex-snapshot

Role of MAP kinases in UVB-induced phosphorylation of p53 at serine 20

inferredcites →https://doi.org/10.1038/sj.onc.1206087openalex-snapshot

BCR/ABL regulates response to DNA damage: the role in resistance to genotoxic treatment and in genomic instability

inferredcites →https://doi.org/10.1042/bj20021539openalex-snapshot

Cbl-ArgBP2 complex mediates ubiquitination and degradation of c-Abl

inferredcites →https://doi.org/10.1073/pnas.2030930100openalex-snapshot

HdmX stimulates Hdm2-mediated ubiquitination and degradation of p53

inferredcites →https://doi.org/10.1073/pnas.88.14.6293openalex-snapshot

p53 in chronic myelogenous leukemia in acute phase.

inferredcites →https://doi.org/10.1073/pnas.94.4.1437openalex-snapshot

Regulation of DNA damage-induced apoptosis by the c-Abl tyrosine kinase

inferredcites →https://doi.org/10.1073/pnas.95.14.8292openalex-snapshot

Functional and physical interactions of the ARF tumor suppressor with p53 and Mdm2

inferredcites →https://doi.org/10.1073/pnas.95.22.13194openalex-snapshot

p19 Arf induces p53-dependent apoptosis during Abelson virus-mediated pre-B cell transformation

inferredcites →https://doi.org/10.1073/pnas.96.24.13777openalex-snapshot

Phosphorylation of Ser-20 mediates stabilization of human p53 in response to DNA damage

inferredcites →https://doi.org/10.1073/pnas.96.26.14973openalex-snapshot

Rapid ATM-dependent phosphorylation of MDM2 precedes p53 accumulation in response to DNA damage

inferredcites →https://doi.org/10.1073/pnas.97.10.5486openalex-snapshot

c-Abl is required for development and optimal cell proliferation in the context of p53 deficiency

inferredcites →https://doi.org/10.1074/jbc.271.43.26457openalex-snapshot

Genotoxic Drugs Induce Interaction of the c-Abl Tyrosine Kinase and the Tumor Suppressor Protein p53

inferredcites →https://doi.org/10.1074/jbc.272.28.17542openalex-snapshot

ArgBP2, a Multiple Src Homology 3 Domain-containing, Arg/Abl-interacting Protein, Is Phosphorylated in v-Abl-transformed Cells and Localized in Stress Fibers and Cardiocyte Z-disks

inferredcites →https://doi.org/10.1074/jbc.274.13.8371openalex-snapshot

c-Abl Neutralizes the Inhibitory Effect of Mdm2 on p53

inferredcites →https://doi.org/10.1074/jbc.274.53.38189openalex-snapshot

Stabilization of the MDM2 Oncoprotein by Interaction with the Structurally Related MDMX Protein

inferredcites →https://doi.org/10.1074/jbc.c200150200openalex-snapshot

Mutual Dependence of MDM2 and MDMX in Their Functional Inactivation of p53

inferredcites →https://doi.org/10.1074/jbc.m003496200openalex-snapshot

Hdmx Stabilizes Mdm2 and p53

inferredcites →https://doi.org/10.1074/jbc.m101414200openalex-snapshot

Targeting of the c-Abl Tyrosine Kinase to Mitochondria in the Necrotic Cell Death Response to Oxidative Stress

inferredcites →https://doi.org/10.1074/jbc.m108795200openalex-snapshot

Hdmx Recruitment into the Nucleus by Hdm2 Is Essential for Its Ability to Regulate p53 Stability and Transactivation

inferredcites →https://doi.org/10.1074/jbc.m213034200openalex-snapshot

Hdmx Protein Stability Is Regulated by the Ubiquitin Ligase Activity of Mdm2

inferredcites →https://doi.org/10.1074/jbc.m301051200openalex-snapshot

c-Abl Tyrosine Kinase Selectively Regulates p73 Nuclear Matrix Association

inferredcites →https://doi.org/10.1074/jbc.m308295200openalex-snapshot

DNA Damage-induced MDMX Degradation Is Mediated by MDM2

inferredcites →https://doi.org/10.1074/jbc.m311793200openalex-snapshot

MDM4 (MDMX) Overexpression Enhances Stabilization of Stress-induced p53 and Promotes Apoptosis

inferredcites →https://doi.org/10.1089/104454902320219077openalex-snapshot

MDMX Inhibits the p300/CBP-Mediated Acetylation of p53

inferredcites →https://doi.org/10.1093/embo-reports/kve227openalex-snapshot

Mdmx stabilizes p53 and Mdm2 via two distinct mechanisms

inferredcites →https://doi.org/10.1093/emboj/18.7.1805openalex-snapshot

Critical role for Ser20 of human p53 in the negative regulation of p53 by Mdm2

inferredcites →https://doi.org/10.1093/emboj/cdf384openalex-snapshot

Tyrosine phosphorylation of Mdm2 by c‐Abl: implications for p53 regulation

inferredcites →https://doi.org/10.1101/gad.14.3.278openalex-snapshot

Chk2/hCds1 functions as a DNA damage checkpoint in G1by stabilizing p53

inferredcites →https://doi.org/10.1101/gad.14.3.289openalex-snapshot

The human homologs of checkpoint kinases Chk1 and Cds1 (Chk2) phosphorylate p53 at multiple DNA damage-inducible sites

inferredcites →https://doi.org/10.1101/gad.886901openalex-snapshot

ATM-dependent phosphorylation of Mdm2 on serine 395: role in p53 activation by DNA damage

inferredcites →https://doi.org/10.1101/gad.9.17.2170openalex-snapshot

Induction of apoptosis in HeLa cells by trans-activation-deficient p53.

inferredcites →https://doi.org/10.1126/science.1080386openalex-snapshot

Polyubiquitination of p53 by a Ubiquitin Ligase Activity of p300

inferredcites →https://doi.org/10.1126/science.1090072openalex-snapshot

p53- and Drug-Induced Apoptotic Responses Mediated by BH3-Only Proteins Puma and Noxa

inferredcites →https://doi.org/10.1126/science.1091362openalex-snapshot

Mono- Versus Polyubiquitination: Differential Control of p53 Fate by Mdm2

inferredcites →https://doi.org/10.1126/science.287.5459.1824openalex-snapshot

DNA Damage-Induced Activation of p53 by the Checkpoint Kinase Chk2

inferredcites →https://doi.org/10.1128/jvi.70.7.4509-4516.1996openalex-snapshot

High-risk human papillomavirus E6 protein has two distinct binding sites within p53, of which only one determines degradation

inferredcites →https://doi.org/10.1128/mcb.13.12.7587openalex-snapshot

A Coiled-Coil Oligomerization Domain of Bcr Is Essential for the Transforming Function of Bcr-Abl Oncoproteins

inferredcites →https://doi.org/10.1128/mcb.18.12.7288openalex-snapshot

Nuclear Export Is Required for Degradation of Endogenous p53 by MDM2 and Human Papillomavirus E6

inferredcites →https://doi.org/10.1128/mcb.20.22.8458-8467.2000openalex-snapshot

Multiple C-Terminal Lysine Residues Target p53 for Ubiquitin-Proteasome-Mediated Degradation

inferredcites →https://doi.org/10.1128/mcb.20.3.1001-1007.2000openalex-snapshot

MdmX Protects p53 from Mdm2-Mediated Degradation

inferredcites →https://doi.org/10.1128/mcb.20.3.741-748.2000openalex-snapshot

Stimulation of p53 DNA Binding by c-Abl Requires the p53 C Terminus and Tetramerization

inferredcites →https://doi.org/10.1128/mcb.21.17.5869-5878.2001openalex-snapshot

c-Abl Regulates p53 Levels under Normal and Stress Conditions by Preventing Its Nuclear Export and Ubiquitination

inferredcites →https://doi.org/10.1128/mcb.21.18.6233-6242.2001openalex-snapshot

Targeting of the c-Abl Tyrosine Kinase to Mitochondria in Endoplasmic Reticulum Stress-Induced Apoptosis

inferredcites →https://doi.org/10.1128/mcb.22.15.5527-5538.2002openalex-snapshot

Mdm4 (Mdmx) Regulates p53-Induced Growth Arrest and Neuronal Cell Death during Early Embryonic Mouse Development

inferredcites →https://doi.org/10.1128/mcb.22.21.7562-7571.2002openalex-snapshot

DNA Damage Induces MDMX Nuclear Translocation by p53-Dependent and -Independent Mechanisms

inferredcites →https://doi.org/10.1128/mcb.24.13.5835-5843.2004openalex-snapshot

Amplification of Mdmx (or Mdm4 ) Directly Contributes to Tumor Formation by Inhibiting p53 Tumor Suppressor Activity

inferredcites →https://doi.org/10.1128/mcb.24.15.6728-6741.2004openalex-snapshot

In Vivo Mitochondrial p53 Translocation Triggers a Rapid First Wave of Cell Death in Response to DNA Damage That Can Precede p53 Target Gene Activation

inferredcites →https://doi.org/10.1128/mmbr.68.2.362-372.2004openalex-snapshot

Pathogenesis of Human Papillomaviruses in Differentiating Epithelia

inferredcites →https://doi.org/10.1146/annurev.immunol.22.012703.104753openalex-snapshot

The BCR-ABL Story: Bench to Bedside and Back

inferredcites →https://doi.org/10.1172/jci115234openalex-snapshot

The spectrum of molecular alterations in the evolution of chronic myelocytic leukemia.

inferredcites →https://doi.org/10.1182/asheducation-2002.1.111openalex-snapshot

Chronic Myelogenous Leukemia

inferredcites →https://doi.org/10.1182/blood.v96.10.3343openalex-snapshot

The molecular biology of chronic myeloid leukemia

inferredcites →https://doi.org/10.1182/blood.v99.10.3530openalex-snapshot

Imatinib induces hematologic and cytogenetic responses in patients with chronic myelogenous leukemia in myeloid blast crisis: results of a phase II study

inferredcites →https://doi.org/10.1182/blood.v99.6.1928openalex-snapshot

Imatinib induces durable hematologic and cytogenetic responses in patients with accelerated phase chronic myeloid leukemia: results of a phase 2 study

inferredcites →https://doi.org/10.1182/blood.v99.8.2957openalex-snapshot

The coiled-coil domain and Tyr177 of bcr are required to induce a murine chronic myelogenous leukemia–like disease by bcr/abl

inferredcites →https://doi.org/10.1242/jcs.00739openalex-snapshot

Apoptosis - the p53 network

inferredcites →https://doi.org/10.4161/cc.1.6.271openalex-snapshot

Genotoxic Stress-Induced Activation of Plk3 is Partly Mediated by Chk2

inferredcites →https://doi.org/10.4161/cc.3.1.603openalex-snapshot

The Development of a CDK2-Docking Site Peptide that Inhibits p53 and Sensitizes Cells to Death

inferredcites →https://doi.org/10.4161/cc.3.11.1229openalex-snapshot

BCR/ABL Recruits p53 Tumor Suppressor Protein to Induce Drug Resistance

inferredcites →https://doi.org/10.4161/cc.3.4.821openalex-snapshot

Cytoplasmic p53: Bax and Forward

inferredcites →https://doi.org/10.4161/cc.3.6.924openalex-snapshot

HAUSP is Required for p53 Destabilization

inferredcites →https://doi.org/10.4161/cc.3.7.956openalex-snapshot

p53 Moves to Mitochondria: A Turn on the Path to Apoptosis

inferredcites →https://doi.org/10.4161/cc.3.7.996openalex-snapshot

p73 and p63: Why Do We Still Need Them?

inferredcites →https://doi.org/10.4161/cc.3.7.997openalex-snapshot

Dynamics in the p53-Mdm2 Ubiquitination Pathway

inferredcites →https://doi.org/10.4161/cc.3.7.998openalex-snapshot

Mdmx and Mdm2: Brothers in Arms?

inferredcites →https://doi.org/10.4161/cc.3.9.1126openalex-snapshot

Treatment of Chronic Myeloid Leukemia Cells with Imatinib (STI571) Impairs p53 Accumulation in Response to DNA Damage

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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c‐Abl kinase regulates cell proliferation and ionizing radiation‐induced G2/M arrest via phosphorylation of FHL2

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The genetics of chronic myelogenous leukaemia

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Cancer models in Caenorhabditis elegans

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A case of ABL deletion in a patient with precursor B cell lymphoblastic leukemia

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Involvement of c-Abl, p53 and the MAP kinase JNK in the cell death program initiated in A2E-laden ARPE-19 cells by exposure to blue light

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Regulation of apoptosis by Caspases under oxidative stress conditions in mice testicular cells: in vitro molecular mechanism

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The Role of p53 Protein in the Regulation of Neuronal Differentiation

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Abnormal cell-cycle expression of the proteins p27, mdm2 and cathepsin B in oral squamous-cell carcinoma infected with human papillomavirus

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p53 in apoptosis control: An introduction

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HCT116 cells deficient in p21Waf1 are hypersensitive to tyrosine kinase inhibitors and adriamycin through a mechanism unrelated to p21 and dependent on p53

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Detection of ABL1 deletion without BCR-ABL rearrangement in ETP-ALL

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Role of Peroxiredoxin I in Rectal Cancer and Related to p53 Status

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Met acts through Abl to regulate p53 transcriptional outcomes and cell survival in the developing liver

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Clonal hematopoiesis in Philadelphia chromosome-negative bone marrow cells of chronic myeloid leukemia patients receiving dasatinib

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c-Abl Phosphorylates E6AP and Regulates Its E3 Ubiquitin Ligase Activity

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Mutant p53 dictates the oncogenic activity of c-Abl in triple-negative breast cancers

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Role of ABL family kinases in cancer: from leukaemia to solid tumours

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The Ron receptor tyrosine kinase activates c-Abl to promote cell proliferation through tyrosine phosphorylation of PCNA in breast cancer

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Controlling Directed Protein Interaction Networks in Cancer

inferred← citeshttps://doi.org/10.1038/sj.cdd.4402270openalex-snapshot

Identification of a novel p53-dependent activation pathway of STAT1 by antitumour genotoxic agents

inferred← citeshttps://doi.org/10.1038/sj.onc.1209569openalex-snapshot

Functional connection between p53 and caspase-2 is essential for apoptosis induced by DNA damage

inferred← citeshttps://doi.org/10.1074/jbc.m110.183012openalex-snapshot

c-Abl Phosphorylation of Mdm2 Facilitates Mdm2-Mdmx Complex Formation

inferred← citeshttps://doi.org/10.1074/jbc.m706201200openalex-snapshot

Cooperative Roles of c-Abl and Cdk5 in Regulation of p53 in Response to Oxidative Stress

inferred← citeshttps://doi.org/10.1074/jbc.m809211200openalex-snapshot

c-Abl Phosphorylates Hdmx and Regulates Its Interaction with p53

inferred← citeshttps://doi.org/10.1080/00207454.2022.2158824openalex-snapshot

Implications of p53 in mitochondrial dysfunction and Parkinson’s disease

inferred← citeshttps://doi.org/10.1089/omi.2008.0086openalex-snapshot

Genome-Wide Identification of Genes Required for Yeast Growth Under Imatinib Stress: Vacuolar H + -ATPase Function Is an Important Target of This Anticancer Drug

inferred← citeshttps://doi.org/10.1093/carcin/bgn039openalex-snapshot

p53 codon 72 polymorphism associated with risk of human papillomavirus-associated squamous cell carcinoma of the oropharynx in never-smokers

inferred← citeshttps://doi.org/10.1093/hmg/ddv151openalex-snapshot

FGF8, c-Abl and p300 participate in a pathway that controls stability and function of the ΔNp63αprotein

inferred← citeshttps://doi.org/10.1093/nar/gks866openalex-snapshot

Along signal paths: an empirical gene set approach exploiting pathway topology

inferred← citeshttps://doi.org/10.1111/jcmm.12955openalex-snapshot

Peroxiredoxin 1 – an antioxidant enzyme in cancer

inferred← citeshttps://doi.org/10.1124/mol.106.026575openalex-snapshot

c-Abl Kinase Regulates Curcumin-Induced Cell Death through Activation of c-Jun N-Terminal Kinase

inferred← citeshttps://doi.org/10.1155/2022/6600944openalex-snapshot

Association of p53 with Neurodegeneration in Parkinson’s Disease

inferred← citeshttps://doi.org/10.1186/s12967-024-05623-8openalex-snapshot

The Abl1 tyrosine kinase is a key player in doxorubicin-induced cardiomyopathy and its p53/p73 cell death mediated signaling differs in atrial and ventricular cardiomyocytes

inferred← citeshttps://doi.org/10.1186/s13024-020-00364-wopenalex-snapshot

α-Synucleinopathy associated c-Abl activation causes p53-dependent autophagy impairment

inferred← citeshttps://doi.org/10.12659/msm.904888openalex-snapshot

Global Regulation of Differential Gene Expression by c-Abl/Arg Oncogenic Kinases

inferred← citeshttps://doi.org/10.1371/journal.pone.0100960openalex-snapshot

Inhibition of MDM2 by Nilotinib Contributes to Cytotoxicity in Both Philadelphia-Positive and Negative Acute Lymphoblastic Leukemia

inferred← citeshttps://doi.org/10.1517/17460440903052559openalex-snapshot

Implications of endoplasmic reticulum stress, the unfolded protein response and apoptosis for molecular cancer therapy. Part I: targeting p53, Mdm2, GADD153/CHOP, GRP78/BiP and heat shock proteins

inferred← citeshttps://doi.org/10.1667/rr003cc.1openalex-snapshot

p53 and RAD9, the DNA Damage Response, and Regulation of Transcription Networks

inferred← citeshttps://doi.org/10.1677/erc-07-0073openalex-snapshot

The radiation response of hormone-resistant prostate cancer induced by long-term hormone therapy

inferred← citeshttps://doi.org/10.1677/erc.1.01172openalex-snapshot

p53 and breast cancer, an update

inferred← citeshttps://doi.org/10.18632/oncotarget.11909openalex-snapshot

c-Abl inhibits breast cancer tumorigenesis through reactivation of p53-mediated p21 expression

inferred← citeshttps://doi.org/10.2147/ott.s238336openalex-snapshot

Entire ABL1 Gene Deletion Without BCR/ABL1 Rearrangement in a Female Patient with B-Cell Precursor Acute Lymphoblastic Leukemia

inferred← citeshttps://doi.org/10.3324/haematol.2019.219261openalex-snapshot

Combined inhibition of MDM2 and BCR-ABL1 tyrosine kinase targets chronic myeloid leukemia stem/progenitor cells in a murine model

inferred← citeshttps://doi.org/10.3389/fncel.2015.00203openalex-snapshot

Reactive oxygen species trigger motoneuron death in non-cell-autonomous models of ALS through activation of c-Abl signaling

inferred← citeshttps://doi.org/10.3390/cells11111855openalex-snapshot

p66Shc in Cardiovascular Pathology

inferred← citeshttps://doi.org/10.3390/ijms20194791openalex-snapshot

Expression Profile of Cell Cycle-Related Genes in Human Fibroblasts Exposed Simultaneously to Radiation and Simulated Microgravity

inferred← citeshttps://doi.org/10.3390/ijms24054982openalex-snapshot

BRCA Mutations—The Achilles Heel of Breast, Ovarian and Other Epithelial Cancers

inferred← citeshttps://doi.org/10.3816/clk.2007.n.014openalex-snapshot

Gene Expression Profile of Chronic Myeloid Leukemia Innately Resistant to Imatinib

inferred← citeshttps://doi.org/10.4161/cc.7.24.7267openalex-snapshot

T cell survival and function requires the c-Abl tyrosine kinase

inferred← citeshttps://doi.org/10.5282/edoc.16524openalex-snapshot

Eine Methode zur direkten Identifizierung chromosomaler Translokationsbruchpunkte

inferred← citeshttps://doi.org/10.5772/60402openalex-snapshot

Novel Cellular and Molecular Interactions During Limb Development, Revealed from Studies on the Split Hand Foot Congenital Malformation

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