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Interaction of c-Abl and p73α and their collaboration to induce apoptosis
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/j.1460-2075.1994.tb06897.xopenalex-snapshot
Identification of Src, Fyn, Lyn, PI3K and Abl SH3 domain ligands using phage display libraries.
inferredcites →https://doi.org/10.1016/0076-6879(87)54085-xopenalex-snapshot
[19] Rapid and efficient site-specific mutagenesis without phenotypic selection
inferredcites →https://doi.org/10.1016/0092-8674(86)90450-2openalex-snapshot
Alternative splicing of RNAs transcribed from the human abl gene and from the bcr-abl fused gene
inferredcites →https://doi.org/10.1016/s0021-9258(19)51013-4openalex-snapshot
Identification and characterization of Src SH3 ligands from phage-displayed random peptide libraries.
inferredcites →https://doi.org/10.1016/s0092-8674(00)80540-1openalex-snapshot
Monoallelically Expressed Gene Related to p53 at 1p36, a Region Frequently Deleted in Neuroblastoma and Other Human Cancers
inferredcites →https://doi.org/10.1016/s0960-9822(98)70156-3openalex-snapshot
Histone–GFP fusion protein enables sensitive analysis of chromosome dynamics in living mammalian cells
inferredcites →https://doi.org/10.1038/38298openalex-snapshot
p73 is a human p53-related protein that can induce apoptosis
inferredcites →https://doi.org/10.1038/387516a0openalex-snapshot
Ataxia telangiectasia mutant protein activates c-Abl tyrosine kinase in response to ionizing radiation
inferredcites →https://doi.org/10.1038/387520a0openalex-snapshot
Interaction between ATM protein and c-Abl in response to DNA damage
inferredcites →https://doi.org/10.1038/sj.onc.1201263openalex-snapshot
The polyproline region of p53 is required to activate apoptosis but not growth arrest
inferredcites →https://doi.org/10.1038/sj.onc.1202314openalex-snapshot
Mutant p53 gain of function: differential effects of different p53 mutants on resistance of cultured cells to chemotherapy
inferredcites →https://doi.org/10.1038/sj.onc.1202571openalex-snapshot
Determination of cell fate by c-Abl activation in the response to DNA damage
inferredcites →https://doi.org/10.1038/sj.onc.1202577openalex-snapshot
The role of ATM in DNA damage responses and cancer
inferredcites →https://doi.org/10.1073/pnas.90.18.8392openalex-snapshot
Production of high-titer helper-free retroviruses by transient transfection.
inferredcites →https://doi.org/10.1073/pnas.93.26.15335openalex-snapshot
Identification of a novel p53 functional domain that is necessary for efficient growth suppression
inferredcites →https://doi.org/10.1074/jbc.274.13.8371openalex-snapshot
c-Abl Neutralizes the Inhibitory Effect of Mdm2 on p53
inferredcites →https://doi.org/10.1093/emboj/17.16.4668openalex-snapshot
The requirement for the p53 proline‐rich functional domain for mediation of apoptosis is correlated with specific PIG3 gene transactivation and with transcriptional repression
inferredcites →https://doi.org/10.1101/gad.9.21.2569openalex-snapshot
Abi-2, a novel SH3-containing protein interacts with the c-Abl tyrosine kinase and modulates c-Abl transforming activity.
inferredcites →https://doi.org/10.1101/gad.9.21.2583openalex-snapshot
Abl-interactor-1, a novel SH3 protein binding to the carboxy-terminal portion of the Abl protein, suppresses v-abl transforming activity.
inferredcites →https://doi.org/10.1126/science.281.5383.1674openalex-snapshot
Enhanced Phosphorylation of p53 by ATM in Response to DNA Damage
inferredcites →https://doi.org/10.1128/jvi.63.2.919-924.1989openalex-snapshot
Liver-specific expression of hepatitis B virus is determined by the combined action of the core gene promoter and the enhancer
inferredcites →https://doi.org/10.2144/98243bm03openalex-snapshot
Analysis of Cell-Cycle Profiles in Transfected Cells Using a Membrane-Targeted GFP
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4781335openalex-extracted
The Abl1 Tyrosine Kinase and p53 are Key Players in Doxorubicin-Induced Cardiomyopathy and Their Cell Death Signaling Differs in Atrial and Ventricular Cardiomyocytes
inferred← citeshttps://doi.org/10.1002/(sici)1097-4652(200002)182:2<171::aid-jcp5>3.0.co;2-3openalex-snapshot
Role of p53 family members in apoptosis
inferred← citeshttps://doi.org/10.1002/1097-0215(20001001)88:1<66::aid-ijc10>3.0.co;2-yopenalex-snapshot
Differential expression of p73 splice variants and protein in benign and malignant ovarian tumours
inferred← citeshttps://doi.org/10.1002/1098-2744(200010)29:2<67::aid-mc3>3.0.co;2-2openalex-snapshot
Cooperation between Ha-ras andfos or transforming growth factor ? overcomes a paradoxic tumor-inhibitory effect ofp53 loss in transgenic mouse epidermis
inferred← citeshttps://doi.org/10.1002/9783527619665.ch20openalex-snapshot
p73 Affects Cell Fate and Tumorigenesis
inferred← citeshttps://doi.org/10.1002/ajmg.a.63996openalex-snapshot
A Novel Missense Mutation of the ABL1 Gene in a Child With Congenital Heart Defects and Skeletal Malformations Syndrome
inferred← citeshttps://doi.org/10.1002/bies.10300openalex-snapshot
The pleiotropic functions of the Y‐box‐binding protein, YB‐1
inferred← citeshttps://doi.org/10.1002/cam4.558openalex-snapshot
RETRACTED: PLK 2 phosphorylates and inhibits enriched TA p73 in human osteosarcoma cells
inferred← citeshttps://doi.org/10.1002/cbf.70292openalex-snapshot
Prunetin Attenuates Dexamethasone‐Induced Pancreatic β‐Cell Apoptosis: Modulation of Oxidative Stress and MAPK Signaling but Not Phosphorylated C‐Abl Levels
inferred← citeshttps://doi.org/10.1002/cbic.200400186openalex-snapshot
Relevance of Atypical Protein Kinase C Isotypes to the Drug Discovery Process
inferred← citeshttps://doi.org/10.1002/gcc.22095openalex-snapshot
The TP73 complex network: Ready for clinical translation in cancer?
inferred← citeshttps://doi.org/10.1002/hed.20531openalex-snapshot
Mutant p53 proteins: Between loss and gain of function
inferred← citeshttps://doi.org/10.1002/humu.22523openalex-snapshot
How theTP53Family ProteinsTP63andTP73Contribute to Tumorigenesis: Regulators and Effectors
inferred← citeshttps://doi.org/10.1002/ijc.10253openalex-snapshot
Expression and mutation analyses of P53R2, a newly identified p53 target for DNA repair in human gastric carcinoma
inferred← citeshttps://doi.org/10.1002/ijc.10580openalex-snapshot
Escape of p53 protein from E6‐mediated degradation in HeLa cells after cisplatin therapy
inferred← citeshttps://doi.org/10.1002/ijc.21033openalex-snapshot
P73 functionally replaces p53 in Adriamycin‐treated, p53‐deficient breast cancer cells
inferred← citeshttps://doi.org/10.1002/ijc.23063openalex-snapshot
c‐Abl activates p38 MAPK independently of its tyrosine kinase activity: Implications in cisplatin‐based therapy
inferred← citeshttps://doi.org/10.1002/jcb.10678openalex-snapshot
Strong induction of p73 protein in vivo coincides with the onset of apoptosis in rat liver after treatment with the hepatocarcinogen N‐nitrosomorpholine (NNM)
inferred← citeshttps://doi.org/10.1002/jcp.20638openalex-snapshot
The course of etoposide‐induced apoptosis in Jurkat cells lacking p53 and Bax
inferred← citeshttps://doi.org/10.1002/med.21546openalex-snapshot
Topoisomerase 1B poisons: Over a half‐century of drug leads, clinical candidates, and serendipitous discoveries
inferred← citeshttps://doi.org/10.1002/path.2066openalex-snapshot
The presence of an intronic deletion in p73 and high levels of ZEB1 alter the TAp73/ΔTAp73 ratio in colorectal carcinomas
inferred← citeshttps://doi.org/10.1002/pros.1109openalex-snapshot
Germline mutations in the p73 gene do not predispose to familial prostate‐brain cancer
inferred← citeshttps://doi.org/10.1002/tox.20631openalex-snapshot
Benzo(a)pyrene induces p73 mRNA expression and necrosis in human lung adenocarcinoma H1299 cells
inferred← citeshttps://doi.org/10.1006/bbrc.2000.2932openalex-snapshot
p63 and p73 Transactivate Differentiation Gene Promoters in Human Keratinocytes
inferred← citeshttps://doi.org/10.1006/bbrc.2000.3627openalex-snapshot
Enhanced p73 Expression during Differentiation and Complex p73 Isoforms in Myeloid Leukemia
inferred← citeshttps://doi.org/10.1006/excr.2002.5535openalex-snapshot
p53 Gene Family p51(p63)-Encoded, Secondary Transactivator p51B(TAp63α) Occurs without Forming an Immunoprecipitable Complex with MDM2, but Responds to Genotoxic Stress by Accumulation
inferred← citeshttps://doi.org/10.1006/geno.2001.6541openalex-snapshot
Cloning, Mapping, and Characterization of the Human Sorbin and SH3 Domain Containing 1 (SORBS1) Gene: A Protein Associated with c-Abl during Insulin Signaling in the Hepatoma Cell Line Hep3B
inferred← citeshttps://doi.org/10.1007/0-387-30127-5openalex-snapshot
The p53 Tumor Suppressor Pathway and Cancer
inferred← citeshttps://doi.org/10.1007/0-387-30127-5_8openalex-snapshot
p53 Family Members: p63 and p73
inferred← citeshttps://doi.org/10.1007/0-387-30127-5_9openalex-snapshot
The Oncogenic Activity of p53 Mutants
inferred← citeshttps://doi.org/10.1007/978-0-387-68744-5_1openalex-snapshot
Mechanisms of Activation of Abl Family Kinases
inferred← citeshttps://doi.org/10.1007/978-1-4020-2922-6_10openalex-snapshot
P73, P63 and Mutant P53: Members of Protein Complexs Floating in Cancer Cells
inferred← citeshttps://doi.org/10.1007/978-1-4020-2922-6_9openalex-snapshot
P53, P63, and P73: Internecine Relations?
inferred← citeshttps://doi.org/10.1007/978-1-4419-8231-5_3openalex-snapshot
TP63, TP73: The Guardian’s Elder Brothers
inferred← citeshttps://doi.org/10.1007/978-1-4614-6220-0_8openalex-snapshot
YAP and p73: A Matter of Mutual Specificity in Tumor Suppression
inferred← citeshttps://doi.org/10.1007/978-1-4614-6220-0_9openalex-snapshot
The c-Abl/YAP/p73 Apoptotic Module and the HIPPO Pathway
inferred← citeshttps://doi.org/10.1007/978-1-4939-8910-2_17openalex-snapshot
Hippo Pathway Regulation by Tyrosine Kinases
inferred← citeshttps://doi.org/10.1007/978-1-59259-218-0_16openalex-snapshot
Integrative Signaling Through c-Abl
inferred← citeshttps://doi.org/10.1007/978-1-59745-199-4_12openalex-snapshot
DNA Damage-Dependent Apoptosis
inferred← citeshttps://doi.org/10.1007/978-3-540-74386-6_2openalex-snapshot
Tumor Growth and Cell Proliferation
inferred← citeshttps://doi.org/10.1007/978-3-540-79725-8_4openalex-snapshot
Genetische Grundlagen der Kanzerogenese
inferred← citeshttps://doi.org/10.1007/978-3-662-04667-8_4openalex-snapshot
ATM: From Phenotype to Functional Genomics — And Back
inferred← citeshttps://doi.org/10.1007/978-3-662-06670-6_4openalex-snapshot
Genetische Grundlagen der Kanzerogenese
inferred← citeshttps://doi.org/10.1007/bf02759672openalex-snapshot
Genep73: Deletions and expression in non-small-cell lung carcinoma cells
inferred← citeshttps://doi.org/10.1007/bf02974277openalex-snapshot
Overexpression of p73 enhances cisplatin-induced apoptosis in HeLa cells
inferred← citeshttps://doi.org/10.1007/s00018-007-7270-0openalex-snapshot
Prolyl isomerase, Pin1: new findings of post-translational modifications and physiological substrates in cancer, asthma and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00018-016-2130-4openalex-snapshot
The DNA damage-induced cell death response: a roadmap to kill cancer cells
inferred← citeshttps://doi.org/10.1007/s00280-012-1823-0openalex-snapshot
Correlation between low-level expression of the tumor suppressor gene TAp73 and the chemoresistance of human glioma stem cells
inferred← citeshttps://doi.org/10.1007/s10495-010-0488-6openalex-snapshot
The c-Abl tyrosine kinase stabilizes Pitx1 in the apoptotic response to DNA damage
inferred← citeshttps://doi.org/10.1007/s10555-013-9424-xopenalex-snapshot
Functions, divergence and clinical value of TAp73 isoforms in cancer
inferred← citeshttps://doi.org/10.1016/b978-0-12-374145-5.00266-7openalex-snapshot
Nuclear and Cytoplasmic Functions of Abl Tyrosine Kinase
inferred← citeshttps://doi.org/10.1016/b978-0-12-384999-1.10009-5openalex-snapshot
Defective DNA Mismatch Repair-dependent c-Abl-p73-GADD45α Expression Confers Cancer Chemoresistance
inferred← citeshttps://doi.org/10.1016/b978-012072651-6/50005-xopenalex-snapshot
CELL DEATH PATHWAYS AS TARGETS FOR ANTICANCER DRUGS
inferred← citeshttps://doi.org/10.1016/b978-012124546-7/50669-0openalex-snapshot
Abl in Cell Signaling
inferred← citeshttps://doi.org/10.1016/j.abb.2004.03.026openalex-snapshot
5′-Heterogeneity of mouse Dda3 transcripts is attributed to differential initiation of transcription and alternative splicing
inferred← citeshttps://doi.org/10.1016/j.bbmt.2012.02.004openalex-snapshot
Graft-versus-Host Disease–Related Cytokine-Driven Apoptosis Depends on p73 in Cytokeratin 15–Positive Target Cells
inferred← citeshttps://doi.org/10.1016/j.bbrc.2004.02.146openalex-snapshot
Molecular dynamics simulation of the C-terminal sterile α-motif domain of human p73α: evidence of a dynamical relationship between helices 3 and 5
inferred← citeshttps://doi.org/10.1016/j.bbrc.2005.03.152openalex-snapshot
C-Abl as a modulator of p53
inferred← citeshttps://doi.org/10.1016/j.bbrc.2005.03.155openalex-snapshot
p73-induced apoptosis: A question of compartments and cooperation
inferred← citeshttps://doi.org/10.1016/j.bbrc.2005.03.156openalex-snapshot
p73 induces apoptosis by different mechanisms
inferred← citeshttps://doi.org/10.1016/j.bbrc.2005.03.158openalex-snapshot
Regulation of the p73 protein stability and degradation
inferred← citeshttps://doi.org/10.1016/j.bbrc.2005.03.192openalex-snapshot
The role of p53 in treatment responses of lung cancer
inferred← citeshttps://doi.org/10.1016/j.bbrc.2005.05.188openalex-snapshot
Calpain cleavage regulates the protein stability of p73
inferred← citeshttps://doi.org/10.1016/j.bbrc.2007.06.104openalex-snapshot
Itch inhibition regulates chemosensitivity in vitro
inferred← citeshttps://doi.org/10.1016/j.bbrc.2007.08.128openalex-snapshot
Transcriptional regulation of the p73 gene, a member of the p53 family, by early growth response-1 (Egr-1)
inferred← citeshttps://doi.org/10.1016/j.bbrc.2007.08.148openalex-snapshot
Filamin A negatively regulates the transcriptional activity of p73α in the cytoplasm
inferred← citeshttps://doi.org/10.1016/j.bbrc.2007.10.145openalex-snapshot
Yin Yang 1 induces transcriptional activity of p73 through cooperation with E2F1
inferred← citeshttps://doi.org/10.1016/j.bbrc.2009.06.035openalex-snapshot
Acetylation status of E2F-1 has an important role in the regulation of E2F-1-mediated transactivation of tumor suppressor p73
inferred← citeshttps://doi.org/10.1016/j.cancergencyto.2003.08.020openalex-snapshot
TP73 alterations in cervical carcinoma
inferred← citeshttps://doi.org/10.1016/j.canlet.2003.11.014openalex-snapshot
Genetic polymorphisms of p73 G4C14-to-A4T14 at exon 2 and p53 Arg72Pro and the risk of cervical cancer in Japanese
inferred← citeshttps://doi.org/10.1016/j.canlet.2004.12.050openalex-snapshot
Functional implication of p73 protein stability in neuronal cell survival and death
inferred← citeshttps://doi.org/10.1016/j.canlet.2006.10.007openalex-snapshot
Activation of mitogen-activated protein kinases by cisplatin and their role in cisplatin-resistance
inferred← citeshttps://doi.org/10.1016/j.canlet.2008.07.019openalex-snapshot
Regulation of p53R2 and its role as potential target for cancer therapy
inferred← citeshttps://doi.org/10.1016/j.ccr.2004.06.005openalex-snapshot
p63 and p73 are not required for the development and p53-dependent apoptosis of T cells
inferred← citeshttps://doi.org/10.1016/j.ccr.2007.10.001openalex-snapshot
Shaping Genetic Alterations in Human Cancer: The p53 Mutation Paradigm
inferred← citeshttps://doi.org/10.1016/j.cellimm.2010.04.005openalex-snapshot
PIASy interacts with p73α and regulates cell cycle in HEK293 cells
inferred← citeshttps://doi.org/10.1016/j.cellsig.2007.01.027openalex-snapshot
PKCδ signaling: Mechanisms of DNA damage response and apoptosis
inferred← citeshttps://doi.org/10.1016/j.cellsig.2013.10.005openalex-snapshot
c-ABL tyrosine kinase modulates p53-dependent p21 induction and ensuing cell fate decision in response to DNA damage
inferred← citeshttps://doi.org/10.1016/j.cellsig.2019.04.016openalex-snapshot
The interaction between PLEKHG2 and ABL1 suppresses cell growth via the NF-κB signaling pathway in HEK293 cells
inferred← citeshttps://doi.org/10.1016/j.devcel.2012.08.001openalex-snapshot
Mig6 Is a Sensor of EGF Receptor Inactivation that Directly Activates c-Abl to Induce Apoptosis during Epithelial Homeostasis
inferred← citeshttps://doi.org/10.1016/j.drup.2007.01.001openalex-snapshot
One, two, three—p53, p63, p73 and chemosensitivity
inferred← citeshttps://doi.org/10.1016/j.drup.2008.08.001openalex-snapshot
Therapeutic prospects for p73 and p63: Rising from the shadow of p53
inferred← citeshttps://doi.org/10.1016/j.ejca.2005.08.012openalex-snapshot
Transcription inhibition: A potential strategy for cancer therapeutics
inferred← citeshttps://doi.org/10.1016/j.ejca.2007.01.042openalex-snapshot
p73: A chiaroscuro gene in cancer
inferred← citeshttps://doi.org/10.1016/j.febslet.2007.02.049openalex-snapshot
STAT1 regulates p73‐mediated Bax gene expression
inferred← citeshttps://doi.org/10.1016/j.febslet.2014.06.047openalex-snapshot
TP63 and TP73 in cancer, an unresolved “family” puzzle of complexity, redundancy and hierarchy
inferred← citeshttps://doi.org/10.1016/j.ijrobp.2007.08.033openalex-snapshot
p73 Protein Expression Correlates With Radiation-Induced Apoptosis in the Lack of p53 Response to Radiation Therapy for Cervical Cancer
inferred← citeshttps://doi.org/10.1016/j.jlr.2024.100579openalex-snapshot
Hyperactivation of SREBP induces pannexin-1-dependent lytic cell death
inferred← citeshttps://doi.org/10.1016/j.molcel.2004.05.007openalex-snapshot
Pin1 Links the Activities of c-Abl and p300 in Regulating p73 Function
inferred← citeshttps://doi.org/10.1016/j.molcel.2005.04.008openalex-snapshot
The Transcriptional Coactivator Yes-Associated Protein Drives p73 Gene-Target Specificity in Response to DNA Damage
inferred← citeshttps://doi.org/10.1016/j.molcel.2006.04.007openalex-snapshot
Interaction between c-Abl and Arg Tyrosine Kinases and Proteasome Subunit PSMA7 Regulates Proteasome Degradation
inferred← citeshttps://doi.org/10.1016/j.molcel.2007.12.022openalex-snapshot
Yap1 Phosphorylation by c-Abl Is a Critical Step in Selective Activation of Proapoptotic Genes in Response to DNA Damage
inferred← citeshttps://doi.org/10.1016/j.nbd.2004.06.007openalex-snapshot
Activation of the neuronal c-Abl tyrosine kinase by amyloid-β-peptide and reactive oxygen species
inferred← citeshttps://doi.org/10.1016/j.phrs.2020.105245openalex-snapshot
p73: From the p53 shadow to a major pharmacological target in anticancer therapy
inferred← citeshttps://doi.org/10.1016/j.semcancer.2020.03.003openalex-snapshot
ITCH as a potential therapeutic target in human cancers
inferred← citeshttps://doi.org/10.1016/j.tibs.2003.10.004openalex-snapshot
Functional regulation of p73 and p63: development and cancer
inferred← citeshttps://doi.org/10.1016/j.yexcr.2009.05.004openalex-snapshot
Evidence for a direct involvement of hMSH5 in promoting ionizing radiation induced apoptosis
inferred← citeshttps://doi.org/10.1016/s0006-291x(02)00723-4openalex-snapshot
p53-independent apoptosis is induced by the p19ARF tumor suppressor
inferred← citeshttps://doi.org/10.1016/s0006-291x(02)02692-xopenalex-snapshot
Arg tyrosine kinase is involved in homologous recombinational DNA repair
inferred← citeshttps://doi.org/10.1016/s0006-291x(02)03069-3openalex-snapshot
Homozygous deletions and point mutations of the Rit1/Bcl11b gene in γ-ray induced mouse thymic lymphomas
inferred← citeshttps://doi.org/10.1016/s0006-291x(03)01209-9openalex-snapshot
Cloning and developmental expression of p73 cDNA in zebrafish
inferred← citeshttps://doi.org/10.1016/s0006-291x(03)01347-0openalex-snapshot
A novel HECT-type E3 ubiquitin ligase, NEDL2, stabilizes p73 and enhances its transcriptional activity
inferred← citeshttps://doi.org/10.1016/s0006-2952(00)00434-2openalex-snapshot
Nuclear tyrosine phosphorylation: the beginning of a map
inferred← citeshttps://doi.org/10.1016/s0006-2952(02)00917-6openalex-snapshot
Expression of p53 -family members and associated target molecules in breast cancer cell lines in response to vincristine treatment
inferred← citeshttps://doi.org/10.1016/s0006-2952(03)00511-2openalex-snapshot
p73 and p63 protein stability: the way to regulate function?
inferred← citeshttps://doi.org/10.1016/s0014-5793(01)02119-6openalex-snapshot
From p63 to p53 across p73
inferred← citeshttps://doi.org/10.1016/s0014-5793(01)02124-xopenalex-snapshot
MDM2 and MDMX can interact differently with ARF and members of the p53 family
inferred← citeshttps://doi.org/10.1016/s0014-5793(01)03141-6openalex-snapshot
Overproduced p73α activates a minimal promoter through a mechanism independent of its transcriptional activity
inferred← citeshttps://doi.org/10.1016/s0014-5793(02)02921-6openalex-snapshot
Induction of p73β by a naphthoquinone analog is mediated by E2F‐1 and triggers apoptosis in HeLa cells
inferred← citeshttps://doi.org/10.1016/s0014-5793(03)01024-xopenalex-snapshot
ING1b decreases cell proliferation through p53‐dependent and ‐independent mechanisms
inferred← citeshttps://doi.org/10.1016/s0026-895x(24)12108-6openalex-snapshot
Inhibition of c-Abl with STI571 Attenuates Stress-Activated Protein Kinase Activation and Apoptosis in the Cellular Response to 1-β-d-Arabinofuranosylcytosine
inferred← citeshttps://doi.org/10.1016/s0027-5107(01)00115-4openalex-snapshot
Homologous recombinational repair of DNA ensures mammalian chromosome stability
inferred← citeshttps://doi.org/10.1016/s0065-230x(02)85003-5openalex-snapshot
The Abl family kinases: Mechanisms of regulation and signaling
inferred← citeshttps://doi.org/10.1016/s0070-2153(04)59007-5openalex-snapshot
Death by Abl: A Matter of Location
inferred← citeshttps://doi.org/10.1016/s0074-7696(02)19013-9openalex-snapshot
Genetic alterations in ovarian cancer cells that might account for sensitivity to chemotherapy in patients
inferred← citeshttps://doi.org/10.1016/s0092-8674(00)00010-6openalex-snapshot
Distinct Initiation and Maintenance Mechanisms Cooperate to Induce G1 Cell Cycle Arrest in Response to DNA Damage
inferred← citeshttps://doi.org/10.1016/s0092-8674(00)80626-1openalex-snapshot
Drosophila p53 Is a Structural and Functional Homolog of the Tumor Suppressor p53
inferred← citeshttps://doi.org/10.1016/s0145-2126(01)00181-3openalex-snapshot
The kinase inhibitor STI571 reverses the Bcr–Abl induced point mutation frequencies observed in pre-leukemic P190Bcr–Abl transgenic mice
inferred← citeshttps://doi.org/10.1016/s0165-3806(03)00192-5openalex-snapshot
Atm and c-Abl cooperate in the response to genotoxic stress during nervous system development
inferred← citeshttps://doi.org/10.1016/s0168-9525(02)02595-7openalex-snapshot
On the shoulders of giants: p63, p73 and the rise of p53
inferred← citeshttps://doi.org/10.1016/s0169-328x(03)00052-4openalex-snapshot
Induction of apoptosis by the p53-related p73 and partial inhibition by the baculovirus-encoded p35 in neuronal cells
inferred← citeshttps://doi.org/10.1016/s0300-483x(03)00291-9openalex-snapshot
Homologous recombination and cell cycle checkpoints: Rad51 in tumour progression and therapy resistance
inferred← citeshttps://doi.org/10.1016/s0301-472x(00)00142-9openalex-snapshot
The tyrosine kinase inhibitor STI571, like interferon-α, preferentially reduces the capacity for amplification of granulocyte-macrophage progenitors from patients with chronic myeloid leukemia
inferred← citeshttps://doi.org/10.1016/s0304-419x(00)00010-xopenalex-snapshot
p63 and p73: old members of a new family
inferred← citeshttps://doi.org/10.1016/s0304-419x(01)00036-1openalex-snapshot
p53, p63 and p73 – solos, alliances and feuds among family members
inferred← citeshttps://doi.org/10.1016/s0305-0491(00)00174-7openalex-snapshot
Molecular characterization of the first non-mammalian p73 cDNA
inferred← citeshttps://doi.org/10.1016/s0898-6568(01)00195-4openalex-snapshot
p73 is a growth-regulated protein in vascular smooth muscle cells and is present at high levels in human atherosclerotic plaque
inferred← citeshttps://doi.org/10.1016/s0959-437x(00)00159-3openalex-snapshot
ATM and ATR: networking cellular responses to DNA damage
inferred← citeshttps://doi.org/10.1016/s0960-9822(00)00483-8openalex-snapshot
Regulation of the hTERT telomerase catalytic subunit by the c-Abl tyrosine kinase
inferred← citeshttps://doi.org/10.1016/s1097-2765(00)00138-6openalex-snapshot
Cytoskeletal Protein PSTPIP1 Directs the PEST-Type Protein Tyrosine Phosphatase to the c-Abl Kinase to Mediate Abl Dephosphorylation
inferred← citeshttps://doi.org/10.1016/s1097-2765(02)00431-8openalex-snapshot
DNA Damage-Dependent Acetylation of p73 Dictates the Selective Activation of Apoptotic Target Genes
inferred← citeshttps://doi.org/10.1016/s1357-4310(99)01545-2openalex-snapshot
The p53 family: same response, different signals?
inferred← citeshttps://doi.org/10.1016/s1383-5742(01)00075-8openalex-snapshot
A role for p53 in the frequency and mechanism of mutation
inferred← citeshttps://doi.org/10.1016/s1383-5742(02)00009-1openalex-snapshot
DNA repair/pro-apoptotic dual-role proteins in five major DNA repair pathways: fail-safe protection against carcinogenesis
inferred← citeshttps://doi.org/10.1016/s1383-5742(02)00069-8openalex-snapshot
Protein kinases and their involvement in the cellular responses to genotoxic stress
inferred← citeshttps://doi.org/10.1016/s1535-6108(03)00078-3openalex-snapshot
Chemosensitivity linked to p73 function
inferred← citeshttps://doi.org/10.1016/s1535-6108(03)00079-5openalex-snapshot
p53 polymorphism influences response in cancer chemotherapy via modulation of p73-dependent apoptosis
inferred← citeshttps://doi.org/10.1016/s1535-6108(04)00056-xopenalex-snapshot
BCR/ABL translocates to the nucleus and disrupts an ATR-dependent intra-S phase checkpoint
inferred← citeshttps://doi.org/10.1016/s1566-3124(01)08003-8openalex-snapshot
Molecular mechanisms regulating telomerase activity
inferred← citeshttps://doi.org/10.1016/s1874-5784(05)80016-9openalex-snapshot
The Role of p53 and p73 Genes in Tumor Formation
inferred← citeshttps://doi.org/10.1017/cbo9781139046947.040openalex-snapshot
DNA-damage-induced apoptosis
inferred← citeshttps://doi.org/10.1021/cb700060topenalex-snapshot
Mitomycin–DNA Adducts Induce p53-Dependent and p53-Independent Cell Death Pathways
inferred← citeshttps://doi.org/10.1021/pr4005508openalex-snapshot
Analysis of TAp73-Dependent Signaling via Omics Technologies
inferred← citeshttps://doi.org/10.1023/a:1009637512917openalex-snapshot
ATM dependent apoptosis in the nervous system
inferred← citeshttps://doi.org/10.1023/a:1009663809458openalex-snapshot
Role of the newer p53 family proteins in malignancy
inferred← citeshttps://doi.org/10.1023/a:1010636426860openalex-snapshot
The Role of P53 in Human Cancer
inferred← citeshttps://doi.org/10.1023/a:1014767311799openalex-snapshot
p73 overexpression and nuclear accumulation in hepatitis C virus-associated hepatocellular carcinoma.
inferred← citeshttps://doi.org/10.1038/35043127openalex-snapshot
p63 and p73: p53 mimics, menaces and more
inferred← citeshttps://doi.org/10.1038/35106009openalex-snapshot
Assessing TP53 status in human tumours to evaluate clinical outcome
inferred← citeshttps://doi.org/10.1038/416560aopenalex-snapshot
p63 and p73 are required for p53-dependent apoptosis in response to DNA damage
inferred← citeshttps://doi.org/10.1038/bjc.2016.331openalex-snapshot
Co-targeting of FAK and MDM2 triggers additive anti-proliferative effects in mesothelioma via a coordinated reactivation of p53
inferred← citeshttps://doi.org/10.1038/cdd.2010.106openalex-snapshot
A positive role for c-Abl in Atm and Atr activation in DNA damage response
inferred← citeshttps://doi.org/10.1038/cdd.2010.132openalex-snapshot
c-Abl tyrosine kinase in the DNA damage response: cell death and more
inferred← citeshttps://doi.org/10.1038/cdd.2010.35openalex-snapshot
p53-family proteins and their regulators: hubs and spokes in tumor suppression
inferred← citeshttps://doi.org/10.1038/cdd.2010.8openalex-snapshot
c-Abl and Arg tyrosine kinases regulate lysosomal degradation of the oncoprotein Galectin-3
inferred← citeshttps://doi.org/10.1038/cdd.2013.100openalex-snapshot
Hippo signaling: to die or not to die
inferred← citeshttps://doi.org/10.1038/cdd.2013.23openalex-snapshot
Analysis of the oligomeric state and transactivation potential of TAp73α
inferred← citeshttps://doi.org/10.1038/cdd.2013.42openalex-snapshot
Genetic disruption of Abl nuclear import reduces renal apoptosis in a mouse model of cisplatin-induced nephrotoxicity
inferred← citeshttps://doi.org/10.1038/cdd.2013.83openalex-snapshot
The Hippo pathway kinase Lats2 prevents DNA damage-induced apoptosis through inhibition of the tyrosine kinase c-Abl
inferred← citeshttps://doi.org/10.1038/cdd.2014.182openalex-snapshot
c-Abl antagonizes the YAP oncogenic function
inferred← citeshttps://doi.org/10.1038/cdd.2014.60openalex-snapshot
AMPK couples p73 with p53 in cell fate decision
inferred← citeshttps://doi.org/10.1038/cdd.2017.99openalex-snapshot
The LATS1 and LATS2 tumor suppressors: beyond the Hippo pathway
inferred← citeshttps://doi.org/10.1038/cddis.2009.15openalex-snapshot
c-Abl phosphorylation of ΔNp63α is critical for cell viability
inferred← citeshttps://doi.org/10.1038/cddis.2009.21openalex-snapshot
c-Abl downregulates the slow phase of double-strand break repair
inferred← citeshttps://doi.org/10.1038/cddis.2012.27openalex-snapshot
Regulation of p73 activity by post-translational modifications
inferred← citeshttps://doi.org/10.1038/cddis.2015.205openalex-snapshot
p53- and p73-independent activation of TIGAR expression in vivo
inferred← citeshttps://doi.org/10.1038/emboj.2011.238openalex-snapshot
Tyrosine phosphorylation enhances RAD52‐mediated annealing by modulating its DNA binding
inferred← citeshttps://doi.org/10.1038/mt.2009.298openalex-snapshot
Targeting of p53-Transcriptional Dysfunction by Conditionally Replicating Adenovirus Is Not Limited by p53-Homologues
inferred← citeshttps://doi.org/10.1038/ncb1228openalex-snapshot
JNK phosphorylation of 14-3-3 proteins regulates nuclear targeting of c-Abl in the apoptotic response to DNA damage
inferred← citeshttps://doi.org/10.1038/ncb1598openalex-snapshot
p73 supports cellular growth through c-Jun-dependent AP-1 transactivation
inferred← citeshttps://doi.org/10.1038/ncb2789openalex-snapshot
TAp73 enhances the pentose phosphate pathway and supports cell proliferation
inferred← citeshttps://doi.org/10.1038/ncb3130openalex-snapshot
Hypoxia-inducible TAp73 supports tumorigenesis by regulating the angiogenic transcriptome
inferred← citeshttps://doi.org/10.1038/ncb998openalex-snapshot
Differential regulation of E2F1 apoptotic target genes in response to DNA damage
inferred← citeshttps://doi.org/10.1038/ng1396openalex-snapshot
Caenorhabditis elegans ABL-1 antagonizes p53-mediated germline apoptosis after ionizing irradiation
inferred← citeshttps://doi.org/10.1038/nm.2033openalex-snapshot
Inhibition of the c-Abl–TAp63 pathway protects mouse oocytes from chemotherapy-induced death
inferred← citeshttps://doi.org/10.1038/nrc3446openalex-snapshot
p63 steps into the limelight: crucial roles in the suppression of tumorigenesis and metastasis
inferred← citeshttps://doi.org/10.1038/nrc3563openalex-snapshot
Role of ABL family kinases in cancer: from leukaemia to solid tumours
inferred← citeshttps://doi.org/10.1038/nrd1691openalex-snapshot
Cellular processing of platinum anticancer drugs
inferred← citeshttps://doi.org/10.1038/nrd4553openalex-snapshot
Exploiting replicative stress to treat cancer
inferred← citeshttps://doi.org/10.1038/onc.2008.315openalex-snapshot
The p53 family and programmed cell death
inferred← citeshttps://doi.org/10.1038/onc.2008.64openalex-snapshot
p73 and caspase-cleaved p73 fragments localize to mitochondria and augment TRAIL-induced apoptosis
inferred← citeshttps://doi.org/10.1038/onc.2009.177openalex-snapshot
The F-box protein FBXO45 promotes the proteasome-dependent degradation of p73
inferred← citeshttps://doi.org/10.1038/onc.2009.344openalex-snapshot
Roles of PKC isoforms in the induction of apoptosis elicited by aberrant Ras
inferred← citeshttps://doi.org/10.1038/onc.2009.513openalex-snapshot
c-Abl regulates estrogen receptor α transcription activity through its stabilization by phosphorylation
inferred← citeshttps://doi.org/10.1038/onc.2010.113openalex-snapshot
F-actin-binding domain of c-Abl regulates localized phosphorylation of C3G: role of C3G in c-Abl-mediated cell death
inferred← citeshttps://doi.org/10.1038/onc.2010.410openalex-snapshot
DNA damage stress response in germ cells: role of c-Abl and clinical implications
inferred← citeshttps://doi.org/10.1038/onc.2012.640openalex-snapshot
Senescence and aging: the critical roles of p53
inferred← citeshttps://doi.org/10.1038/onc.2013.84openalex-snapshot
The Ron receptor tyrosine kinase activates c-Abl to promote cell proliferation through tyrosine phosphorylation of PCNA in breast cancer
inferred← citeshttps://doi.org/10.1038/onc.2014.365openalex-snapshot
A balancing act: orchestrating amino-truncated and full-length p73 variants as decisive factors in cancer progression
inferred← citeshttps://doi.org/10.1038/onc.2014.436openalex-snapshot
p73 engages A2B receptor signalling to prime cancer cells to chemotherapy-induced death
inferred← citeshttps://doi.org/10.1038/onc.2015.111openalex-snapshot
Tyr99 phosphorylation determines the regulatory milieu of tumor suppressor p73
inferred← citeshttps://doi.org/10.1038/oncsis.2016.40openalex-snapshot
Improvement of gemcitabine sensitivity of p53-mutated pancreatic cancer MiaPaCa-2 cells by RUNX2 depletion-mediated augmentation of TAp73-dependent cell death
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