On the public citation graph

Human GRIN2B variants in neurodevelopmental disorders

https://doi.org/10.1016/j.jphs.2016.10.002

0 signed · 292 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/ajmg.a.36079openalex-snapshot

Interstitial 12p13.1 deletion involving GRIN2B in three patients with intellectual disability

inferredcites →https://doi.org/10.1002/ana.24073openalex-snapshot

GRIN2B mutations in west syndrome and intellectual disability with focal epilepsy

inferredcites →https://doi.org/10.1002/cne.903470112openalex-snapshot

Differential expression of five N‐methyl‐D‐aspartate receptor subunit mRNAs in the cerebellum of developing and adult rats

inferredcites →https://doi.org/10.1002/hup.2519openalex-snapshot

Genetic association analysis of N‐methyl‐ d ‐aspartate receptor subunit geneGRIN2Band clinical response to clozapine

inferredcites →https://doi.org/10.1007/s00439-015-1575-0openalex-snapshot

High diagnostic yield of clinical exome sequencing in Middle Eastern patients with Mendelian disorders

inferredcites →https://doi.org/10.1007/s00702-013-1125-7openalex-snapshot

Potential involvement of GRIN2B encoding the NMDA receptor subunit NR2B in the spectrum of Alzheimer’s disease

inferredcites →https://doi.org/10.1016/0896-6273(94)90210-0openalex-snapshot

Developmental and regional expression in the rat brain and functional properties of four NMDA receptors

inferredcites →https://doi.org/10.1016/j.ajhg.2010.07.019openalex-snapshot

Direct Measure of the De Novo Mutation Rate in Autism and Schizophrenia Cohorts

inferredcites →https://doi.org/10.1016/j.ajhg.2016.10.002openalex-snapshot

Mechanistic Insight into NMDA Receptor Dysregulation by Rare Variants in the GluN2A and GluN2B Agonist Binding Domains

inferredcites →https://doi.org/10.1016/j.cell.2012.03.028openalex-snapshot

Sequencing Chromosomal Abnormalities Reveals Neurodevelopmental Loci that Confer Risk across Diagnostic Boundaries

inferredcites →https://doi.org/10.1016/j.coph.2014.11.008openalex-snapshot

NMDA receptor subunit mutations in neurodevelopmental disorders

inferredcites →https://doi.org/10.1016/j.ejmg.2013.12.009openalex-snapshot

Report on 3 patients with 12p duplication including GRIN2B

inferredcites →https://doi.org/10.1016/j.neuron.2005.08.016openalex-snapshot

The Synaptic Localization of NR2B-Containing NMDA Receptors Is Controlled by Interactions with PDZ Proteins and AP-2

inferredcites →https://doi.org/10.1016/j.schres.2012.08.024openalex-snapshot

Absence of de novo point mutations in exons of GRIN2B in a large schizophrenia trio sample

inferredcites →https://doi.org/10.1016/j.ymgme.2014.04.001openalex-snapshot

Three rare diseases in one Sib pair: RAI1, PCK1, GRIN2B mutations associated with Smith–Magenis Syndrome, cytosolic PEPCK deficiency and NMDA receptor glutamate insensitivity

inferredcites →https://doi.org/10.1016/s0022-3565(24)35002-5openalex-snapshot

Stimulatory and Inhibitory Properties of Aminoglycoside Antibiotics at N-Methyl-d-Aspartate Receptors

inferredcites →https://doi.org/10.1016/s0140-6736(12)61480-9openalex-snapshot

Range of genetic mutations associated with severe non-syndromic sporadic intellectual disability: an exome sequencing study

inferredcites →https://doi.org/10.1016/s0896-6273(00)80051-3openalex-snapshot

Impairment of Suckling Response, Trigeminal Neuronal Pattern Formation, and Hippocampal LTD in NMDA Receptor ε2 Subunit Mutant Mice

inferredcites →https://doi.org/10.1016/s0896-6273(00)80157-9openalex-snapshot

SAP102, a Novel Postsynaptic Protein That Interacts with NMDA Receptor Complexes In Vivo

inferredcites →https://doi.org/10.1016/s1474-4422(15)00199-4openalex-snapshot

A roadmap for precision medicine in the epilepsies

inferredcites →https://doi.org/10.1038/385439a0openalex-snapshot

Competitive binding of α-actinin and calmodulin to the NMDA receptor

inferredcites →https://doi.org/10.1038/43432openalex-snapshot

Genetic enhancement of learning and memory in mice

inferredcites →https://doi.org/10.1038/ejhg.2015.186openalex-snapshot

Novel genetic causes for cerebral visual impairment

inferredcites →https://doi.org/10.1038/gim.2014.191openalex-snapshot

Whole-exome sequencing in undiagnosed genetic diseases: interpreting 119 trios

inferredcites →https://doi.org/10.1038/mp.2013.127openalex-snapshot

Excess of rare novel loss-of-function variants in synaptic genes in schizophrenia and autism spectrum disorders

inferredcites →https://doi.org/10.1038/nature08624openalex-snapshot

X-ray structure, symmetry and mechanism of an AMPA-subtype glutamate receptor

inferredcites →https://doi.org/10.1038/nature10989openalex-snapshot

Sporadic autism exomes reveal a highly interconnected protein network of de novo mutations

inferredcites →https://doi.org/10.1038/nature12439openalex-snapshot

De novo mutations in epileptic encephalopathies

inferredcites →https://doi.org/10.1038/nature13548openalex-snapshot

NMDA receptor structures reveal subunit arrangement and pore architecture

inferredcites →https://doi.org/10.1038/nature19057openalex-snapshot

Analysis of protein-coding genetic variation in 60,706 humans

inferredcites →https://doi.org/10.1038/ncb990openalex-snapshot

NMDA receptor trafficking through an interaction between PDZ proteins and the exocyst complex

inferredcites →https://doi.org/10.1038/ng.677openalex-snapshot

Mutations in GRIN2A and GRIN2B encoding regulatory subunits of NMDA receptors cause variable neurodevelopmental phenotypes

inferredcites →https://doi.org/10.1038/ng.835openalex-snapshot

Exome sequencing in sporadic autism spectrum disorders identifies severe de novo mutations

inferredcites →https://doi.org/10.1038/nn.4352openalex-snapshot

Meta-analysis of 2,104 trios provides support for 10 new genes for intellectual disability

inferredcites →https://doi.org/10.1038/srep08296openalex-snapshot

Association of genetic variants of GRIN2B with autism

inferredcites →https://doi.org/10.1038/tp.2011.52openalex-snapshot

Rare mutations in N-methyl-D-aspartate glutamate receptors in autism spectrum disorders and schizophrenia

inferredcites →https://doi.org/10.1056/nejmoa1206524openalex-snapshot

Diagnostic Exome Sequencing in Persons with Severe Intellectual Disability

inferredcites →https://doi.org/10.1073/pnas.062046299openalex-snapshot

NMDA receptor function is regulated by the inhibitory scaffolding protein, RACK1

inferredcites →https://doi.org/10.1074/jbc.m402599200openalex-snapshot

Export from the Endoplasmic Reticulum of Assembled N-Methyl-D-aspartic Acid Receptors Is Controlled by a Motif in the C Terminus of the NR2 Subunit

inferredcites →https://doi.org/10.1101/049056openalex-snapshot

Prevalence, phenotype and architecture of developmental disorders caused by de novo mutation: The Deciphering Developmental Disorders Study

inferredcites →https://doi.org/10.1111/j.1528-1167.2010.02555.xopenalex-snapshot

Deletions in 16p13 including GRIN2A in patients with intellectual disability, various dysmorphic features, and seizure disorders of the rolandic region

inferredcites →https://doi.org/10.1124/mol.115.103036openalex-snapshot

A Novel Binding Mode Reveals Two Distinct Classes of NMDA Receptor GluN2B-selective Antagonists

inferredcites →https://doi.org/10.1124/pr.109.002451openalex-snapshot

Glutamate Receptor Ion Channels: Structure, Regulation, and Function

inferredcites →https://doi.org/10.1126/science.1227764openalex-snapshot

Multiplex Targeted Sequencing Identifies Recurrently Mutated Genes in Autism Spectrum Disorders

inferredcites →https://doi.org/10.1126/science.1251915openalex-snapshot

Crystal structure of a heterotetrameric NMDA receptor ion channel

inferredcites →https://doi.org/10.1126/science.7569905openalex-snapshot

Domain Interaction Between NMDA Receptor Subunits and the Postsynaptic Density Protein PSD-95

inferredcites →https://doi.org/10.1146/annurev.cellbio.24.110707.175235openalex-snapshot

Communication Between the Synapse and the Nucleus in Neuronal Development, Plasticity, and Disease

inferredcites →https://doi.org/10.1186/1744-9081-9-20openalex-snapshot

Behavioral phenotype in five individuals with de novo mutations within the GRIN2B gene

inferredcites →https://doi.org/10.1371/journal.pgen.1003709openalex-snapshot

Genic Intolerance to Functional Variation and the Interpretation of Personal Genomes

inferredcites →https://doi.org/10.1371/journal.pgen.1004772openalex-snapshot

De Novo Mutations in Moderate or Severe Intellectual Disability

inferredcites →https://doi.org/10.1371/journal.pone.0141782openalex-snapshot

Gene Mutation Analysis in 253 Chinese Children with Unexplained Epilepsy and Intellectual/Developmental Disabilities

inferredcites →https://doi.org/10.1523/jneurosci.16-07-02157.1996openalex-snapshot

Interaction between the C terminus of NMDA receptor subunits and multiple members of the PSD-95 family of membrane-associated guanylate kinases

inferredcites →https://doi.org/10.1523/jneurosci.1890-04.2004openalex-snapshot

Subunit-Specific Regulation of NMDA Receptor Endocytosis

inferredcites →https://doi.org/10.1523/jneurosci.19-11-04189.1999openalex-snapshot

Characterization of MALS/Velis-1, -2, and -3: a Family of Mammalian LIN-7 Homologs Enriched at Brain Synapses in Association with the Postsynaptic Density-95/NMDA Receptor Postsynaptic Complex

inferredcites →https://doi.org/10.1523/jneurosci.3793-07.2007openalex-snapshot

NR2B Signaling Regulates the Development of Synaptic AMPA Receptor Current

inferredcites →https://doi.org/10.4161/cib.27887openalex-snapshot

Glutamate receptor mutations in psychiatric and neurodevelopmental disorders

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4643380openalex-extracted

inferred← citeshttps://doi.org/10.1002/agm2.12075openalex-snapshot

Glutamate ionotropic receptor NMDA type subunit 2A ( GRIN 2A) gene polymorphism (rs4998386) and Parkinson's disease susceptibility: A meta‐analysis

inferred← citeshttps://doi.org/10.1002/ajmg.b.32821openalex-snapshot

Distinct genetic patterns of shared and unique genes across four neurodevelopmental disorders

inferred← citeshttps://doi.org/10.1002/ana.25032openalex-snapshot

GABBR2 mutations determine phenotype in rett syndrome and epileptic encephalopathy

inferred← citeshttps://doi.org/10.1002/aur.70125openalex-snapshot

Sex Differences in Auditory Brainstem Responses of Two Rat Models of Autism: Environmental and Genetic Contributions to Autism‐Like Auditory Function

inferred← citeshttps://doi.org/10.1002/ccs3.70024openalex-snapshot

GRIN2B alleviates mid‐gestational sevoflurane exposure‐induced early differentiation of rat neural stem cells by interacting with KIF17

inferred← citeshttps://doi.org/10.1002/hipo.23332openalex-snapshot

Behind the scenes: Are latent memories supported by calcium independent plasticity?

inferred← citeshttps://doi.org/10.1002/humu.23895openalex-snapshot

De novoGRINvariants in NMDA receptor M2 channel pore‐forming loop are associated with neurological diseases

inferred← citeshttps://doi.org/10.1002/jat.70185openalex-snapshot

Tannic Acid Mitigates Monosodium Glutamate‐Induced Cortical Damage by Modulating Synaptic and Calcium‐Related Gene and Protein Expression

inferred← citeshttps://doi.org/10.1002/jdn.10326openalex-snapshot

Single nucleotide polymorphisms of ANKK1, DDR4, and GRIN2B genes predict behavior in a prospective cohort of Mexican children and adolescents

inferred← citeshttps://doi.org/10.1002/jnr.24560openalex-snapshot

Dysfunction of the corticostriatal pathway in autism spectrum disorders

inferred← citeshttps://doi.org/10.1002/mnfr.201700461openalex-snapshot

Maternal Chronic Folate Supplementation Ameliorates Behavior Disorders Induced by Prenatal High‐Fat Diet Through Methylation Alteration of BDNF and Grin2b in Offspring Hippocampus

inferred← citeshttps://doi.org/10.1002/prot.25595openalex-snapshot

The GluN2B‐Glu413Gly NMDA receptor variant arising from a de novo GRIN2B mutation promotes ligand‐unbinding and domain opening

inferred← citeshttps://doi.org/10.1002/prp2.480openalex-snapshot

Exploring the overlapping binding sites of ifenprodil and EVT ‐101 in GluN2B‐containing NMDA receptors using novel chicken embryo forebrain cultures and molecular modeling

inferred← citeshttps://doi.org/10.1007/978-1-0716-3830-9_10openalex-snapshot

Estimating the Ca2+ Permeability of NMDA Receptors with Whole-Cell Patch-Clamp Electrophysiology

inferred← citeshttps://doi.org/10.1007/978-1-0716-3830-9_13openalex-snapshot

Behavioral Assays Dissecting NMDA Receptor Function in Zebrafish

inferred← citeshttps://doi.org/10.1007/978-3-030-35213-4_4openalex-snapshot

Prenatal Exposure to Endocrine Disrupting Chemicals and Their Effect on Health Later in Life

inferred← citeshttps://doi.org/10.1007/978-3-030-67928-6_127-1openalex-snapshot

Addictions and Polymorphism

inferred← citeshttps://doi.org/10.1007/978-3-030-92392-1_127openalex-snapshot

Addictions and Polymorphism

inferred← citeshttps://doi.org/10.1007/978-3-031-69491-2_19openalex-snapshot

EEG Biomarkers for Autism: Rational, Support, and the Qualification Process

inferred← citeshttps://doi.org/10.1007/s00018-022-04667-7openalex-snapshot

Novel neuroactive steroids as positive allosteric modulators of NMDA receptors: mechanism, site of action, and rescue pharmacology on GRIN variants associated with neurological conditions

inferred← citeshttps://doi.org/10.1007/s00018-023-05062-6openalex-snapshot

Disease-associated nonsense and frame-shift variants resulting in the truncation of the GluN2A or GluN2B C-terminal domain decrease NMDAR surface expression and reduce potentiating effects of neurosteroids

inferred← citeshttps://doi.org/10.1007/s00018-025-06057-1openalex-snapshot

Mice carrying a GluN2B protein-truncating variant have altered NMDA receptor subunit composition and their behavior recapitulates patient phenotypes

inferred← citeshttps://doi.org/10.1007/s10803-023-05897-9openalex-snapshot

Characterizing Sensory Phenotypes of Subgroups with a Known Genetic Etiology Pertaining to Diagnoses of Autism Spectrum Disorder and Intellectual Disability

inferred← citeshttps://doi.org/10.1007/s11064-022-03554-8openalex-snapshot

The GluN2B-Trp373 NMDA Receptor Variant is Associated with Autism-, Epilepsy-Related Phenotypes and Reduces NMDA Receptor Currents in Rats

inferred← citeshttps://doi.org/10.1007/s11064-026-04790-yopenalex-snapshot

β-Asarone, Tenuifolin, and YuanZhi Decoction Restore Cognitive Function and Modulate GRIN2B-Associated Autophagy in Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/s12264-022-00888-9openalex-snapshot

KIF17 Modulates Epileptic Seizures and Membrane Expression of the NMDA Receptor Subunit NR2B

inferred← citeshttps://doi.org/10.1007/s13258-024-01545-3openalex-snapshot

Analysis of single-cell transcriptome data from a mouse model implicates protein synthesis dysfunction in schizophrenia

inferred← citeshttps://doi.org/10.1007/s40263-020-00702-yopenalex-snapshot

Genetic Influence on Efficacy of Pharmacotherapy for Pediatric Attention-Deficit/Hyperactivity Disorder: Overview and Current Status of Research

inferred← citeshttps://doi.org/10.1007/s40473-017-0130-9openalex-snapshot

Vascular Endothelial Growth Factor (VEGF) in Neurodevelopmental Disorders

inferred← citeshttps://doi.org/10.1016/b978-0-443-10944-7.00053-6openalex-snapshot

Emerging Gene-Based Pathways: Channelopathies (Epilepsy and Beyond)

inferred← citeshttps://doi.org/10.1016/bs.pmbts.2017.11.022openalex-snapshot

Chromosomal Conformations and Epigenomic Regulation in Schizophrenia

inferred← citeshttps://doi.org/10.1016/j.amolm.2024.100045openalex-snapshot

Identification of biomarkers and molecular mechanisms implicated in genetic variations underlying Alzheimer's disease pathogenesis

inferred← citeshttps://doi.org/10.1016/j.biopha.2024.117201openalex-snapshot

Potent and reversible open-channel blocker of NMDA receptor derived from dizocilpine with enhanced membrane-to-channel inhibition

inferred← citeshttps://doi.org/10.1016/j.biopsych.2017.05.028openalex-snapshot

RETRACTED: Rett-like Severe Encephalopathy Caused by a De Novo GRIN2B Mutation Is Attenuated by D-serine Dietary Supplement

inferred← citeshttps://doi.org/10.1016/j.biopsych.2021.04.013openalex-snapshot

A Different Outlook at Psychiatric and Neurological Diseases: Brain Somatic Mutations Are Implicated in Schizophrenia

inferred← citeshttps://doi.org/10.1016/j.brainres.2022.148209openalex-snapshot

Hyperthyroidism Leads Learning and Memory Impairment Possibly Via GRIN2B Expression Alterations

inferred← citeshttps://doi.org/10.1016/j.brainresbull.2021.03.014openalex-snapshot

Role of calcium/calmodulin-dependent kinase 2 in neurodevelopmental disorders

inferred← citeshttps://doi.org/10.1016/j.cell.2020.05.037openalex-snapshot

Determinants of Base Editing Outcomes from Target Library Analysis and Machine Learning

inferred← citeshttps://doi.org/10.1016/j.conctc.2023.101233openalex-snapshot

Effectiveness of L-serine supplementation in children with a GRIN2B loss-of-function mutation: Rationale and protocol for single patient (n-of-1) multiple cross-over trials

inferred← citeshttps://doi.org/10.1016/j.cophys.2017.12.013openalex-snapshot

De novo mutations and rare variants occurring in NMDA receptors

inferred← citeshttps://doi.org/10.1016/j.cophys.2026.100973openalex-snapshot

Dynamic synaptic mechanisms linking genetic risk to neuropsychiatric disorders

inferred← citeshttps://doi.org/10.1016/j.ebr.2021.100477openalex-snapshot

Genetic cause of epilepsy in a Greek cohort of children and young adults with heterogeneous epilepsy syndromes

inferred← citeshttps://doi.org/10.1016/j.ejmech.2020.112242openalex-snapshot

Identification of tetracyclic lactams as NMDA receptor antagonists with potential application in neurological disorders

inferred← citeshttps://doi.org/10.1016/j.ejmech.2025.117538openalex-snapshot

Heterocycle-fused phenylcyclohexylamines as novel multi-target antagonists of N-methyl-D-aspartate (NMDA) receptor and monoamine transporter for treating depression

inferred← citeshttps://doi.org/10.1016/j.ejpn.2025.06.001openalex-snapshot

Genotype variability in early-onset Hereditary Spastic Paraplegia: a single-center study

inferred← citeshttps://doi.org/10.1016/j.envint.2020.106240openalex-snapshot

Effects of a human-based mixture of persistent organic pollutants on the in vivo exposed cerebellum and cerebellar neuronal cultures exposed in vitro

inferred← citeshttps://doi.org/10.1016/j.envint.2021.106617openalex-snapshot

DNA methylation at GRIN2B partially mediates the association between prenatal bisphenol F exposure and cognitive functions in 7-year-old children in the SELMA study

inferred← citeshttps://doi.org/10.1016/j.envpol.2026.129076openalex-snapshot

Exploring potential targets and mechanisms of 4-methylbenzylidene camphor-induced developmental neurotoxicity: Integration of network toxicology and zebrafish experimental validation

inferred← citeshttps://doi.org/10.1016/j.expneurol.2017.10.005openalex-snapshot

Hypoxia-ischemia modifies postsynaptic GluN2B-containing NMDA receptor complexes in the neonatal mouse brain

inferred← citeshttps://doi.org/10.1016/j.gene.2025.149215openalex-snapshot

Incidence of alternative splicing associated with sex and opioid effects in the axon guidance pathway

inferred← citeshttps://doi.org/10.1016/j.heliyon.2024.e30420openalex-snapshot

Seeing beyond words: Visualizing autism spectrum disorder biomarker insights

inferred← citeshttps://doi.org/10.1016/j.imu.2023.101282openalex-snapshot

Identification of priority genes involved in ADHD using multiple system biology approach and establishment of Asiatic acid as a natural inhibitor against GRIN2B receptor

inferred← citeshttps://doi.org/10.1016/j.isci.2019.06.014openalex-snapshot

Chemogenetic Activation of Prefrontal Cortex in Shank3-Deficient Mice Ameliorates Social Deficits, NMDAR Hypofunction, and Sgk2 Downregulation

inferred← citeshttps://doi.org/10.1016/j.jpsychires.2020.09.022openalex-snapshot

Childhood adversity increases methylation in the GRIN2B gene

inferred← citeshttps://doi.org/10.1016/j.molpha.2026.100154openalex-snapshot

Mechanism of activity inversion for GluN2B-selective negative allosteric modulators

inferred← citeshttps://doi.org/10.1016/j.neubiorev.2017.11.015openalex-snapshot

Cellular diversity of the somatosensory cortical map plasticity

inferred← citeshttps://doi.org/10.1016/j.neubiorev.2018.07.001openalex-snapshot

Prenatal exposure to environmental insults and enhanced risk of developing Schizophrenia and Autism Spectrum Disorder: focus on biological pathways and epigenetic mechanisms

inferred← citeshttps://doi.org/10.1016/j.neubiorev.2020.09.023openalex-snapshot

The Role of Glutamate in Language and Language Disorders - Evidence from ERP and Pharmacologic Studies

inferred← citeshttps://doi.org/10.1016/j.neubiorev.2023.105313openalex-snapshot

Genetic architecture of ADHD and overlap with other psychiatric disorders and cognition-related phenotypes

inferred← citeshttps://doi.org/10.1016/j.neulet.2018.04.018openalex-snapshot

Enhanced sampling of glutamate receptor ligand-binding domains

inferred← citeshttps://doi.org/10.1016/j.neuron.2019.03.032openalex-snapshot

Alternative Splicing of Presynaptic Neurexins Differentially Controls Postsynaptic NMDA and AMPA Receptor Responses

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2018.08.010openalex-snapshot

7-Methoxyderivative of tacrine is a ‘foot-in-the-door’ open-channel blocker of GluN1/GluN2 and GluN1/GluN3 NMDA receptors with neuroprotective activity in vivo

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2020.108247openalex-snapshot

Positive allosteric modulators that target NMDA receptors rectify loss-of-function GRIN variants associated with neurological and neuropsychiatric disorders

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2021.108624openalex-snapshot

The diverse and complex modes of action of anti-NMDA receptor autoantibodies

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2021.108634openalex-snapshot

Regulation of the NMDA receptor by its cytoplasmic domains: (How) is the tail wagging the dog?

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2021.108805openalex-snapshot

Clinical and therapeutic significance of genetic variation in the GRIN gene family encoding NMDARs

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2022.109297openalex-snapshot

The pathogenic N650K variant in the GluN1 subunit regulates the trafficking, conductance, and pharmacological properties of NMDA receptors

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2023.109703openalex-snapshot

An integrated approach to evaluate the functional effects of disease-associated NMDA receptor variants

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2018.02.025openalex-snapshot

Unconventional Signaling by Extracellular CRMP2: Possible Role as an Atypical Neurotransmitter?

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2023.06.018openalex-snapshot

Functional Evaluation of a Novel GRIN2B Missense Variant Associated with Epilepsy and Intellectual Disability

inferred← citeshttps://doi.org/10.1016/j.pbb.2021.173312openalex-snapshot

From atypical senses to autism: towards new therapeutic targets and improved diagnostics

inferred← citeshttps://doi.org/10.1016/j.pharmthera.2019.107426openalex-snapshot

The origin of NMDA receptor hypofunction in schizophrenia

inferred← citeshttps://doi.org/10.1016/j.pharmthera.2025.108888openalex-snapshot

N-methyl-D-aspartate receptors (NMDARs): a glutamate-activated cation channel with biased signaling and therapeutic potential in brain disorders

inferred← citeshttps://doi.org/10.1016/j.physbeh.2026.115456openalex-snapshot

Memory impairments induced by neonatal excitotoxicity are associated with altered expression of NMDA receptor subunits NR2A and NR2B

inferred← citeshttps://doi.org/10.1016/j.pnpbp.2017.09.014openalex-snapshot

Glutamatergic synapses in neurodevelopmental disorders

inferred← citeshttps://doi.org/10.1016/j.pnpbp.2025.111569openalex-snapshot

NMDA glutamate receptor polymorphisms modulate antipsychotic-induced hyperprolactinemia in schizophrenia

inferred← citeshttps://doi.org/10.1016/j.pnpbp.2025.111591openalex-snapshot

Uncovering the female phenotype in the VPA autism model: Brain-region specific synaptic pattern, microglial priming and behavioral singularity

inferred← citeshttps://doi.org/10.1016/j.stemcr.2018.05.018openalex-snapshot

Disruption of GRIN2B Impairs Differentiation in Human Neurons

inferred← citeshttps://doi.org/10.1016/j.str.2018.05.004openalex-snapshot

Glutamate and Glycine Binding to the NMDA Receptor

inferred← citeshttps://doi.org/10.1016/j.str.2018.09.010openalex-snapshot

Intradomain Interactions in an NMDA Receptor Fragment Mediate N-Glycan Processing and Conformational Sampling

inferred← citeshttps://doi.org/10.1016/j.yebeh.2022.108604openalex-snapshot

De novo GRIN2A variants associated with epilepsy and autism and literature review

inferred← citeshttps://doi.org/10.1016/j.ymgme.2023.107523openalex-snapshot

Evidence for effect of l-serine, a novel therapy for GRIN2B-related neurodevelopmental disorder

inferred← citeshttps://doi.org/10.1016/j.ymgme.2025.109268openalex-snapshot

Potential benefits of l-serine in children with GRIN2B loss-of-function variants: Randomized n-of-1 trials

inferred← citeshttps://doi.org/10.1021/acs.jctc.7b00817openalex-snapshot

Simulated Dynamics of Glycans on Ligand-Binding Domain of NMDA Receptors Reveals Strong Dynamic Coupling between Glycans and Protein Core

inferred← citeshttps://doi.org/10.1021/acs.jmedchem.8b00255openalex-snapshot

Positive Modulators of theN-Methyl- d -aspartate Receptor: Structure–Activity Relationship Study of Steroidal 3-Hemiesters

inferred← citeshttps://doi.org/10.1021/acschemneuro.8b00591openalex-snapshot

Synthesis and Preliminary Evaluations of a Triazole-Cored Antagonist as a PET Imaging Probe ([18F]N2B-0518) for GluN2B Subunit in the Brain

inferred← citeshttps://doi.org/10.1021/acsmedchemlett.7b00309openalex-snapshot

Modulating GluN2B for the Treatment of Neurological and Psychiatric Disorders

inferred← citeshttps://doi.org/10.1038/jhg.2017.19openalex-snapshot

GRIN1 mutation associated with intellectual disability alters NMDA receptor trafficking and function

inferred← citeshttps://doi.org/10.1038/s12276-018-0129-7openalex-snapshot

Genomics in neurodevelopmental disorders: an avenue to personalized medicine

inferred← citeshttps://doi.org/10.1038/s41380-024-02572-yopenalex-snapshot

Anterior cingulate cortex-related functional hyperconnectivity underlies sensory hypersensitivity in Grin2b-mutant mice

inferred← citeshttps://doi.org/10.1038/s41398-023-02535-xopenalex-snapshot

Early maturation and hyperexcitability is a shared phenotype of cortical neurons derived from different ASD-associated mutations

inferred← citeshttps://doi.org/10.1038/s41398-025-03563-5openalex-snapshot

Meta-analysis of the brain transcriptomes of multiple genetic mouse models of schizophrenia highlights dysregulation in striatum and thalamus

inferred← citeshttps://doi.org/10.1038/s41467-023-39420-6openalex-snapshot

Schizophrenia-associated NRXN1 deletions induce developmental-timing- and cell-type-specific vulnerabilities in human brain organoids

inferred← citeshttps://doi.org/10.1038/s41467-026-69368-2openalex-snapshot

Single-cell multiomic human brain atlas reveals regulatory drivers of cortical regionality

inferred← citeshttps://doi.org/10.1038/s41514-026-00363-zopenalex-snapshot

Ordinal GWAS analysis of the frailty phenotype identified a novel locus at 12q22 that underscores the role of the neurological and immune systems

inferred← citeshttps://doi.org/10.1038/s41559-018-0611-6openalex-snapshot

Red fox genome assembly identifies genomic regions associated with tame and aggressive behaviours

inferred← citeshttps://doi.org/10.1038/s41589-019-0449-5openalex-snapshot

Biased modulators of NMDA receptors control channel opening and ion selectivity

inferred← citeshttps://doi.org/10.1038/s41598-018-29732-9openalex-snapshot

Prenatal Bisphenol A Exposure is Linked to Epigenetic Changes in Glutamate Receptor Subunit Gene Grin2b in Female Rats and Humans

inferred← citeshttps://doi.org/10.1038/s41598-019-45883-9openalex-snapshot

The Route to ‘Chemobrain’ - Computational probing of neuronal LTP pathway

inferred← citeshttps://doi.org/10.1038/s41598-020-66379-xopenalex-snapshot

Long term transcriptional and behavioral effects in mice developmentally exposed to a mixture of endocrine disruptors associated with delayed human neurodevelopment

inferred← citeshttps://doi.org/10.1038/s41598-020-75646-wopenalex-snapshot

The pathogenic S688Y mutation in the ligand-binding domain of the GluN1 subunit regulates the properties of NMDA receptors

inferred← citeshttps://doi.org/10.1038/s41598-021-90803-5openalex-snapshot

Alcohol intoxication, but not hangover, differentially impairs learning and automatization of complex motor response sequences

inferred← citeshttps://doi.org/10.1038/s42003-026-09585-zopenalex-snapshot

Sex- and etiology-specific effects on predictive processing in the inferior colliculus of two rat models of autism

inferred← citeshttps://doi.org/10.1038/s42003-026-09884-5openalex-snapshot

Neuronal ARHGAP8 controls synapse structure and AMPA receptor-mediated synaptic transmission

inferred← citeshttps://doi.org/10.1039/d5ra07716hopenalex-snapshot

Positive modulators of N -methyl- d -aspartate receptor: structure–activity relationship study on steroidal C-17 and C-20 oxime ethers

inferred← citeshttps://doi.org/10.1055/s-0037-1602822openalex-snapshot

New Genes for Epilepsy–Autism Comorbidity

inferred← citeshttps://doi.org/10.1055/s-0041-1727146openalex-snapshot

GRIN2A and GRIN2B and Their Related Phenotypes

inferred← citeshttps://doi.org/10.1055/s-0041-1739130openalex-snapshot

Favorable Response to “Memantine” in a Child with GRIN2B Epileptic Encephalopathy

inferred← citeshttps://doi.org/10.1073/pnas.2426094122openalex-snapshot

A general genome editing strategy using CRISPR lipid nanoparticle spherical nucleic acids

inferred← citeshttps://doi.org/10.1073/pnas.2518282123openalex-snapshot

Grin2b 3’UTR is necessary for synaptic plasticity and spatial learning

inferred← citeshttps://doi.org/10.1085/jgp.201912537openalex-snapshot

Cadmium activates AMPA and NMDA receptors with M3 helix cysteine substitutions

inferred← citeshttps://doi.org/10.1085/jgp.202513799openalex-snapshot

GRIN2B disease-associated mutations disrupt the function of BK channels and NMDA receptor signaling nanodomains

inferred← citeshttps://doi.org/10.1093/brain/awaa147openalex-snapshot

Modelling and treating GRIN2A developmental and epileptic encephalopathy in mice

inferred← citeshttps://doi.org/10.1093/brain/awaf355openalex-snapshot

Inhibition of GluN2B-containing N -methyl- D- aspartate receptors by radiprodil

inferred← citeshttps://doi.org/10.1093/brain/awx358openalex-snapshot

De novo mutations in GRIN1 cause extensive bilateral polymicrogyria

inferred← citeshttps://doi.org/10.1093/brain/awz232openalex-snapshot

Heterogeneous clinical and functional features of GRIN2D-related developmental and epileptic encephalopathy

inferred← citeshttps://doi.org/10.1093/nar/gkae571openalex-snapshot

Evidence for widespread translation of 5′ untranslated regions

inferred← citeshttps://doi.org/10.1093/oxfordhb/9780190669164.013.16openalex-snapshot

N-Methyl-D-Aspartate Receptors

inferred← citeshttps://doi.org/10.1097/ypg.0000000000000265openalex-snapshot

A genetic window to auditory-verbal problems in bipolar disorder

inferred← citeshttps://doi.org/10.1101/2020.01.10.901272openalex-snapshot

Gene size matters: What determines gene length in the human genome?

inferred← citeshttps://doi.org/10.1101/2020.05.26.117903openalex-snapshot

Language impairment with a microduplication in 1q42.3q43

inferred← citeshttps://doi.org/10.1101/2022.08.24.505165openalex-snapshot

Schizophrenia-associated NRXN1 deletions induce developmental-timing- and cell-type-specific vulnerabilities in human brain organoids

inferred← citeshttps://doi.org/10.1101/2025.10.02.680004openalex-snapshot

Phenotype of mice carrying an NMDA receptor GluN2B protein-truncating variant associated with intellectual disability

inferred← citeshttps://doi.org/10.1101/737239openalex-snapshot

Modeling and treating GRIN2A developmental and epileptic encephalopathy in mice

inferred← citeshttps://doi.org/10.1109/tcbb.2017.2748944openalex-snapshot

Integrating Imaging Genomic Data in the Quest for Biomarkers of Schizophrenia Disease

inferred← citeshttps://doi.org/10.1111/bph.15841openalex-snapshot

Pregnane‐based steroids are novel positive NMDA receptor modulators that may compensate for the effect of loss‐of‐function disease‐associated GRIN mutations

inferred← citeshttps://doi.org/10.1111/ejn.14841openalex-snapshot

GSK‐3β regulates the synaptic expression of NMDA receptors via phosphorylation of phosphatidylinositol 4 kinase type IIα

inferred← citeshttps://doi.org/10.1111/ejn.14842openalex-snapshot

The C‐terminal domains of the NMDA receptor: How intrinsically disordered tails affect signalling, plasticity and disease

inferred← citeshttps://doi.org/10.1111/gbb.12911openalex-snapshot

Transcriptomics analysis reveals potential regulatory role of nSMase2 (Smpd3) in nervous system development and function of middle‐aged mouse brains

inferred← citeshttps://doi.org/10.1111/jcpp.13510openalex-snapshot

A systematic review of brain MRI findings in monogenic disorders strongly associated with autism spectrum disorder

inferred← citeshttps://doi.org/10.1111/jnc.14970openalex-snapshot

Regulation of NMDA glutamate receptor functions by the GluN2 subunits

inferred← citeshttps://doi.org/10.1111/jnc.15942openalex-snapshot

Differential responses of disease‐related GRIN variants located in pore‐forming M2 domain of N‐methyl‐D ‐aspartate receptor to FDA ‐approved inhibitors

inferred← citeshttps://doi.org/10.1113/jp276093openalex-snapshot

A structurally derived model of subunit‐dependent NMDA receptor function

inferred← citeshttps://doi.org/10.1113/jp278705openalex-snapshot

From bedside‐to‐bench: What disease‐associated variants are teaching us about the NMDA receptor

inferred← citeshttps://doi.org/10.1113/jp279028openalex-snapshot

Architecture and function of NMDA receptors: an evolutionary perspective

inferred← citeshttps://doi.org/10.1124/jpet.121.001000openalex-snapshot

Opportunities for Precision Treatment of GRIN2A and GRIN2B Gain-of-Function Variants in Triheteromeric N-Methyl-D-Aspartate Receptors

inferred← citeshttps://doi.org/10.1124/mol.116.106781openalex-snapshot

Functional Evaluation of a De Novo GRIN2A Mutation Identified in a Patient with Profound Global Developmental Delay and Refractory Epilepsy

inferred← citeshttps://doi.org/10.1124/mol.117.110940openalex-snapshot

The Bioactive Protein-Ligand Conformation of GluN2C-Selective Positive Allosteric Modulators Bound to the NMDA Receptor

inferred← citeshttps://doi.org/10.1124/mol.117.111427openalex-snapshot

Properties of Triheteromeric N-Methyl-d-Aspartate Receptors Containing Two Distinct GluN1 Isoforms

inferred← citeshttps://doi.org/10.1124/pharmrev.120.000131openalex-snapshot

Structure, Function, and Pharmacology of Glutamate Receptor Ion Channels

inferred← citeshttps://doi.org/10.1126/sciadv.adx4647openalex-snapshot

Cryo-EM snapshots of NMDA receptor activation illuminate sequential rearrangements

inferred← citeshttps://doi.org/10.1126/science.aal3729openalex-snapshot

Cryo-EM structures of the triheteromeric NMDA receptor and its allosteric modulation

inferred← citeshttps://doi.org/10.1126/scisignal.aaw0936openalex-snapshot

l -Serine dietary supplementation is associated with clinical improvement of loss-of-function GRIN2B -related pediatric encephalopathy

inferred← citeshttps://doi.org/10.1136/jmedgenet-2016-104509openalex-snapshot

GRIN2B encephalopathy: novel findings on phenotype, variant clustering, functional consequences and treatment aspects

inferred← citeshttps://doi.org/10.1139/gen-2021-0006openalex-snapshot

Differential DNA methylation analysis reveals key genes in Chinese Qingyu and Landrace pigs

inferred← citeshttps://doi.org/10.1159/000500004openalex-snapshot

Phenotypic Spectrum and Severity of Disease Depending on the Mutated Protein Domain of NMDA Receptor-Encoding Genes

inferred← citeshttps://doi.org/10.1159/000515363openalex-snapshot

Precision Medicine: from Molecular Diagnoses to Treatment Opportunities in Medical Genetics

inferred← citeshttps://doi.org/10.1159/000516657openalex-snapshot

Dendritic Integration Dysfunction in Neurodevelopmental Disorders

inferred← citeshttps://doi.org/10.1159/000524710openalex-snapshot

Delving into the Genetic Causes of Language Impairment in a Case of Partial Deletion of NRXN1

inferred← citeshttps://doi.org/10.11606/t.17.2021.tde-04082021-134444openalex-snapshot

Estudo translacional do receptor N-metil-d-aspartato em primeiro episódio psicótico e em modelos animais de psicose

inferred← citeshttps://doi.org/10.11606/t.17.2021.tde-11062021-080115openalex-snapshot

Caracterização de um novo RNA longo não codificador e seu provável papel no desenvolvimento neuronal

inferred← citeshttps://doi.org/10.1167/iovs.67.8.21openalex-snapshot

Genomic Copy Number Variants Associated With Strabismus and Amblyopia in the All of Us Research Program

inferred← citeshttps://doi.org/10.1177/2329048x18787946openalex-snapshot

Atypical Rett Syndrome and Intractable Epilepsy With Novel GRIN2B Mutation

inferred← citeshttps://doi.org/10.1186/s12863-018-0670-3openalex-snapshot

A genome-wide association study for harness racing success in the Norwegian-Swedish coldblooded trotter reveals genes for learning and energy metabolism

inferred← citeshttps://doi.org/10.1186/s12864-025-12274-6openalex-snapshot

Conservation of human NMDA receptor subunits and disease variants in zebrafish

inferred← citeshttps://doi.org/10.1186/s12888-023-04543-zopenalex-snapshot

Disclosing common biological signatures and predicting new therapeutic targets in schizophrenia and obsessive–compulsive disorder by integrated bioinformatics analysis

inferred← citeshttps://doi.org/10.1186/s12888-024-05952-4openalex-snapshot

Developmental dyslexia genes are selectively targeted by diverse environmental pollutants

inferred← citeshttps://doi.org/10.1186/s12920-023-01502-1openalex-snapshot

Associative gene networks reveal novel candidates important for ADHD and dyslexia comorbidity

inferred← citeshttps://doi.org/10.1186/s13020-021-00512-4openalex-snapshot

Neurobehavioral alternations of the female offspring born to polycystic ovary syndrome model rats administered by Chinese herbal medicine

inferred← citeshttps://doi.org/10.1186/s13229-021-00428-8openalex-snapshot

Hippocampal neurons isolated from rats subjected to the valproic acid model mimic in vivo synaptic pattern: evidence of neuronal priming during early development in autism spectrum disorders

inferred← citeshttps://doi.org/10.1186/s13229-022-00516-3openalex-snapshot

Disruption of grin2B, an ASD-associated gene, produces social deficits in zebrafish

inferred← citeshttps://doi.org/10.12688/f1000research.18949.1openalex-snapshot

Distinct roles of GRIN2A and GRIN2B variants in neurological conditions

inferred← citeshttps://doi.org/10.1371/journal.pbio.3000525openalex-snapshot

The deubiquitinase USP6 affects memory and synaptic plasticity through modulating NMDA receptor stability

inferred← citeshttps://doi.org/10.1371/journal.pbio.3000717openalex-snapshot

Early correction of synaptic long-term depression improves abnormal anxiety-like behavior in adult GluN2B-C456Y-mutant mice

inferred← citeshttps://doi.org/10.1371/journal.pgen.1006536openalex-snapshot

Molecular Mechanism of Disease-Associated Mutations in the Pre-M1 Helix of NMDA Receptors and Potential Rescue Pharmacology

inferred← citeshttps://doi.org/10.1371/journal.pgen.1011524openalex-snapshot

Fitness landscapes of human microsatellites

inferred← citeshttps://doi.org/10.1371/journal.pone.0207824openalex-snapshot

Frailty Index associates with GRIN2B in two representative samples from the United States and the United Kingdom

inferred← citeshttps://doi.org/10.1523/jneurosci.0599-17.2018openalex-snapshot

Rescue of Learning and Memory Deficits in the Human Nonsyndromic Intellectual Disability Cereblon Knock-Out Mouse Model by Targeting the AMP-Activated Protein Kinase–mTORC1 Translational Pathway

inferred← citeshttps://doi.org/10.1523/jneurosci.0827-16.2017openalex-snapshot

A Rare Variant Identified Within the GluN2B C-Terminus in a Patient with Autism Affects NMDA Receptor Surface Expression and Spine Density

inferred← citeshttps://doi.org/10.1523/jneurosci.0946-25.2025openalex-snapshot

Disruption of grin2A , an Epilepsy-Associated Gene, Produces Altered Spontaneous Swim Behavior in Zebrafish

inferred← citeshttps://doi.org/10.1523/jneurosci.2211-19.2020openalex-snapshot

Nitric Oxide Signaling Strengthens Inhibitory Synapses of Cerebellar Molecular Layer Interneurons through a GABARAP-Dependent Mechanism

inferred← citeshttps://doi.org/10.1523/jneurosci.2291-23.2024openalex-snapshot

Characterization of Mice Carrying a Neurodevelopmental Disease-Associated GluN2B(L825V) Variant

inferred← citeshttps://doi.org/10.1523/jneurosci.3010-19.2020openalex-snapshot

Site of Action of Brain Neurosteroid Pregnenolone Sulfate at the N-Methyl-D-Aspartate Receptor

inferred← citeshttps://doi.org/10.20944/preprints202205.0083.v1openalex-snapshot

Gene Expression Profile Provides Novel Insights of Fasting-Refeeding Response in Zebrafish Skeletal Muscle

inferred← citeshttps://doi.org/10.21203/rs.3.rs-10189227/v1openalex-snapshot

Clonidine Ameliorates Anxiety-Like Behavior via GRIN2B Demethylation and NR2B Activation in a Two-Vessel Occlusion Rat Model

inferred← citeshttps://doi.org/10.21203/rs.3.rs-1766152/v1openalex-snapshot

Disruption of grin2B, an ASD-associated gene, produces social deficits in zebrafish.

inferred← citeshttps://doi.org/10.21203/rs.3.rs-76658/v1openalex-snapshot

Unpacking The Autism Spectrum Disorder Profile of Children With De Novo Disruptive GRIN2B Variants: A Quantitative and Qualitative Analysis

inferred← citeshttps://doi.org/10.2147/ndt.s585060openalex-snapshot

Autism Spectrum Disorder in the Genomic Era: A Comprehensive Review of Etiology, Precision Diagnostics, Clinical Outcomes, and Emerging Gene-Editing Therapies

inferred← citeshttps://doi.org/10.2217/epi-2018-0127openalex-snapshot

GRIN2B Promoter Methylation Deficits in Early-Onset Schizophrenia and its Association with Cognitive Function

inferred← citeshttps://doi.org/10.2478/enr-2022-0019openalex-snapshot

Prediction of the cognitive impairment development in patients with autoimmune thyroiditis and hypothyroidism

inferred← citeshttps://doi.org/10.26599/bsa.2022.9050020openalex-snapshot

NMDARs regulate the excitatory-inhibitory balance within neural circuits

inferred← citeshttps://doi.org/10.29399/npa.27407openalex-snapshot

Investigation of the Relationship between Autism Spectrum Disorder and FOXP2, GRIN2B, KATNAL2 and GABRA4 Genes

inferred← citeshttps://doi.org/10.30574/wjarr.2022.15.2.0811openalex-snapshot

Behaviors observed in different live species depending on nutrition

inferred← citeshttps://doi.org/10.31234/osf.io/aw25nopenalex-snapshot

Delving into the genetic causes of language impairment in people with partial deletions of NRXN1

inferred← citeshttps://doi.org/10.3233/jad-200963openalex-snapshot

Risk Variants in Three Alzheimer’s Disease Genes Show Association with EEG Endophenotypes

inferred← citeshttps://doi.org/10.3389/fcell.2020.595357openalex-snapshot

Overexpression of Human SNX27 Enhances Learning and Memory Through Modulating Synaptic Plasticity in Mice

inferred← citeshttps://doi.org/10.3389/fgene.2021.559998openalex-snapshot

Gene Size Matters: An Analysis of Gene Length in the Human Genome

inferred← citeshttps://doi.org/10.3389/fgene.2021.694312openalex-snapshot

A de novo GRIN1 Variant Associated With Myoclonus and Developmental Delay: From Molecular Mechanism to Rescue Pharmacology

inferred← citeshttps://doi.org/10.3389/fgene.2024.1376883openalex-snapshot

A high-density genome-wide approach reveals novel genetic markers linked to small ruminant lentivirus susceptibility in sheep

inferred← citeshttps://doi.org/10.3389/fgene.2025.1620058openalex-snapshot

Whole-transcriptome insights into follicle selection: deciphering key regulatory networks in Luxi gamecock

inferred← citeshttps://doi.org/10.3389/fnagi.2021.646901openalex-snapshot

Genetic Variability in Molecular Pathways Implicated in Alzheimer's Disease: A Comprehensive Review

inferred← citeshttps://doi.org/10.3389/fncel.2026.1813072openalex-snapshot

Regulation of NMDA receptor interactions with the actin cytoskeleton in dendritic spine development

inferred← citeshttps://doi.org/10.3389/fneur.2019.00685openalex-snapshot

Advances in the Potential Biomarkers of Epilepsy

inferred← citeshttps://doi.org/10.3389/fnins.2021.603715openalex-snapshot

The Extracellular Domains of GluN Subunits Play an Essential Role in Processing NMDA Receptors in the ER

inferred← citeshttps://doi.org/10.3389/fnins.2024.1385189openalex-snapshot

Effects of hyperbaric oxygen therapy on autistic behaviors and GRIN2B gene expression in valproic acid-exposed rats

inferred← citeshttps://doi.org/10.3389/fnmol.2018.00110openalex-snapshot

Surface Expression, Function, and Pharmacology of Disease-Associated Mutations in the Membrane Domain of the Human GluN2B Subunit

inferred← citeshttps://doi.org/10.3389/fnmol.2018.00113openalex-snapshot

The LILI Motif of M3-S2 Linkers Is a Component of the NMDA Receptor Channel Gate

inferred← citeshttps://doi.org/10.3389/fnmol.2020.575575openalex-snapshot

Using Zebrafish to Model Autism Spectrum Disorder: A Comparison of ASD Risk Genes Between Zebrafish and Their Mammalian Counterparts

inferred← citeshttps://doi.org/10.3389/fnsyn.2022.1090865openalex-snapshot

GRIN2B-related neurodevelopmental disorder: current understanding of pathophysiological mechanisms

showing the first 200 of 236 · page through all 236

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.