On the public citation graph
Notch-Jagged signalling can give rise to clusters of cells exhibiting a hybrid epithelial/mesenchymal phenotype
0 signed · 192 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/jcb.24813openalex-snapshot
Niche Inheritance: A Cooperative Pathway to Enhance Cancer Cell Fitness Through Ecosystem Engineering
inferredcites →https://doi.org/10.1007/0-387-28671-3_8openalex-snapshot
Cutaneous Wound Reepithelialization
inferredcites →https://doi.org/10.1016/j.ajpath.2010.12.003openalex-snapshot
Circulating Tumor Cells as a Window on Metastasis Biology in Lung Cancer
inferredcites →https://doi.org/10.1016/j.bbcan.2009.03.002openalex-snapshot
Epithelial–mesenchymal transition in cancer metastasis: Mechanisms, markers and strategies to overcome drug resistance in the clinic
inferredcites →https://doi.org/10.1016/j.bpj.2013.07.011openalex-snapshot
Coupled Reversible and Irreversible Bistable Switches Underlying TGFβ-induced Epithelial to Mesenchymal Transition
inferredcites →https://doi.org/10.1016/j.canlet.2013.08.027openalex-snapshot
Deadly crosstalk: Notch signaling at the intersection of EMT and cancer stem cells
inferredcites →https://doi.org/10.1016/j.ccr.2012.12.021openalex-snapshot
Endothelial Cells Promote the Colorectal Cancer Stem Cell Phenotype through a Soluble Form of Jagged-1
inferredcites →https://doi.org/10.1016/j.cell.2004.06.006openalex-snapshot
Twist, a Master Regulator of Morphogenesis, Plays an Essential Role in Tumor Metastasis
inferredcites →https://doi.org/10.1016/j.cell.2006.11.001openalex-snapshot
Cancer Metastasis: Building a Framework
inferredcites →https://doi.org/10.1016/j.cell.2009.03.025openalex-snapshot
The Notch Ligands Dll4 and Jagged1 Have Opposing Effects on Angiogenesis
inferredcites →https://doi.org/10.1016/j.cell.2014.07.013openalex-snapshot
Circulating Tumor Cell Clusters Are Oligoclonal Precursors of Breast Cancer Metastasis
inferredcites →https://doi.org/10.1016/j.gene.2009.01.003openalex-snapshot
TNF induction of jagged-1 in endothelial cells is NFκB-dependent
inferredcites →https://doi.org/10.1016/j.molonc.2015.10.002openalex-snapshot
Conversion to stem‐cell state in response to microenvironmental cues is regulated by balance between epithelial and mesenchymal features in lung cancer cells
inferredcites →https://doi.org/10.1016/j.stem.2013.03.002openalex-snapshot
A microRNA miR-34a-Regulated Bimodal Switch Targets Notch in Colon Cancer Stem Cells
inferredcites →https://doi.org/10.1016/j.stem.2015.02.015openalex-snapshot
The Cancer Stem Cell Niche: How Essential Is the Niche in Regulating Stemness of Tumor Cells?
inferredcites →https://doi.org/10.1016/j.stemcr.2013.11.009openalex-snapshot
Breast Cancer Stem Cells Transition between Epithelial and Mesenchymal States Reflective of their Normal Counterparts
inferredcites →https://doi.org/10.1016/j.tim.2012.06.001openalex-snapshot
Bacterial survival strategies suggest rethinking cancer cooperativity
inferredcites →https://doi.org/10.1016/j.ydbio.2009.05.566openalex-snapshot
Notch signaling is required for lateral induction of Jagged1 during FGF-induced lens fiber differentiation
inferredcites →https://doi.org/10.1016/s0012-1606(03)00107-6openalex-snapshot
Control of epidermal stem cell clusters by Notch-mediated lateral induction☆☆Supplementary data associated with this article can be found at doi:10.1016/S0012-1606(03)00107-6.
inferredcites →https://doi.org/10.1038/bjc.2011.405openalex-snapshot
Detection of circulating tumour cells with a hybrid (epithelial/mesenchymal) phenotype in patients with metastatic non-small cell lung cancer
inferredcites →https://doi.org/10.1038/cddis.2013.442openalex-snapshot
An EMT spectrum defines an anoikis-resistant and spheroidogenic intermediate mesenchymal state that is sensitive to e-cadherin restoration by a src-kinase inhibitor, saracatinib (AZD0530)
inferredcites →https://doi.org/10.1038/emboj.2010.349openalex-snapshot
The ZEB1/miR‐200 feedback loop controls Notch signalling in cancer cells
inferredcites →https://doi.org/10.1038/embor.2008.74openalex-snapshot
A reciprocal repression between ZEB1 and members of the miR‐200 family promotes EMT and invasion in cancer cells
inferredcites →https://doi.org/10.1038/ncb3157openalex-snapshot
Matrix stiffness drives epithelial–mesenchymal transition and tumour metastasis through a TWIST1–G3BP2 mechanotransduction pathway
inferredcites →https://doi.org/10.1038/ncomms7139openalex-snapshot
Temporally sequenced anticancer drugs overcome adaptive resistance by targeting a vulnerable chemotherapy-induced phenotypic transition
inferredcites →https://doi.org/10.1038/nm.3336openalex-snapshot
The epigenetics of epithelial-mesenchymal plasticity in cancer
inferredcites →https://doi.org/10.1038/nrc3447openalex-snapshot
Regulatory networks defining EMT during cancer initiation and progression
inferredcites →https://doi.org/10.1038/nrd4253openalex-snapshot
Tackling the cancer stem cells — what challenges do they pose?
inferredcites →https://doi.org/10.1038/onc.2008.226openalex-snapshot
Rational targeting of Notch signaling in cancer
inferredcites →https://doi.org/10.1039/c3mb70431aopenalex-snapshot
Extracellular matrix density promotes EMT by weakening cell–cell adhesions
inferredcites →https://doi.org/10.1039/c4ib00191eopenalex-snapshot
Cancer stem cells: small subpopulation or evolving fraction?
inferredcites →https://doi.org/10.1073/pnas.0802047105openalex-snapshot
Notch signaling mediates hypoxia-induced tumor cell migration and invasion
inferredcites →https://doi.org/10.1073/pnas.1002827107openalex-snapshot
Notch signaling specifies prosensory domains via lateral induction in the developing mammalian inner ear
inferredcites →https://doi.org/10.1073/pnas.1310490110openalex-snapshot
Wnt/Rspondin/β-catenin signals control axonal sorting and lineage progression in Schwann cell development
inferredcites →https://doi.org/10.1073/pnas.1318192110openalex-snapshot
MicroRNA-based regulation of epithelial–hybrid–mesenchymal fate determination
inferredcites →https://doi.org/10.1073/pnas.1416287112openalex-snapshot
Jagged–Delta asymmetry in Notch signaling can give rise to a Sender/Receiver hybrid phenotype
inferredcites →https://doi.org/10.1074/jbc.m206400200openalex-snapshot
Snail Induction of Epithelial to Mesenchymal Transition in Tumor Cells Is Accompanied by MUC1 Repression andZEB1 Expression
inferredcites →https://doi.org/10.1083/jcb.200710067openalex-snapshot
Slug is a direct Notch target required for initiation of cardiac cushion cellularization
inferredcites →https://doi.org/10.1088/1367-2630/17/5/055021openalex-snapshot
Operating principles of Notch–Delta–Jagged module of cell–cell communication
inferredcites →https://doi.org/10.1098/rsif.2012.0766openalex-snapshot
On the role of lateral stabilization during early patterning in the pancreas
inferredcites →https://doi.org/10.1098/rsif.2014.0962openalex-snapshot
Towards elucidating the connection between epithelial–mesenchymal transitions and stemness
inferredcites →https://doi.org/10.1101/gad.1640608openalex-snapshot
The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2
inferredcites →https://doi.org/10.1101/gad.225334.113openalex-snapshot
Epithelial–mesenchymal plasticity in carcinoma metastasis
inferredcites →https://doi.org/10.1126/science.1228522openalex-snapshot
Circulating Breast Tumor Cells Exhibit Dynamic Changes in Epithelial and Mesenchymal Composition
inferredcites →https://doi.org/10.1126/science.1234850openalex-snapshot
Epithelial Plasticity: A Common Theme in Embryonic and Cancer Cells
inferredcites →https://doi.org/10.1158/0008-5472.can-08-1942openalex-snapshot
A Double-Negative Feedback Loop between ZEB1-SIP1 and the microRNA-200 Family Regulates Epithelial-Mesenchymal Transition
inferredcites →https://doi.org/10.1158/0008-5472.can-14-0225openalex-snapshot
Network Modeling of TGFβ Signaling in Hepatocellular Carcinoma Epithelial-to-Mesenchymal Transition Reveals Joint Sonic Hedgehog and Wnt Pathway Activation
inferredcites →https://doi.org/10.1158/0008-5472.can-14-2535openalex-snapshot
Molecular Portraits of Epithelial, Mesenchymal, and Hybrid States in Lung Adenocarcinoma and Their Relevance to Survival
inferredcites →https://doi.org/10.1158/1541-7786.mcr-10-0490openalex-snapshot
Circulating Tumor Cells from Patients with Advanced Prostate and Breast Cancer Display Both Epithelial and Mesenchymal Markers
inferredcites →https://doi.org/10.1158/2159-8290.cd-14-0650openalex-snapshot
NOTCH Decoys That Selectively Block DLL/NOTCH or JAG/NOTCH Disrupt Angiogenesis by Unique Mechanisms to Inhibit Tumor Growth
inferredcites →https://doi.org/10.1161/circulationaha.111.047159openalex-snapshot
Notch Activation of Jagged1 Contributes to the Assembly of the Arterial Wall
inferredcites →https://doi.org/10.1172/jci32533openalex-snapshot
IL-6 triggers malignant features in mammospheres from human ductal breast carcinoma and normal mammary gland
inferredcites →https://doi.org/10.1172/jci63608openalex-snapshot
ZEB1 drives prometastatic actin cytoskeletal remodeling by downregulating miR-34a expression
inferredcites →https://doi.org/10.1242/dev.063610openalex-snapshot
Notch signaling: simplicity in design, versatility in function
inferredcites →https://doi.org/10.1242/dev.108100openalex-snapshot
Ligand-dependent Notch signaling strength orchestrates lateral induction and lateral inhibition in the developing inner ear
inferredcites →https://doi.org/10.1371/journal.pcbi.1002628openalex-snapshot
Hybrid Models and Biological Model Reduction with PyDSTool
inferredcites →https://doi.org/10.1371/journal.pcbi.1004025openalex-snapshot
Evolution and Phenotypic Selection of Cancer Stem Cells
inferredcites →https://doi.org/10.1371/journal.pone.0024584openalex-snapshot
MiR-34a Targeting of Notch Ligand Delta-Like 1 Impairs CD15+/CD133+ Tumor-Propagating Cells and Supports Neural Differentiation in Medulloblastoma
inferredcites →https://doi.org/10.1371/journal.pone.0096093openalex-snapshot
Tumour Control Probability in Cancer Stem Cells Hypothesis
inferredcites →https://doi.org/10.1371/journal.pone.0126522openalex-snapshot
Stemness of the hybrid Epithelial/Mesenchymal State in Breast Cancer and Its Association with Poor Survival
inferredcites →https://doi.org/10.1371/journal.pone.0135797openalex-snapshot
Modeling of Cancer Stem Cell State Transitions Predicts Therapeutic Response
inferredcites →https://doi.org/10.15252/emmm.201303698openalex-snapshot
Biology, detection, and clinical implications of circulating tumor cells
inferredcites →https://doi.org/10.1615/critrevoncog.2014011802openalex-snapshot
The EMT Universe: Space between Cancer Cell Dissemination and Metastasis Initiation
inferredcites →https://doi.org/10.18632/oncotarget.3623openalex-snapshot
OVOL guides the epithelial-hybrid-mesenchymal transition
inferredcites →https://doi.org/10.18632/oncotarget.4629openalex-snapshot
Coupling the modules of EMT and stemness: A tunable ‘stemness window’ model
inferredcites →https://doi.org/10.3389/fnins.2011.00078openalex-snapshot
Dynamic Expression of Notch Signaling Genes in Neural Stem/Progenitor Cells
inferredcites →https://doi.org/10.3389/fonc.2014.00254openalex-snapshot
The Notch ligand Jagged1 as a target for anti-tumor therapy
inferredcites →https://doi.org/10.3389/fonc.2015.00155openalex-snapshot
Implications of the Hybrid Epithelial/Mesenchymal Phenotype in Metastasis
inferredcites →https://doi.org/10.4161/15384101.2014.985071openalex-snapshot
Reinforcing targeted therapeutics with phenotypic stability factors
inferredcites →https://doi.org/10.4161/cc.10.24.18552openalex-snapshot
miR-34 and SNAIL form a double-negative feedback loop to regulate epithelial-mesenchymal transitions
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4768258openalex-extracted
Microenvironmental Entropy Dynamics Analysis Reveals Novel Insights In Notch-Delta-Jagged Decision-Making Mechanism
inferred← citeshttps://doi.org/10.1002/1878-0261.12083openalex-snapshot
EMT and MET : necessary or permissive for metastasis?
inferred← citeshttps://doi.org/10.1002/1878-0261.12084openalex-snapshot
Epithelial/mesenchymal plasticity: how have quantitative mathematical models helped improve our understanding?
inferred← citeshttps://doi.org/10.1002/1878-0261.70221openalex-snapshot
COMP – PMEPA1 axis promotes epithelial‐to‐mesenchymal transition in breast cancer cells
inferred← citeshttps://doi.org/10.1002/advs.202206744openalex-snapshot
m 6 A‐Dependent Modulation via IGF2BP3/MCM5/Notch Axis Promotes Partial EMT and LUAD Metastasis
inferred← citeshttps://doi.org/10.1002/cso2.1015openalex-snapshot
Investigating epithelial‐mesenchymal heterogeneity of tumors and circulating tumor cells with transcriptomic analysis and biophysical modeling
inferred← citeshttps://doi.org/10.1002/cso2.1021openalex-snapshot
Computational systems‐biology approaches for modeling gene networks driving epithelial–mesenchymal transitions
inferred← citeshttps://doi.org/10.1002/dvdy.24506openalex-snapshot
Epithelial–mesenchymal plasticity and circulating tumor cells: Travel companions to metastases
inferred← citeshttps://doi.org/10.1002/dvdy.24541openalex-snapshot
Epithelial–mesenchymal transition, a spectrum of states: Role in lung development, homeostasis, and disease
inferred← citeshttps://doi.org/10.1002/wsbm.1488openalex-snapshot
Computational models to explore the complexity of the epithelial to mesenchymal transition in cancer
inferred← citeshttps://doi.org/10.1007/978-1-0716-0779-4_28openalex-snapshot
Mathematical Modeling of Plasticity and Heterogeneity in EMT
inferred← citeshttps://doi.org/10.1007/978-1-4939-8982-9_14openalex-snapshot
Quantitative Characteristic of ncRNA Regulation in Gene Regulatory Networks
inferred← citeshttps://doi.org/10.1007/978-3-030-32857-3_10openalex-snapshot
Critical Steps in Epithelial-Mesenchymal Transition as Target for Cancer Treatment
inferred← citeshttps://doi.org/10.1007/978-3-030-91311-3_8openalex-snapshot
Cancer Stem Cells: An Ever-Hiding Foe
inferred← citeshttps://doi.org/10.1007/978-3-031-22802-5_3openalex-snapshot
Biophysical and Biochemical Mechanisms Underlying Collective Cell Migration in Cancer Metastasis
inferred← citeshttps://doi.org/10.1007/s10549-022-06819-6openalex-snapshot
Geometric tumor embolic budding characterizes inflammatory breast cancer
inferred← citeshttps://doi.org/10.1007/s10585-021-10111-0openalex-snapshot
Collective metastasis: coordinating the multicellular voyage
inferred← citeshttps://doi.org/10.1007/s40778-019-0150-3openalex-snapshot
Deciphering the Dynamics of Epithelial-Mesenchymal Transition and Cancer Stem Cells in Tumor Progression
inferred← citeshttps://doi.org/10.1007/s41745-020-00179-0openalex-snapshot
Cellular Plasticity in Matrix-attached and -Detached Cells: Implications in Metastasis
inferred← citeshttps://doi.org/10.1016/b978-0-12-817996-3.00018-9openalex-snapshot
Epithelial-mesenchymal transition in cancer
inferred← citeshttps://doi.org/10.1016/j.bbcan.2018.07.001openalex-snapshot
Hybrid epithelial/mesenchymal phenotype(s): The ‘fittest’ for metastasis?
inferred← citeshttps://doi.org/10.1016/j.biopha.2021.111314openalex-snapshot
Heterogeneity of CTC contributes to the organotropism of breast cancer
inferred← citeshttps://doi.org/10.1016/j.bpj.2024.05.005openalex-snapshot
Spatial heterogeneity in tumor adhesion qualifies collective cell invasion
inferred← citeshttps://doi.org/10.1016/j.ccell.2021.05.005openalex-snapshot
Single-cell lineage tracing of metastatic cancer reveals selection of hybrid EMT states
inferred← citeshttps://doi.org/10.1016/j.celrep.2025.116909openalex-snapshot
A Jagged1-regulated hybrid-EMT state identifies pancreatic cancer stem cells
inferred← citeshttps://doi.org/10.1016/j.coisb.2017.02.004openalex-snapshot
Computational systems biology of epithelial-hybrid-mesenchymal transitions
inferred← citeshttps://doi.org/10.1016/j.ijnonlinmec.2022.104063openalex-snapshot
A hybrid modeling environment to describe aggregates of cells heterogeneous for genotype and behavior with possible phenotypic transitions
inferred← citeshttps://doi.org/10.1016/j.isci.2024.110310openalex-snapshot
Cell-state transitions and density-dependent interactions together explain the dynamics of spontaneous epithelial-mesenchymal heterogeneity
inferred← citeshttps://doi.org/10.1016/j.isci.2024.110569openalex-snapshot
Microenvironmental entropy dynamics analysis reveals novel insights into Notch-Delta-Jagged decision-making mechanism
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2018.09.007openalex-snapshot
Hybrid epithelial/mesenchymal phenotypes promote metastasis and therapy resistance across carcinomas
inferred← citeshttps://doi.org/10.1016/j.semcancer.2023.02.001openalex-snapshot
A role for partial epithelial-to-mesenchymal transition in enabling stemness in homeostasis and cancer
inferred← citeshttps://doi.org/10.1016/j.semcancer.2023.09.007openalex-snapshot
Dynamical hallmarks of cancer: Phenotypic switching in melanoma and epithelial-mesenchymal plasticity
inferred← citeshttps://doi.org/10.1016/j.stemcr.2018.03.001openalex-snapshot
Heterogeneity of Human Breast Stem and Progenitor Cells as Revealed by Transcriptional Profiling
inferred← citeshttps://doi.org/10.1016/j.tranon.2017.02.007openalex-snapshot
Detection of Circulating Tumor Cells and Circulating Tumor Microemboli in Gastric Cancer
inferred← citeshttps://doi.org/10.1016/j.tranon.2020.100845openalex-snapshot
Hypoxia, partial EMT and collective migration: Emerging culprits in metastasis
inferred← citeshttps://doi.org/10.1017/erm.2017.6openalex-snapshot
Epithelial, mesenchymal and hybrid epithelial/mesenchymal phenotypes and their clinical relevance in cancer metastasis
inferred← citeshttps://doi.org/10.1021/acs.chemrev.0c00356openalex-snapshot
Engineering in Medicine To Address the Challenge of Cancer Drug Resistance: From Micro- and Nanotechnologies to Computational and Mathematical Modeling
inferred← citeshttps://doi.org/10.1021/acsapm.9b00334openalex-snapshot
A Supramolecular Platform for the Introduction of Fc-Fusion Bioactive Proteins on Biomaterial Surfaces
inferred← citeshttps://doi.org/10.1021/acsomega.2c07989openalex-snapshot
Dynamics of Epithelial–Mesenchymal Plasticity: What Have Single-Cell Investigations Elucidated So Far?
inferred← citeshttps://doi.org/10.1038/s41416-022-01768-9openalex-snapshot
Circulating tumour cells in the -omics era: how far are we from achieving the ‘singularity’?
inferred← citeshttps://doi.org/10.1038/s41416-025-03074-6openalex-snapshot
Wnt/ERK/CDK4/6 activation in the partial EMT state coordinates mammary cancer stemness with self-renewal and inhibition of differentiation
inferred← citeshttps://doi.org/10.1038/s41523-017-0023-9openalex-snapshot
Inflammatory breast cancer: a model for investigating cluster-based dissemination
inferred← citeshttps://doi.org/10.1038/s41540-025-00502-4openalex-snapshot
An integrative phenotype-structured partial differential equation model for the population dynamics of epithelial-mesenchymal transition
inferred← citeshttps://doi.org/10.1038/s41568-021-00376-8openalex-snapshot
Decoding leader cells in collective cancer invasion
inferred← citeshttps://doi.org/10.1038/s41576-023-00601-0openalex-snapshot
The epithelial–mesenchymal plasticity landscape: principles of design and mechanisms of regulation
inferred← citeshttps://doi.org/10.1038/s41580-025-00858-9openalex-snapshot
Collective cell migration modes in development, tissue repair and cancer
inferred← citeshttps://doi.org/10.1039/d0ma00455copenalex-snapshot
Nanoparticle mediated alteration of EMT dynamics: an approach to modulate cancer therapeutics
inferred← citeshttps://doi.org/10.1039/d2sm01508kopenalex-snapshot
The alternate ligand Jagged enhances the robustness of Notch signaling patterns
inferred← citeshttps://doi.org/10.1063/5.0302960openalex-snapshot
Precursors for cell-state transitions: Stability, resilience, and predictability in Notch signaling pathways
inferred← citeshttps://doi.org/10.1073/pnas.1815345116openalex-snapshot
Toward understanding cancer stem cell heterogeneity in the tumor microenvironment
inferred← citeshttps://doi.org/10.1073/pnas.2102050118openalex-snapshot
Identification of EMT signaling cross-talk and gene regulatory networks by single-cell RNA sequencing
inferred← citeshttps://doi.org/10.1080/15384047.2026.2725344openalex-snapshot
Fibroblast-associated TPM2 links cell-matrix remodeling to EMT-Notch signaling and gemcitabine resistance in intrahepatic cholangiocarcinoma
inferred← citeshttps://doi.org/10.1088/1478-3975/aaffa1openalex-snapshot
Towards control of cellular decision-making networks in the epithelial-to-mesenchymal transition
inferred← citeshttps://doi.org/10.1088/1478-3975/ab34dfopenalex-snapshot
A possible role for epigenetic feedback regulation in the dynamics of the epithelial–mesenchymal transition (EMT)
inferred← citeshttps://doi.org/10.1088/1478-3975/ac28a4openalex-snapshot
Ordered hexagonal patterns via notch–delta signaling
inferred← citeshttps://doi.org/10.1088/1478-3975/ae2c34openalex-snapshot
The interaction between dynamic ligand signaling and epigenetics in Notch-induced cancer metastasis
inferred← citeshttps://doi.org/10.1088/1478-3975/ae5016openalex-snapshot
Androgen receptor activation stabilizes a hybrid epithelial/mesenchymal phenotype in presence of Notch-Jagged signaling
inferred← citeshttps://doi.org/10.1088/2057-1976/ac7896openalex-snapshot
SNAIL driven by a feed forward loop motif promotes TGF β induced epithelial to mesenchymal transition
inferred← citeshttps://doi.org/10.1089/ten.teb.2022.0120openalex-snapshot
Engineering Strategies to Move from Understanding to Steering Renal Tubulogenesis
inferred← citeshttps://doi.org/10.1093/jb/mvy063openalex-snapshot
Epithelial-mesenchymal transition in haematopoietic stem cell development and homeostasis
inferred← citeshttps://doi.org/10.1093/nar/gkaa725openalex-snapshot
Inference and multiscale model of epithelial-to-mesenchymal transition via single-cell transcriptomic data
inferred← citeshttps://doi.org/10.1096/fj.202300120rropenalex-snapshot
Epithelial multicellular clustering enabled by polarized macrophages on soft matrices
inferred← citeshttps://doi.org/10.1098/rsif.2017.0512openalex-snapshot
Numb prevents a complete epithelial–mesenchymal transition by modulating Notch signalling
inferred← citeshttps://doi.org/10.1098/rsif.2022.0627openalex-snapshot
Epigenetic memory acquired during long-term EMT induction governs the recovery to the epithelial state
inferred← citeshttps://doi.org/10.1101/2021.04.21.440858openalex-snapshot
Nrf2 modulates the hybrid epithelial/mesenchymal phenotype and Notch signaling during collective cancer migration
inferred← citeshttps://doi.org/10.1101/2021.11.16.468748openalex-snapshot
Lattice-based microenvironmental uncertainty driven phenotypic decision-making: a comparison with Notch-Delta-Jagged signaling
inferred← citeshttps://doi.org/10.1101/2022.08.06.503043openalex-snapshot
A spatialised agent-based model of NOTCH signalling pathway in Endothelial Cells predicts emergent heterogeneity due to continual dynamic phenotypic adjustments
inferred← citeshttps://doi.org/10.1101/2023.12.07.567986openalex-snapshot
Cell-state transitions and frequency-dependent interactions among subpopulations together explain the dynamics of spontaneous epithelial-mesenchymal heterogeneity in breast cancer
inferred← citeshttps://doi.org/10.1101/2024.08.06.606905openalex-snapshot
Multiple Notch ligands in the synchronization of the segmentation clock
inferred← citeshttps://doi.org/10.1101/347815openalex-snapshot
E-cadherin represses anchorage-independent growth in sarcomas through both signaling and mechanical mechanisms
inferred← citeshttps://doi.org/10.1101/563049openalex-snapshot
A biophysical model of Epithelial-Mesenchymal Transition uncovers the frequency and size distribution of Circulating Tumor Cell clusters across cancer types
inferred← citeshttps://doi.org/10.1101/cshperspect.a036905openalex-snapshot
Cancer Stem Cells and Epithelial-to-Mesenchymal Transition in Cancer Metastasis
inferred← citeshttps://doi.org/10.1103/7g6x-b238openalex-snapshot
Multiple Notch ligands in the synchronization of the segmentation clock
inferred← citeshttps://doi.org/10.1126/scisignal.aas8779openalex-snapshot
Targeting tumor phenotypic plasticity and metabolic remodeling in adaptive cross-drug tolerance
inferred← citeshttps://doi.org/10.1146/annurev-pathmechdis-031521-023557openalex-snapshot
Orchestration of Collective Migration and Metastasis by Tumor Cell Clusters
inferred← citeshttps://doi.org/10.1158/0008-5472.can-16-2874openalex-snapshot
MCAM Mediates Chemoresistance in Small-Cell Lung Cancer via the PI3K/AKT/SOX2 Signaling Pathway
inferred← citeshttps://doi.org/10.1158/0008-5472.can-16-3521openalex-snapshot
Survival Outcomes in Cancer Patients Predicted by a Partial EMT Gene Expression Scoring Metric
inferred← citeshttps://doi.org/10.1158/0008-5472.can-24-1703openalex-snapshot
Modeling Drug Responses and Evolutionary Dynamics Using Patient-Derived Xenografts Reveals Precision Medicine Strategies for Triple-Negative Breast Cancer
inferred← citeshttps://doi.org/10.1158/1541-7786.mcr-18-0763openalex-snapshot
E-Cadherin Represses Anchorage-Independent Growth in Sarcomas through Both Signaling and Mechanical Mechanisms
inferred← citeshttps://doi.org/10.1186/s12885-018-4640-yopenalex-snapshot
Bulk tumour cell migration in lung carcinomas might be more common than epithelial-mesenchymal transition and be differently regulated
inferred← citeshttps://doi.org/10.1186/s12964-022-00885-5openalex-snapshot
Optogenetic control of NOTCH1 signaling
inferred← citeshttps://doi.org/10.1186/s12964-025-02405-7openalex-snapshot
Intercellular signaling reinforces single-cell level phenotypic transitions and facilitates robust re-equilibrium of heterogeneous cancer cell populations
inferred← citeshttps://doi.org/10.1186/s13058-018-0963-5openalex-snapshot
Dependence receptor UNC5A restricts luminal to basal breast cancer plasticity and metastasis
inferred← citeshttps://doi.org/10.1186/s41236-017-0005-8openalex-snapshot
Distinguishing mechanisms underlying EMT tristability
inferred← citeshttps://doi.org/10.1186/s43556-022-00099-8openalex-snapshot
The role of Hedgehog and Notch signaling pathway in cancer
inferred← citeshttps://doi.org/10.1242/jcs.250225openalex-snapshot
Combined heterogeneity in cell size and deformability promotes cancer invasiveness
inferred← citeshttps://doi.org/10.1242/jcs.259104openalex-snapshot
High-content image analysis to study phenotypic heterogeneity in endothelial cell monolayers
inferred← citeshttps://doi.org/10.1371/journal.pcbi.1006919openalex-snapshot
Notch signaling and taxis mechanisms regulate early stage angiogenesis: A mathematical and computational model
inferred← citeshttps://doi.org/10.1371/journal.pcbi.1007619openalex-snapshot
A mechanism for epithelial-mesenchymal heterogeneity in a population of cancer cells
inferred← citeshttps://doi.org/10.1371/journal.pcbi.1009011openalex-snapshot
Cluster size distribution of cells disseminating from a primary tumor
inferred← citeshttps://doi.org/10.1371/journal.pcbi.1012465openalex-snapshot
Emergent order in epithelial sheets by interplay of cell divisions and cell fate regulation
inferred← citeshttps://doi.org/10.1371/journal.pone.0291104openalex-snapshot
Increased collective migration correlates with germline stem cell competition in a basal chordate
inferred← citeshttps://doi.org/10.18632/oncotarget.25692openalex-snapshot
A mechanism-based computational model to capture the interconnections among epithelial-mesenchymal transition, cancer stem cells and Notch-Jagged signaling
inferred← citeshttps://doi.org/10.21203/rs.3.rs-1377051/v1openalex-snapshot
Geometric Tumor Embolic Budding Characterizes Inflammatory Breast Cancer
inferred← citeshttps://doi.org/10.24275/uami.jm214p263openalex-snapshot
Mecanismo de acción de la dehidroepiandrosterona en la transición mesénquima-epitelio en célula de cáncer de mama MDA-MB-231
inferred← citeshttps://doi.org/10.3389/fcell.2022.1038841openalex-snapshot
“In medio stat virtus”: Insights into hybrid E/M phenotype attitudes
inferred← citeshttps://doi.org/10.3389/fgene.2020.604528openalex-snapshot
Phenotypic Heterogeneity in Tumor Progression, and Its Possible Role in the Onset of Cancer
inferred← citeshttps://doi.org/10.3389/fgene.2020.604585openalex-snapshot
Inference of Intercellular Communications and Multilayer Gene-Regulations of Epithelial–Mesenchymal Transition From Single-Cell Transcriptomic Data
inferred← citeshttps://doi.org/10.3389/fmolb.2022.807324openalex-snapshot
Nrf2 Modulates the Hybrid Epithelial/Mesenchymal Phenotype and Notch Signaling During Collective Cancer Migration
inferred← citeshttps://doi.org/10.3389/fonc.2017.00162openalex-snapshot
Deciphering Epithelial–Mesenchymal Transition Regulatory Networks in Cancer through Computational Approaches
inferred← citeshttps://doi.org/10.3389/fonc.2018.00050openalex-snapshot
Phenotypic Plasticity, Bet-Hedging, and Androgen Independence in Prostate Cancer: Role of Non-Genetic Heterogeneity
inferred← citeshttps://doi.org/10.3389/fonc.2018.00244openalex-snapshot
Analysis of Hierarchical Organization in Gene Expression Networks Reveals Underlying Principles of Collective Tumor Cell Dissemination and Metastatic Aggressiveness of Inflammatory Breast Cancer
inferred← citeshttps://doi.org/10.3389/fphys.2020.00929openalex-snapshot
Understanding the Principles of Pattern Formation Driven by Notch Signaling by Integrating Experiments and Theoretical Models
inferred← citeshttps://doi.org/10.3389/fsysb.2023.1099951openalex-snapshot
Network motifs and hypermotifs in TGFβ-induced epithelial to mesenchymal transition and metastasis
inferred← citeshttps://doi.org/10.3390/cancers12061632openalex-snapshot
EMT-Associated Heterogeneity in Circulating Tumor Cells: Sticky Friends on the Road to Metastasis
inferred← citeshttps://doi.org/10.3390/cancers12103065openalex-snapshot
Circulating Tumor Cells: From the Laboratory to the Cancer Clinic
inferred← citeshttps://doi.org/10.3390/cancers13112795openalex-snapshot
Cell Plasticity and Prostate Cancer: The Role of Epithelial–Mesenchymal Transition in Tumor Progression, Invasion, Metastasis and Cancer Therapy Resistance
inferred← citeshttps://doi.org/10.3390/cancers13246219openalex-snapshot
Shooting at Moving and Hidden Targets—Tumour Cell Plasticity and the Notch Signalling Pathway in Head and Neck Squamous Cell Carcinomas
inferred← citeshttps://doi.org/10.3390/cancers16193289openalex-snapshot
Epithelial–Mesenchymal Plasticity and Epigenetic Heterogeneity in Cancer
inferred← citeshttps://doi.org/10.3390/cancers9070070openalex-snapshot
Phenotypic Plasticity and Cell Fate Decisions in Cancer: Insights from Dynamical Systems Theory
inferred← citeshttps://doi.org/10.3390/cancers9080104openalex-snapshot
Complex Determinants of Epithelial: Mesenchymal Phenotypic Plasticity in Ovarian Cancer
inferred← citeshttps://doi.org/10.3390/cells10010094openalex-snapshot
Context Matters: NOTCH Signatures and Pathway in Cancer Progression and Metastasis
inferred← citeshttps://doi.org/10.3390/e23030288openalex-snapshot
Coupled Feedback Loops Involving PAGE4, EMT and Notch Signaling Can Give Rise to Non-Genetic Heterogeneity in Prostate Cancer Cells
inferred← citeshttps://doi.org/10.3390/e23070867openalex-snapshot
Close to Optimal Cell Sensing Ensures the Robustness of Tissue Differentiation Process: The Avian Photoreceptor Mosaic Case
inferred← citeshttps://doi.org/10.3390/ijms21165691openalex-snapshot
Notch Transduction in Non-Small Cell Lung Cancer
inferred← citeshttps://doi.org/10.3390/ijms252212047openalex-snapshot
Characterization and Experimental Use of Multiple Myeloma Bone Marrow Endothelial Cells and Progenitors
inferred← citeshttps://doi.org/10.3390/jcm8050725openalex-snapshot
Quantifying Cancer Epithelial-Mesenchymal Plasticity and its Association with Stemness and Immune Response
inferred← citeshttps://doi.org/10.3390/jcm8101542openalex-snapshot
Dynamics of Phenotypic Heterogeneity Associated with EMT and Stemness during Cancer Progression
inferred← citeshttps://doi.org/10.3410/f.734883241.793575374openalex-snapshot
Faculty Opinions recommendation of Towards control of cellular decision-making networks in the epithelial-to-mesenchymal transition.
inferred← citeshttps://doi.org/10.3892/or.2017.5625openalex-snapshot
Chloroform fraction of Scutellaria barbata D. Don inhibits the growth of colorectal cancer cells by activating miR-34a
inferred← citeshttps://doi.org/10.6084/m9.figshare.19793288openalex-snapshot
Additional file 2 of Optogenetic control of NOTCH1 signaling
inferred← citeshttps://doi.org/10.70675/1e724eebz5eeaz48f0za621z4d96e4fb08e2openalex-snapshot
Rôle de la protéine sécrétée OLFM4 dans la carcinogénèse colorectale et mammaire : importance du contexte cellulaire
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.