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Quantifying Cancer Epithelial-Mesenchymal Plasticity and its Association with Stemness and Immune Response

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Outgoing claims: the papers this work cites, builds on, or argues with.

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EMT and MET : necessary or permissive for metastasis?

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Epithelial/mesenchymal plasticity: how have quantitative mathematical models helped improve our understanding?

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New insights into the role of EMT in tumor immune escape

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Epithelial–mesenchymal transition, a spectrum of states: Role in lung development, homeostasis, and disease

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Soluble factors regulated by epithelial–mesenchymal transition mediate tumour angiogenesis and myeloid cell recruitment

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Pattern Formation by Lateral Inhibition with Feedback: a Mathematical Model of Delta-Notch Intercellular Signalling

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Quantitative Characteristic of ncRNA Regulation in Gene Regulatory Networks

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Structural-Mechanical and Biochemical Functions of Classical Cadherins at Cellular Junctions: A Review and Some Hypotheses

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Epithelial-mesenchymal transition in tissue repair and fibrosis

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CCL2 is critical for immunosuppression to promote cancer metastasis

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Collective invasion in ductal and lobular breast cancer associates with distant metastasis

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CTC clusters in cancer progression and metastasis

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Regulation of Epithelial-Mesenchymal Transition by Transmission of Mechanical Stress through Epithelial Tissues

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Deciphering the Dynamics of Epithelial-Mesenchymal Transition and Cancer Stem Cells in Tumor Progression

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Mechanical Force Affects Expression of an In Vitro Metastasis-Like Phenotype in HCT-8 Cells

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A Computational Model of YAP/TAZ Mechanosensing

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TGF-β directly targets cytotoxic T cell functions during tumor evasion of immune surveillance

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Cancer Metastasis Is Accelerated through Immunosuppression during Snail-Induced EMT of Cancer Cells

inferredcites →https://doi.org/10.1016/j.ccr.2012.02.022openalex-snapshot

Accessories to the Crime: Functions of Cells Recruited to the Tumor Microenvironment

inferredcites →https://doi.org/10.1016/j.ccr.2012.09.022openalex-snapshot

Spatiotemporal Regulation of Epithelial-Mesenchymal Transition Is Essential for Squamous Cell Carcinoma Metastasis

inferredcites →https://doi.org/10.1016/j.ccr.2012.10.012openalex-snapshot

Metastatic Colonization Requires the Repression of the Epithelial-Mesenchymal Transition Inducer Prrx1

inferredcites →https://doi.org/10.1016/j.ccr.2014.01.009openalex-snapshot

Identification of Distinct Basal and Luminal Subtypes of Muscle-Invasive Bladder Cancer with Different Sensitivities to Frontline Chemotherapy

inferredcites →https://doi.org/10.1016/j.ccr.2014.03.021openalex-snapshot

A Positive Feedback Loop between Mesenchymal-like Cancer Cells and Macrophages Is Essential to Breast Cancer Metastasis

inferredcites →https://doi.org/10.1016/j.cell.2008.03.027openalex-snapshot

The Epithelial-Mesenchymal Transition Generates Cells with Properties of Stem Cells

inferredcites →https://doi.org/10.1016/j.cell.2014.07.013openalex-snapshot

Circulating Tumor Cell Clusters Are Oligoclonal Precursors of Breast Cancer Metastasis

inferredcites →https://doi.org/10.1016/j.cell.2016.06.028openalex-snapshot

EMT: 2016

inferredcites →https://doi.org/10.1016/j.cell.2017.10.044openalex-snapshot

Single-Cell Transcriptomic Analysis of Primary and Metastatic Tumor Ecosystems in Head and Neck Cancer

inferredcites →https://doi.org/10.1016/j.celrep.2015.12.064openalex-snapshot

let-7 Modulates Chromatin Configuration and Target Gene Repression through Regulation of the ARID3B Complex

inferredcites →https://doi.org/10.1016/j.cels.2018.05.019openalex-snapshot

Combinatorial Targeting by MicroRNAs Co-ordinates Post-transcriptional Control of EMT

inferredcites →https://doi.org/10.1016/j.cmet.2017.04.017openalex-snapshot

Hybrid Cellular Metabolism Coordinated by Zic3 and Esrrb Synergistically Enhances Induction of Naive Pluripotency

inferredcites →https://doi.org/10.1016/j.cytogfr.2014.07.014openalex-snapshot

TGFβ in T cell biology and tumor immunity: Angel or devil?

inferredcites →https://doi.org/10.1016/j.devcel.2008.03.003openalex-snapshot

Collective Epithelial Migration and Cell Rearrangements Drive Mammary Branching Morphogenesis

inferredcites →https://doi.org/10.1016/j.devcel.2018.05.027openalex-snapshot

EMT Subtype Influences Epithelial Plasticity and Mode of Cell Migration

inferredcites →https://doi.org/10.1016/j.ebiom.2016.01.007openalex-snapshot

Phenotypic Plasticity Determines Cancer Stem Cell Therapeutic Resistance in Oral Squamous Cell Carcinoma

inferredcites →https://doi.org/10.1016/j.gde.2011.07.007openalex-snapshot

From Notch signaling to fine-grained patterning: Modeling meets experiments

inferredcites →https://doi.org/10.1016/j.immuni.2018.06.006openalex-snapshot

Tumor Cell-Intrinsic Factors Underlie Heterogeneity of Immune Cell Infiltration and Response to Immunotherapy

inferredcites →https://doi.org/10.1016/j.jtbi.2011.10.027openalex-snapshot

A mathematical model of tumor–immune interactions

inferredcites →https://doi.org/10.1016/j.jtbi.2014.02.028openalex-snapshot

A mathematical model for pancreatic cancer growth and treatments

inferredcites →https://doi.org/10.1016/j.jtbi.2014.04.031openalex-snapshot

Mathematical model of macrophage-facilitated breast cancer cells invasion

inferredcites →https://doi.org/10.1016/j.jtbi.2015.10.034openalex-snapshot

The re-polarisation of M2 and M1 macrophages and its role on cancer outcomes

inferredcites →https://doi.org/10.1016/j.jtbi.2018.05.003openalex-snapshot

Modeling triple-negative breast cancer heterogeneity: Effects of stromal macrophages, fibroblasts and tumor vasculature

inferredcites →https://doi.org/10.1016/j.jtbi.2019.03.002openalex-snapshot

Mathematical modeling of tumor-immune cell interactions

inferredcites →https://doi.org/10.1016/j.molimm.2014.12.017openalex-snapshot

TGF-β and EGF induced HLA-I downregulation is associated with epithelial-mesenchymal transition (EMT) through upregulation of snail in prostate cancer cells

inferredcites →https://doi.org/10.1016/j.pharmthera.2018.09.007openalex-snapshot

Hybrid epithelial/mesenchymal phenotypes promote metastasis and therapy resistance across carcinomas

inferredcites →https://doi.org/10.1016/j.semcancer.2017.08.002openalex-snapshot

Epithelial-mesenchymal transition and inflammation at the site of the primary tumor

inferredcites →https://doi.org/10.1016/j.stem.2013.03.002openalex-snapshot

A microRNA miR-34a-Regulated Bimodal Switch Targets Notch in Colon Cancer Stem Cells

inferredcites →https://doi.org/10.1016/j.stemcr.2013.11.009openalex-snapshot

Breast Cancer Stem Cells Transition between Epithelial and Mesenchymal States Reflective of their Normal Counterparts

inferredcites →https://doi.org/10.1016/j.stemcr.2018.03.001openalex-snapshot

Heterogeneity of Human Breast Stem and Progenitor Cells as Revealed by Transcriptional Profiling

inferredcites →https://doi.org/10.1016/j.tcb.2015.07.012openalex-snapshot

Epithelial–Mesenchymal Plasticity: A Central Regulator of Cancer Progression

inferredcites →https://doi.org/10.1016/j.tcb.2016.07.003openalex-snapshot

Intravital Correlative Microscopy: Imaging Life at the Nanoscale

inferredcites →https://doi.org/10.1016/j.tcb.2018.12.001openalex-snapshot

EMT Transition States during Tumor Progression and Metastasis

inferredcites →https://doi.org/10.1016/j.ydbio.2011.12.041openalex-snapshot

Neural crest delamination and migration: From epithelium-to-mesenchyme transition to collective cell migration

inferredcites →https://doi.org/10.1016/s0140-6736(16)00561-4openalex-snapshot

Atezolizumab in patients with locally advanced and metastatic urothelial carcinoma who have progressed following treatment with platinum-based chemotherapy: a single-arm, multicentre, phase 2 trial

inferredcites →https://doi.org/10.1038/35102167openalex-snapshot

Stem cells, cancer, and cancer stem cells

inferredcites →https://doi.org/10.1038/41580.1471-0080openalex-snapshot

Nature Reviews Molecular Cell Biology

inferredcites →https://doi.org/10.1038/bjc.2012.126openalex-snapshot

Dynamic equilibrium between cancer stem cells and non-stem cancer cells in human SW620 and MCF-7 cancer cell populations

inferredcites →https://doi.org/10.1038/bjc.2013.634openalex-snapshot

Tumour-infiltrating CD8+ lymphocytes as an independent predictive factor for pathological complete response to primary systemic therapy in breast cancer

inferredcites →https://doi.org/10.1038/bjc.2013.768openalex-snapshot

Cancer-associated fibroblasts induce epithelial–mesenchymal transition of breast cancer cells through paracrine TGF-β signalling

inferredcites →https://doi.org/10.1038/bjc.2015.29openalex-snapshot

Epithelial-to-mesenchymal transition (EMT) induced by inflammatory priming elicits mesenchymal stromal cell-like immune-modulatory properties in cancer cells

inferredcites →https://doi.org/10.1038/cddis.2013.442openalex-snapshot

An EMT spectrum defines an anoikis-resistant and spheroidogenic intermediate mesenchymal state that is sensitive to e-cadherin restoration by a src-kinase inhibitor, saracatinib (AZD0530)

inferredcites →https://doi.org/10.1038/cr.2009.5openalex-snapshot

TGF-β-induced epithelial to mesenchymal transition

inferredcites →https://doi.org/10.1038/emboj.2010.349openalex-snapshot

The ZEB1/miR‐200 feedback loop controls Notch signalling in cancer cells

inferredcites →https://doi.org/10.1038/nature10137openalex-snapshot

Role of YAP/TAZ in mechanotransduction

inferredcites →https://doi.org/10.1038/nature14404openalex-snapshot

Melanoma-intrinsic β-catenin signalling prevents anti-tumour immunity

inferredcites →https://doi.org/10.1038/nature15748openalex-snapshot

Epithelial-to-mesenchymal transition is not required for lung metastasis but contributes to chemoresistance

inferredcites →https://doi.org/10.1038/nature16064openalex-snapshot

Epithelial-to-mesenchymal transition is dispensable for metastasis but induces chemoresistance in pancreatic cancer

inferredcites →https://doi.org/10.1038/nbt.3973openalex-snapshot

Multiplexed quantification of proteins and transcripts in single cells

inferredcites →https://doi.org/10.1038/ncb3157openalex-snapshot

Matrix stiffness drives epithelial–mesenchymal transition and tumour metastasis through a TWIST1–G3BP2 mechanotransduction pathway

inferredcites →https://doi.org/10.1038/ncb3513openalex-snapshot

The EMT-activator Zeb1 is a key factor for cell plasticity and promotes metastasis in pancreatic cancer

inferredcites →https://doi.org/10.1038/ncb3611openalex-snapshot

Cell plasticity in epithelial homeostasis and tumorigenesis

inferredcites →https://doi.org/10.1038/ncomms15095openalex-snapshot

Spatial computation of intratumoral T cells correlates with survival of patients with pancreatic cancer

inferredcites →https://doi.org/10.1038/ncomms15773openalex-snapshot

EMT cells increase breast cancer metastasis via paracrine GLI activation in neighbouring tumour cells

inferredcites →https://doi.org/10.1038/ncomms3299openalex-snapshot

NF-κB non-cell-autonomously regulates cancer stem cell populations in the basal-like breast cancer subtype

inferredcites →https://doi.org/10.1038/ncomms6241openalex-snapshot

Metastasis is regulated via microRNA-200/ZEB1 axis control of tumour cell PD-L1 expression and intratumoral immunosuppression

inferredcites →https://doi.org/10.1038/ncomms7139openalex-snapshot

Temporally sequenced anticancer drugs overcome adaptive resistance by targeting a vulnerable chemotherapy-induced phenotypic transition

inferredcites →https://doi.org/10.1038/ncomms8998openalex-snapshot

A role for E-cadherin in ensuring cohesive migration of a heterogeneous population of non-epithelial cells

inferredcites →https://doi.org/10.1038/ng.729openalex-snapshot

Non-genetic heterogeneity from stochastic partitioning at cell division

inferredcites →https://doi.org/10.1038/ni.2703openalex-snapshot

Innate and adaptive immune cells in the tumor microenvironment

inferredcites →https://doi.org/10.1038/nm.3336openalex-snapshot

The epigenetics of epithelial-mesenchymal plasticity in cancer

inferredcites →https://doi.org/10.1038/nmeth.3404openalex-snapshot

A microfluidic device for label-free, physical capture of circulating tumor cell clusters

inferredcites →https://doi.org/10.1038/nmeth.4380openalex-snapshot

Simultaneous epitope and transcriptome measurement in single cells

inferredcites →https://doi.org/10.1038/npjsba.2015.14openalex-snapshot

Combinatorial interventions inhibit TGFβ-driven epithelial-to-mesenchymal transition and support hybrid cellular phenotypes

inferredcites →https://doi.org/10.1038/nrc.2017.118openalex-snapshot

EMT in cancer

inferredcites →https://doi.org/10.1038/nrc1694openalex-snapshot

Migrating cancer stem cells — an integrated concept of malignant tumour progression

inferredcites →https://doi.org/10.1038/nrc1740openalex-snapshot

The origin of the cancer stem cell: current controversies and new insights

inferredcites →https://doi.org/10.1038/nrc2355openalex-snapshot

Adoptive cell transfer: a clinical path to effective cancer immunotherapy

inferredcites →https://doi.org/10.1038/nrc3239openalex-snapshot

The blockade of immune checkpoints in cancer immunotherapy

inferredcites →https://doi.org/10.1038/nrc3447openalex-snapshot

Regulatory networks defining EMT during cancer initiation and progression

inferredcites →https://doi.org/10.1038/nrc822openalex-snapshot

Epithelial–mesenchymal transitions in tumour progression

inferredcites →https://doi.org/10.1038/nrclinonc.2017.44openalex-snapshot

EMT, CSCs, and drug resistance: the mechanistic link and clinical implications

inferredcites →https://doi.org/10.1038/nrm.2016.76openalex-snapshot

Epigenetic control of adult stem cell function

inferredcites →https://doi.org/10.1038/nrm.2016.94openalex-snapshot

Notch signalling in context

inferredcites →https://doi.org/10.1038/nrurol.2014.195openalex-snapshot

NUMB inhibition of NOTCH signalling as a therapeutic target in prostate cancer

inferredcites →https://doi.org/10.1038/onc.2008.271openalex-snapshot

The tumor microenvironment and its role in promoting tumor growth

inferredcites →https://doi.org/10.1038/onc.2010.619openalex-snapshot

TGF-β-induced IRAK-M expression in tumor-associated macrophages regulates lung tumor growth

inferredcites →https://doi.org/10.1038/onc.2015.497openalex-snapshot

Distinct routes to metastasis: plasticity-dependent and plasticity-independent pathways

inferredcites →https://doi.org/10.1038/onc.2016.358openalex-snapshot

ZEB1 induces LOXL2-mediated collagen stabilization and deposition in the extracellular matrix to drive lung cancer invasion and metastasis

inferredcites →https://doi.org/10.1038/s41467-018-07538-7openalex-snapshot

Hysteresis control of epithelial-mesenchymal transition dynamics conveys a distinct program with enhanced metastatic ability

inferredcites →https://doi.org/10.1038/s41523-017-0023-9openalex-snapshot

Inflammatory breast cancer: a model for investigating cluster-based dissemination

inferredcites →https://doi.org/10.1038/s41540-018-0076-xopenalex-snapshot

Role of noise and parametric variation in the dynamics of gene regulatory circuits

inferredcites →https://doi.org/10.1038/s41556-018-0196-yopenalex-snapshot

Non-redundant functions of EMT transcription factors

inferredcites →https://doi.org/10.1038/s41556-018-0268-zopenalex-snapshot

Snail-induced claudin-11 prompts collective migration for tumour progression

inferredcites →https://doi.org/10.1038/s41586-018-0040-3openalex-snapshot

Identification of the tumour transition states occurring during EMT

inferredcites →https://doi.org/10.1038/s41586-019-0915-yopenalex-snapshot

Neutrophils escort circulating tumour cells to enable cell cycle progression

inferredcites →https://doi.org/10.1038/s41598-017-16486-zopenalex-snapshot

Stiffness-dependent motility and proliferation uncoupled by deletion of CD44

inferredcites →https://doi.org/10.1038/s41598-018-21061-1openalex-snapshot

Epithelial-mesenchymal transition (EMT) signature is inversely associated with T-cell infiltration in non-small cell lung cancer (NSCLC)

inferredcites →https://doi.org/10.1038/s41598-018-27021-zopenalex-snapshot

Snail knockdown reverses stemness and inhibits tumour growth in ovarian cancer

inferredcites →https://doi.org/10.1038/s41598-018-27409-xopenalex-snapshot

CSF-1/CSF-1R axis is associated with epithelial/mesenchymal hybrid phenotype in epithelial-like inflammatory breast cancer

inferredcites →https://doi.org/10.1038/srep09188openalex-snapshot

Characterization of macrophage - cancer cell crosstalk in estrogen receptor positive and triple-negative breast cancer

inferredcites →https://doi.org/10.1038/srep14273openalex-snapshot

Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression

inferredcites →https://doi.org/10.1039/c6cp03174aopenalex-snapshot

Quantifying the landscape and kinetic paths for epithelial–mesenchymal transition from a core circuit

inferredcites →https://doi.org/10.1039/c6ib00139dopenalex-snapshot

Morphological single cell profiling of the epithelial–mesenchymal transition

inferredcites →https://doi.org/10.1063/1.5024874openalex-snapshot

Interconnected feedback loops among ESRP1, HAS2, and CD44 regulate epithelial-mesenchymal plasticity in cancer

inferredcites →https://doi.org/10.1073/pnas.0802047105openalex-snapshot

Notch signaling mediates hypoxia-induced tumor cell migration and invasion

inferredcites →https://doi.org/10.1073/pnas.1013171108openalex-snapshot

Random partitioning of molecules at cell division

inferredcites →https://doi.org/10.1073/pnas.1018898108openalex-snapshot

Inducible formation of breast cancer stem cells and their dynamic equilibrium with non-stem cancer cells via IL6 secretion

inferredcites →https://doi.org/10.1073/pnas.1318192110openalex-snapshot

MicroRNA-based regulation of epithelial–hybrid–mesenchymal fate determination

inferredcites →https://doi.org/10.1073/pnas.1416287112openalex-snapshot

Jagged–Delta asymmetry in Notch signaling can give rise to a Sender/Receiver hybrid phenotype

inferredcites →https://doi.org/10.1073/pnas.1508541113openalex-snapshot

Polyclonal breast cancer metastases arise from collective dissemination of keratin 14-expressing tumor cell clusters

inferredcites →https://doi.org/10.1073/pnas.1511814112openalex-snapshot

Jagged mediates differences in normal and tumor angiogenesis by affecting tip-stalk fate decision

inferredcites →https://doi.org/10.1073/pnas.1521812113openalex-snapshot

Immunoproteasome deficiency is a feature of non-small cell lung cancer with a mesenchymal phenotype and is associated with a poor outcome

inferredcites →https://doi.org/10.1073/pnas.1618298114openalex-snapshot

Integrin-β4 identifies cancer stem cell-enriched populations of partially mesenchymal carcinoma cells

inferredcites →https://doi.org/10.1073/pnas.1720113115openalex-snapshot

Fatty acid metabolism complements glycolysis in the selective regulatory T cell expansion during tumor growth

inferredcites →https://doi.org/10.1073/pnas.1722609115openalex-snapshot

Topography of epithelial–mesenchymal plasticity

inferredcites →https://doi.org/10.1073/pnas.1812876116openalex-snapshot

Acquisition of a hybrid E/M state is essential for tumorigenicity of basal breast cancer cells

inferredcites →https://doi.org/10.1073/pnas.1814204116openalex-snapshot

Dynamically stiffened matrix promotes malignant transformation of mammary epithelial cells via collective mechanical signaling

inferredcites →https://doi.org/10.1073/pnas.1815345116openalex-snapshot

Toward understanding cancer stem cell heterogeneity in the tumor microenvironment

inferredcites →https://doi.org/10.1073/pnas.1816391116openalex-snapshot

Elucidating cancer metabolic plasticity by coupling gene regulation with metabolic pathways

inferredcites →https://doi.org/10.1083/jcb.200601018openalex-snapshot

The epithelial–mesenchymal transition: new insights in signaling, development, and disease

inferredcites →https://doi.org/10.1083/jcb.200710067openalex-snapshot

Slug is a direct Notch target required for initiation of cardiac cushion cellularization

inferredcites →https://doi.org/10.1083/jcb.95.1.333openalex-snapshot

Epithelia suspended in collagen gels can lose polarity and express characteristics of migrating mesenchymal cells.

inferredcites →https://doi.org/10.1084/jem.20131916openalex-snapshot

PD-L1 is a novel direct target of HIF-1α, and its blockade under hypoxia enhanced MDSC-mediated T cell activation

inferredcites →https://doi.org/10.1088/1478-3975/aaf7b7openalex-snapshot

Stability and mean residence times for hybrid epithelial/mesenchymal phenotype

inferredcites →https://doi.org/10.1088/1478-3975/aaf8d4openalex-snapshot

Testing the gene expression classification of the EMT spectrum

inferredcites →https://doi.org/10.1088/978-0-7503-1134-2ch12openalex-snapshot

The role of macrophages during cancer cell transendothelial migration

inferredcites →https://doi.org/10.1091/mbc.e06-12-1096openalex-snapshot

Tumor Cell Apoptosis Polarizes Macrophages—Role of Sphingosine-1-Phosphate

inferredcites →https://doi.org/10.1091/mbc.e11-06-0537openalex-snapshot

Matrix rigidity regulates a switch between TGF-β1–induced apoptosis and epithelial–mesenchymal transition

inferredcites →https://doi.org/10.1093/carcin/bgz119openalex-snapshot

Functional balance between Tcf21–Slug defines cellular plasticity and migratory modalities in high grade serous ovarian cancer cell lines

inferredcites →https://doi.org/10.1093/intbio/zyz021openalex-snapshot

NRF2 activates a partial epithelial-mesenchymal transition and is maximally present in a hybrid epithelial/mesenchymal phenotype

inferredcites →https://doi.org/10.1096/fj.201600387ropenalex-snapshot

Numb and Numbl act to determine mammary myoepithelial cell fate, maintain epithelial identity, and support lactogenesis

inferredcites →https://doi.org/10.1098/rsif.2015.1106openalex-snapshot

Notch-Jagged signalling can give rise to clusters of cells exhibiting a hybrid epithelial/mesenchymal phenotype

inferredcites →https://doi.org/10.1098/rsif.2017.0512openalex-snapshot

Numb prevents a complete epithelial–mesenchymal transition by modulating Notch signalling

inferredcites →https://doi.org/10.1126/science.1099390openalex-snapshot

Bacterial Persistence as a Phenotypic Switch

inferredcites →https://doi.org/10.1126/science.1129139openalex-snapshot

Type, Density, and Location of Immune Cells Within Human Colorectal Tumors Predict Clinical Outcome

inferredcites →https://doi.org/10.1126/science.1147888openalex-snapshot

Stochasticity and Cell Fate

inferredcites →https://doi.org/10.1126/science.1203486openalex-snapshot

Cancer Immunoediting: Integrating Immunity’s Roles in Cancer Suppression and Promotion

inferredcites →https://doi.org/10.1126/science.1203543openalex-snapshot

A Perspective on Cancer Cell Metastasis

inferredcites →https://doi.org/10.1126/science.1228522openalex-snapshot

Circulating Breast Tumor Cells Exhibit Dynamic Changes in Epithelial and Mesenchymal Composition

inferredcites →https://doi.org/10.1126/science.7513443openalex-snapshot

Control of Cell Behavior During Vertebrate Development by Slug , a Zinc Finger Gene

inferredcites →https://doi.org/10.1126/scisignal.2005304openalex-snapshot

TGF-β–induced epithelial-to-mesenchymal transition proceeds through stepwise activation of multiple feedback loops

inferredcites →https://doi.org/10.1152/physrev.00005.2017openalex-snapshot

Notch Signaling in Development, Tissue Homeostasis, and Disease

inferredcites →https://doi.org/10.1155/2017/9624760openalex-snapshot

Macrophages as Key Drivers of Cancer Progression and Metastasis

inferredcites →https://doi.org/10.1158/0008-5472.can-04-1449openalex-snapshot

A Paracrine Loop between Tumor Cells and Macrophages Is Required for Tumor Cell Migration in Mammary Tumors

inferredcites →https://doi.org/10.1158/0008-5472.can-05-0699openalex-snapshot

The Fallacy of Epithelial Mesenchymal Transition in Neoplasia

inferredcites →https://doi.org/10.1158/0008-5472.can-06-3946openalex-snapshot

Cross-talk between Paracrine-Acting Cytokine and Chemokine Pathways Promotes Malignancy in Benign Human Prostatic Epithelium

inferredcites →https://doi.org/10.1158/0008-5472.can-08-1444openalex-snapshot

Epithelial-Mesenchymal Transition Induced by Growth Suppressor p12CDK2-AP1 Promotes Tumor Cell Local Invasion but Suppresses Distant Colony Growth

inferredcites →https://doi.org/10.1158/0008-5472.can-08-4312openalex-snapshot

Acquisition of Epithelial-Mesenchymal Transition Phenotype of Gemcitabine-Resistant Pancreatic Cancer Cells Is Linked with Activation of the Notch Signaling Pathway

inferredcites →https://doi.org/10.1158/0008-5472.can-09-2557openalex-snapshot

NOTCH Signaling Is Required for Formation and Self-Renewal of Tumor-Initiating Cells and for Repression of Secretory Cell Differentiation in Colon Cancer

inferredcites →https://doi.org/10.1158/0008-5472.can-09-3292openalex-snapshot

Antiestrogens Induce Transforming Growth Factor β–Mediated Immunosuppression in Breast Cancer

inferredcites →https://doi.org/10.1158/0008-5472.can-10-4269openalex-snapshot

Endothelial Cells Create a Stem Cell Niche in Glioblastoma by Providing NOTCH Ligands That Nurture Self-Renewal of Cancer Stem-Like Cells

inferredcites →https://doi.org/10.1158/0008-5472.can-12-2233openalex-snapshot

LOX-Mediated Collagen Crosslinking Is Responsible for Fibrosis-Enhanced Metastasis

inferredcites →https://doi.org/10.1158/0008-5472.can-13-0992openalex-snapshot

A Mechanism of Hypoxia-Mediated Escape from Adaptive Immunity in Cancer Cells

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Hypoxia-Induced Epithelial-to-Mesenchymal Transition in Hepatocellular Carcinoma Induces an Immunosuppressive Tumor Microenvironment to Promote Metastasis

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Epithelial-to-Mesenchymal Transition Contributes to Immunosuppression in Breast Carcinomas

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Survival Outcomes in Cancer Patients Predicted by a Partial EMT Gene Expression Scoring Metric

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A Biophysical Model Uncovers the Size Distribution of Migrating Cell Clusters across Cancer Types

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Epithelial–Mesenchymal Transition Is Associated with a Distinct Tumor Microenvironment Including Elevation of Inflammatory Signals and Multiple Immune Checkpoints in Lung Adenocarcinoma

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Epithelial–Mesenchymal Transition and Immune Evasion during Lung Cancer Progression: The Chicken or the Egg?

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Altered Expression and Splicing of ESRP1 in Malignant Melanoma Correlates with Epithelial–Mesenchymal Status and Tumor-Associated Immune Cytolytic Activity

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Molecular Drivers of the Non–T-cell-Inflamed Tumor Microenvironment in Urothelial Bladder Cancer

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PD-1 and PD-L1 Immune Checkpoint Blockade to Treat Breast Cancer

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Claudin-low bladder tumors are immune infiltrated and actively immune suppressed

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Epithelial-mesenchymal transition can suppress major attributes of human epithelial tumor-initiating cells

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Macrophage plasticity and polarization: in vivo veritas

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Hypoxic Microenvironment Induces EMT and Upgrades Stem-Like Properties of Gastric Cancer Cells

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Macrophages within NSCLC tumour islets are predominantly of a cytotoxic M1 phenotype associated with extended survival

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The M1 form of tumor-associated macrophages in non-small cell lung cancer is positively associated with survival time

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A high M1/M2 ratio of tumor-associated macrophages is associated with extended survival in ovarian cancer patients

inferredcites →https://doi.org/10.1186/bcr3344openalex-snapshot

Selective gene-expression profiling of migratory tumor cells in vivo predicts clinical outcome in breast cancer patients

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Macrophage phenotypic subtypes diametrically regulate epithelial-mesenchymal plasticity in breast cancer cells

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Human breast cancer cells educate macrophages toward the M2 activation status

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Mathematical modeling of tumor-associated macrophage interactions with the cancer microenvironment

inferredcites →https://doi.org/10.1186/s41236-017-0005-8openalex-snapshot

Distinguishing mechanisms underlying EMT tristability

inferredcites →https://doi.org/10.1200/jco.2010.30.5037openalex-snapshot

Tumor-Infiltrating CD8+Lymphocytes Predict Clinical Outcome in Breast Cancer

inferredcites →https://doi.org/10.1242/dev.063610openalex-snapshot

Notch signaling: simplicity in design, versatility in function

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Matrix stiffness mechanically conditions EMT and migratory behavior of oral squamous cell carcinoma

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Correction: Analysis of Epithelial and Mesenchymal Markers in Ovarian Cancer Reveals Phenotypic Heterogeneity and Plasticity

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Hybrid T-Helper Cells: Stabilizing the Moderate Center in a Polarized System

inferredcites →https://doi.org/10.1371/journal.pcbi.1005251openalex-snapshot

Population Heterogeneity in the Epithelial to Mesenchymal Transition Is Controlled by NFAT and Phosphorylated Sp1

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Interrogating the topological robustness of gene regulatory circuits by randomization

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A mechanism for epithelial-mesenchymal heterogeneity in a population of cancer cells

inferredcites →https://doi.org/10.1371/journal.pone.0016186openalex-snapshot

Analysis of Epithelial and Mesenchymal Markers in Ovarian Cancer Reveals Phenotypic Heterogeneity and Plasticity

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MiR-34a Targeting of Notch Ligand Delta-Like 1 Impairs CD15+/CD133+ Tumor-Propagating Cells and Supports Neural Differentiation in Medulloblastoma

inferredcites →https://doi.org/10.1371/journal.pone.0046391openalex-snapshot

Hypoxia Induces EMT in Low and Highly Aggressive Pancreatic Tumor Cells but Only Cells with Cancer Stem Cell Characteristics Acquire Pronounced Migratory Potential

showing the first 200 of 225

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Non-Genomic Actions of Estrogens on the DNA Repair Pathways Are Associated With Chemotherapy Resistance in Breast Cancer

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Emerging Role of Circulating Tumor Cells in Gastric Cancer

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Cysteine and Folate Metabolism Are Targetable Vulnerabilities of Metastatic Colorectal Cancer

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A Theoretical Approach to Coupling the Epithelial-Mesenchymal Transition (EMT) to Extracellular Matrix (ECM) Stiffness via LOXL2

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Immunity Depletion, Telomere Imbalance, and Cancer-Associated Metabolism Pathway Aberrations in Intestinal Mucosa upon Short-Term Caloric Restriction

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Lineage Plasticity in Cancer: The Tale of a Skin-Walker

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Single-Cell RNA-Seq Analysis Reveals the Acquisition of Cancer Stem Cell Traits and Increase of Cell–Cell Signaling during EMT Progression

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CD24 : un nouvel acteur de la résistance aux traitements et de la plasticité cellulaire dans les cancers du sein

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Partial EMT in Squamous Cell Carcinoma: A Snapshot

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