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Characterizing genetic intra-tumor heterogeneity across 2,658 human cancer genomes
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Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/cncr.28150openalex-snapshot
High intratumor genetic heterogeneity is related to worse outcome in patients with head and neck squamous cell carcinoma
inferredcites →https://doi.org/10.1002/path.1145openalex-snapshot
Significance of loss of heterozygosity of the RB1 gene during tumour progression in well‐differentiated liposarcomas
inferredcites →https://doi.org/10.1007/s12105-015-0617-1openalex-snapshot
Mutant Allele Tumor Heterogeneity (MATH) and Head and Neck Squamous Cell Carcinoma
inferredcites →https://doi.org/10.1016/j.cell.2012.04.023openalex-snapshot
The Life History of 21 Breast Cancers
inferredcites →https://doi.org/10.1016/j.cell.2013.01.019openalex-snapshot
Evolution and Impact of Subclonal Mutations in Chronic Lymphocytic Leukemia
inferredcites →https://doi.org/10.1016/j.cell.2013.03.002openalex-snapshot
Lessons from the Cancer Genome
inferredcites →https://doi.org/10.1016/j.cell.2017.01.018openalex-snapshot
Clonal Heterogeneity and Tumor Evolution: Past, Present, and the Future
inferredcites →https://doi.org/10.1016/j.cell.2017.09.042openalex-snapshot
Universal Patterns of Selection in Cancer and Somatic Tissues
inferredcites →https://doi.org/10.1016/j.cell.2018.03.029openalex-snapshot
The Evolutionary Landscape of Localized Prostate Cancers Drives Clinical Aggression
inferredcites →https://doi.org/10.1016/j.cell.2018.03.043openalex-snapshot
Deterministic Evolutionary Trajectories Influence Primary Tumor Growth: TRACERx Renal
inferredcites →https://doi.org/10.1016/j.cell.2018.03.057openalex-snapshot
Tracking Cancer Evolution Reveals Constrained Routes to Metastases: TRACERx Renal
inferredcites →https://doi.org/10.1016/j.oraloncology.2012.09.007openalex-snapshot
MATH, a novel measure of intratumor genetic heterogeneity, is high in poor-outcome classes of head and neck squamous cell carcinoma
inferredcites →https://doi.org/10.1038/300143a0openalex-snapshot
Mechanism of activation of a human oncogene
inferredcites →https://doi.org/10.1038/modpathol.3800447openalex-snapshot
Alterations of the RB1 gene in dedifferentiated liposarcoma
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The evolutionary history of lethal metastatic prostate cancer
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Absolute quantification of somatic DNA alterations in human cancer
inferredcites →https://doi.org/10.1038/ncomms14093openalex-snapshot
DNA copy number changes define spatial patterns of heterogeneity in colorectal cancer
inferredcites →https://doi.org/10.1038/ncomms9971openalex-snapshot
Systematic pan-cancer analysis of tumour purity
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Identification of neutral tumor evolution across cancer types
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Somatic ERCC2 mutations are associated with a distinct genomic signature in urothelial tumors
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Divergent modes of clonal spread and intraperitoneal mixing in high-grade serous ovarian cancer
inferredcites →https://doi.org/10.1038/ng.3891openalex-snapshot
Between-region genetic divergence reflects the mode and tempo of tumor evolution
inferredcites →https://doi.org/10.1038/ng1768openalex-snapshot
Genetic clonal diversity predicts progression to esophageal adenocarcinoma
inferredcites →https://doi.org/10.1038/nm.3886openalex-snapshot
Subclonal diversification of primary breast cancer revealed by multiregion sequencing
inferredcites →https://doi.org/10.1038/nm.3984openalex-snapshot
Pan-cancer analysis of the extent and consequences of intratumor heterogeneity
inferredcites →https://doi.org/10.1038/nrc2401openalex-snapshot
Tailoring to RB: tumour suppressor status and therapeutic response
inferredcites →https://doi.org/10.1038/nrclinonc.2015.175openalex-snapshot
The influence of subclonal resistance mutations on targeted cancer therapy
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inferredcites →https://doi.org/10.1038/s41467-020-14352-7openalex-snapshot
Reconstructing evolutionary trajectories of mutation signature activities in cancer using TrackSig
inferredcites →https://doi.org/10.1038/s41586-019-1907-7openalex-snapshot
The evolutionary history of 2,658 cancers
inferredcites →https://doi.org/10.1038/s41586-020-1943-3openalex-snapshot
The repertoire of mutational signatures in human cancer
inferredcites →https://doi.org/10.1038/s41586-020-1965-xopenalex-snapshot
Analyses of non-coding somatic drivers in 2,658 cancer whole genomes
inferredcites →https://doi.org/10.1038/s41586-020-1969-6openalex-snapshot
Pan-cancer analysis of whole genomes
inferredcites →https://doi.org/10.1038/s41587-019-0364-zopenalex-snapshot
A community effort to create standards for evaluating tumor subclonal reconstruction
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Quantification of subclonal selection in cancer from bulk sequencing data
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inferredcites →https://doi.org/10.1056/nejmoa1113205openalex-snapshot
Intratumor Heterogeneity and Branched Evolution Revealed by Multiregion Sequencing
inferredcites →https://doi.org/10.1056/nejmoa1508887openalex-snapshot
Resensitization to Crizotinib by the Lorlatinib ALK Resistance Mutation L1198F
inferredcites →https://doi.org/10.1056/nejmoa1616288openalex-snapshot
Tracking the Evolution of Non–Small-Cell Lung Cancer
inferredcites →https://doi.org/10.1073/pnas.0506580102openalex-snapshot
Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles
inferredcites →https://doi.org/10.1073/pnas.0801523105openalex-snapshot
Subclonal phylogenetic structures in cancer revealed by ultra-deep sequencing
inferredcites →https://doi.org/10.1073/pnas.1009843107openalex-snapshot
Allele-specific copy number analysis of tumors
inferredcites →https://doi.org/10.1073/pnas.1219747110openalex-snapshot
Intratumor heterogeneity in human glioblastoma reflects cancer evolutionary dynamics
inferredcites →https://doi.org/10.1084/jem.20120833openalex-snapshot
Cellular origin and pathophysiology of chronic lymphocytic leukemia
inferredcites →https://doi.org/10.1093/annonc/mdx784openalex-snapshot
Polyclonal RB1 mutations and acquired resistance to CDK 4/6 inhibitors in patients with metastatic breast cancer
inferredcites →https://doi.org/10.1097/00000658-197011000-00017openalex-snapshot
Thickness, Cross-Sectional Areas and Depth of Invasion in the Prognosis of Cutaneous Melanoma
inferredcites →https://doi.org/10.1097/nen.0000000000000226openalex-snapshot
BRAF Fusion Analysis in Pilocytic Astrocytomas: KIAA1549-BRAF 15-9 Fusions Are More Frequent in the Midline Than Within the Cerebellum
inferredcites →https://doi.org/10.1101/cshperspect.a026625openalex-snapshot
Principles of Reconstructing the Subclonal Architecture of Cancers
inferredcites →https://doi.org/10.1111/ics.12036openalex-snapshot
Beyond UV radiation: A skin under challenge
inferredcites →https://doi.org/10.1126/science.1253462openalex-snapshot
Spatial and temporal diversity in genomic instability processes defines lung cancer evolution
inferredcites →https://doi.org/10.1126/science.959840openalex-snapshot
The Clonal Evolution of Tumor Cell Populations
inferredcites →https://doi.org/10.1126/scitranslmed.aaa1408openalex-snapshot
Clonal status of actionable driver events and the timing of mutational processes in cancer evolution
inferredcites →https://doi.org/10.1136/gutjnl-2016-312268openalex-snapshot
Palbociclib (PD-0332991), a selective CDK4/6 inhibitor, restricts tumour growth in preclinical models of hepatocellular carcinoma
inferredcites →https://doi.org/10.1158/0008-5472.can-04-1738openalex-snapshot
The Combination of Genetic Instability and Clonal Expansion Predicts Progression to Esophageal Adenocarcinoma
inferredcites →https://doi.org/10.1186/s13059-014-0453-8openalex-snapshot
Deciphering intratumor heterogeneity and temporal acquisition of driver events to refine precision medicine
inferredcites →https://doi.org/10.3233/blc-170105openalex-snapshot
CDK4/6 Inhibitors in Cancer Therapy: A Novel Treatement Strategy for Bladder Cancer
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Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Reconstructing evolutionary trajectories of mutation signature activities in cancer using TrackSig
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Accurate quantification of copy-number aberrations and whole-genome duplications in multi-sample tumor sequencing data
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Mutation or not, what directly establishes a neoplastic state, namely cellular immortality and autonomy, still remains unknown and should be prioritized in our research
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Improving susceptibility of neuroendocrine tumors to radionuclide therapies: personalized approaches towards complementary treatments
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