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SCD1 inhibition enhances the effector functions of CD8 + T cells via ACAT1 ‐dependent reduction of esterified cholesterol
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Cholesterol Induces CD8+ T Cell Exhaustion in the Tumor Microenvironment
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Role of stearoyl-coenzyme A desaturase in lipid metabolism
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X-ray structure of a mammalian stearoyl-CoA desaturase
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SCD1 inhibition enhances the effector functions of CD8 + T cells via ACAT1 ‐dependent reduction of esterified cholesterol
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What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Cholesterol Metabolism-Mediated Immune Regulation and Cancer Immunotherapy
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inferred← citeshttps://doi.org/10.1038/s41598-024-64043-2openalex-snapshot
FADS2 confers SCD1 inhibition resistance to cancer cells by modulating the ER stress response
inferred← citeshttps://doi.org/10.1080/07853890.2024.2447403openalex-snapshot
The role of cisplatin in modulating the tumor immune microenvironment and its combination therapy strategies: a new approach to enhance anti-tumor efficacy
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Propofol suppressed cell proliferation through inhibition of SREBP1c-mediated De novo lipogenesis in colorectal cancer cells
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Scd-1 deficiency promotes the differentiation of CD8+ T effector
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